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anti-Human Angiotensin II/III Antibodies:
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Results indicate that urinary angiotensinogen (show AGT Antibodies) was associated with blood pressure elevation in this population of obese young adults.
our analysis revealed that the associations of the AGT (show AGXT Antibodies) variants with T2DM were independently associated.
The urinary angiotensinogen (show AGT Antibodies) level correlates with the overall maturity of renal function during the early postnatal period in critically ill neonates.
the association of polymorphisms in angiotensinogen (AGT (show AGT Antibodies)) and angiotensin II type 1 receptor (AGTR1 (show AGTR1 Antibodies)), with blood lead levels and lead-related blood pressure in lead-exposed male workers in Korea, was investigated.
the AGT (show AGXT Antibodies) polymorphisms have played a vital role in determining an individual's susceptibility to essential hypertension.
There were significant differences in genotype and allele distributions of ACE (show ACE Antibodies) gene rs4343 (pgentoype = 0.002 and pallele < 0.001) and AGTR1 (show AGTR1 Antibodies) gene rs5186 polymorphisms.
rs699 in the AGT gene and rs5186 in the ATR1 gene were not associated with the risk and clinical outcomes of IgAN.
The threonine variant of AGT (show AGXT Antibodies) M268T is a significant risk factor for abnormalities in specific white matter tracts and cognitive domains in healthy older adults.
Increased plasma renin, AT and VEGF might play a role in the onset or development of superficial infantile hemangiomas.
Blood pressure was significantly lower in angiotensinogen (show AGT Antibodies) knock-out mice compared to controls fed a normal or high sodium diet.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8