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anti-Human Cardiotrophin 1 Antibodies:
anti-Mouse (Murine) Cardiotrophin 1 Antibodies:
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Human Polyclonal Cardiotrophin 1 Primary Antibody for EIA, WB - ABIN115501
Liao, Brar, Cai, Stephanou, OLeary, Pennica, Yellon, Latchman: Cardiotrophin-1 (CT-1) can protect the adult heart from injury when added both prior to ischaemia and at reperfusion. in Cardiovascular research 2002
Show all 6 references for ABIN115501
Human Monoclonal Cardiotrophin 1 Primary Antibody for EIA, WB - ABIN114699
Talwar, Squire, Obrien, Downie, Davies, Ng: Plasma cardiotrophin-1 following acute myocardial infarction: relationship with left ventricular systolic dysfunction. in Clinical science (London, England : 1979) 2001
Show all 5 references for ABIN114699
Human Monoclonal Cardiotrophin 1 Primary Antibody for WB - ABIN108472
Freed, Moon, Borowiec, Jones, Zahradka, Dixon: Cardiotrophin-1: expression in experimental myocardial infarction and potential role in post-MI wound healing. in Molecular and cellular biochemistry 2003
Show all 4 references for ABIN108472
Human Polyclonal Cardiotrophin 1 Primary Antibody for EIA, WB - ABIN951045
Fritzenwanger, Meusel, Jung, Franz, Wang, Foerster, Figulla: Cardiotrophin-1 induces tumor necrosis factor alpha synthesis in human peripheral blood mononuclear cells. in Mediators of inflammation 2010
Show all 2 references for ABIN951045
Cow (Bovine) Polyclonal Cardiotrophin 1 Primary Antibody for WB - ABIN2784702
Yu, Kissling, Freyschmidt-Paul, Hoffmann, Shapiro, McElwee: Interleukin-6 cytokine family member oncostatin M is a hair-follicle-expressed factor with hair growth inhibitory properties. in Experimental dermatology 2007
This paper will review many aspects of CT-1 physiological role in several organs and discuss data for consideration in therapeutic approaches.
biliary epithelium-derived CT-1 may exert a profibrogenic potential in PCLD (show PRKCSH Antibodies).
Data show that cardiotrophin-1 is positively related to brachial-ankle pulse-wave velocity (baPWV) independent of traditional cardiometabolic risk factors for arterial stiffness.
this study, even though preliminary and awaiting further confirmation by independent replication, provides first evidence that common genetic variation in CTF1 could contribute to insulin (show INS Antibodies) sensitivity in humans.
Study correlates CT-1 levels with ambulatory and central BP, as well as with pulse wave velocity in patients with essential hypertension.
CT-1 is differentially induced in the myocardium of infants with congenital cardiac defects depending on hypoxemia, and may mediate myocardial hypertrophy and dysfunction.
Subjects with impaired glucose tolerance (IGT) and newly diagnosed diabetes (NDD) have significantly higher CT-1 concentrations than those with normal glucose tolerance. IGT and NDD are positively associated with CT-1 concentrations.
exaggerated cardiomyocyte production of cardiotrophin-1 in response to increased left ventricular end-diastolic stress may contribute to fibrosis through stimulation of fibroblasts in heart failure of hypertensive origin.
A reduction in serum CT-1 levels after a WL program.
CT-1 induces the proteolytic potential in human aortic endothelial cells by upregulating MMP-1 (show MMP1 Antibodies) expression.
Nuclear translocation of CT-1 regulates cardiomyogenesis of ES cells and involves calcium, NO, ROS (show ROS1 Antibodies) as well as CT-1 regulated signaling pathways.
Data suggest that Ctf1 up-regulates lipolysis in white adipocytes via 1) induction of perilipin (show PLIN1 Antibodies), 2) activation of hormone sensitive lipase (show LIPE Antibodies) (via phosphorylation by PKA), and 3) inactivation of adipose triglyceride lipase (show PNPLA2 Antibodies) (via up-regulation of G0S2 (show G0S2 Antibodies)).
Cardiotrophin-1 (CT-1) is a hepatoprotective cytokine that modulates fat and glucose metabolism. Here we analyzed the changes in hepatic fat stores induced by recombinant CT-1 and its therapeutic potential in non-alcoholic fatty liver disease.
The transgenic expression of CTF1 brought about a marked improvement on cognitive functioning in the APPswe/PS1dE9 transgenic mouse model of Alzheimer's disease.
CT-1 improves beta cell function and survival, and protects mice against STZ-induced diabetes
CT-1 may be a major regulator of arterial stiffness with a major impact on the aging process.
Hypoxia increased cardiotrophin-1 levels in cardiac cells through a direct regulation of CT-1 promoter by HIF-1alpha (show HIF1A Antibodies), protecting cells from apoptosis.
Data suggest a key role for CT-1 in cardiac remodeling induced by aldosterone independent of changes in blood pressure levels.
We conclude that CT-1 is a master regulator of fat and glucose metabolism with potential applications for treatment of obesity and insulin (show INS Antibodies) resistance.
Treatment of embryoid bodies grown from pluripotent murine embryonic stem (ES) cells with cardiotrophin 1 significantly stimulated cardiomyogenesis and increased nuclear expression of the proliferation marker Ki-67 (show MKI67 Antibodies).
CT1 (show SLC6A8 Antibodies) decreases cell death through a mechanism related to Stat3 (show STAT3 Antibodies) and Akt (show AKT1 Antibodies) phosphorylation and activation of calpastatin (show CAST Antibodies) in D-galactosamine-treated hepatocytes.
The protein encoded by this gene is a secreted cytokine that induces cardiac myocyte hypertrophy in vitro. It has been shown to bind and activate the ILST/gp130 receoptor. Two transcript variants encoding different isoforms have been found for this gene.
, cardiophin 1