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anti-Human Angiotensin II/III Antibodies:
anti-Rat (Rattus) Angiotensin II/III Antibodies:
anti-Mouse (Murine) Angiotensin II/III Antibodies:
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Human Monoclonal Angiotensin II/III Primary Antibody for IHC (p), IHC - ABIN253542
Ni, Ma, Wang, Liu, Zhang, Lv, Ni, Chen, Ruan, Liu: Activation of renin-angiotensin system is involved in dyslipidemia-mediated renal injuries in apolipoprotein E knockout mice and HK-2 cells. in Lipids in health and disease 2014
Show all 7 references for ABIN253542
endoplasmic reticulum stress induces apoptosis in human alveolar epithelial cells through mediation of unfolded protein response pathways, which in turn regulate the autocrine ANGII/ANG1 (show ANGPT1 Antibodies)-7 system.
Angiotensin II stimulates PYY secretion, in turn inhibiting epithelial anion fluxes, thereby reducing net fluid secretion into the colonic lumen.
NOXs had two time-dependent reactions in response to Ang II (show AGT Antibodies) stimulation via MAPK (show MAPK1 Antibodies) pathwa
increased Ang-II/Ang-(1 (show ANGPT1 Antibodies)-7) ratios may lead to Ang-II (show AGT Antibodies) over-activation and aggravate atherosclerosis progression.
urinary angiotensinogen (show AGT Antibodies) could be a prognostic marker of the albuminuria reduction effects of olmesartan in patients with metabolic syndrome
AGT (show AGXT Antibodies) expression is significantly upregulated in human masticatory mucosa during wound healing
Urinary angiotensinogen reflects intrarenal RAS status, and is of value to assess the severity of acute tubular necrosis .
Data suggest that enhanced survival of normal prostate epithelial cells (PNT1A cell line) in presence of either or both angiotensin II (Ang II) and relaxin 2 (RLN2 (show RLN1 Antibodies)) appears to be associated with increased ratio of BCL2 (show BCL2 Antibodies)/BAX (show BAX Antibodies) messenger RNA expression; thus, Ang II (show AGT Antibodies) and/or RLN2 (show RLN1 Antibodies) appear to be involved in regulation of apoptosis in prostate epithelium.
increasing the serum levels of AngII increased the risk of acute myocardial infarction (AMI (show CFD Antibodies)); the risk of AMI (show CFD Antibodies) increased when the serum levels of AngII and KLK1 (show KLK1 Antibodies) simultaneously increased; individuals with the combined genotypes of ACE (show ACE Antibodies) DD and KLK1 (show KLK1 Antibodies) GG showed increased risk of AMI (show CFD Antibodies) compared with those with the combined genotypes of ACE (show ACE Antibodies) II and KLK1 (show KLK1 Antibodies) AA
genetic association study and meta-analysis revealed a significant association between M235T polymorphism of angiotensinogen (show AGT Antibodies) and myocardial infarction risk
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8