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Human CSF2 ELISA Kit for Sandwich ELISA - ABIN414381
Pollard, Rockstroh, Pantaleo, Asmuth, Peters, Lazzarin, Garcia, Ellefsen, Podzamczer, van Lunzen, Arastéh, Schürmann, Clotet, Hardy, Mitsuyasu, Moyle, Plettenberg, Fisher, Fätkenheuer, Fischl, Taiwo et al.: Safety and efficacy of the peptide-based therapeutic vaccine for HIV-1, Vacc-4x: a phase 2 randomised, double-blind, placebo-controlled trial. ... in The Lancet. Infectious diseases 2014
Mouse (Murine) CSF2 ELISA Kit for Sandwich ELISA - ABIN415372
Cao, Xu, Jin, Zhang, Lu, Nie, Tong: Effects of 900-MHz microwave radiation on gamma-ray-induced damage to mouse hematopoietic system. in Journal of toxicology and environmental health. Part A 2010
TRAF6 (show TRAF6 ELISA Kits) is also required for GM-CSF-induced ubiquitination and activation of Akt (show AKT1 ELISA Kits).
Data suggest that the intratumoral GM-CSF expression, as a potentially independent prognostic biomarker for recurrence, might improve conventional clinical and pathologic analysis to refine outcome prediction for clinically localized clear-cell renal cell carcinoma (ccRCC) patients after surgery.
High GM-CSF expression is associated with breast Cancer.
In gastric cancer (GC), tumour-derived GM-CSF activated neutrophils and induced neutrophil PD-L1 (show CD274 ELISA Kits) expression via Janus kinase (JAK (show JAK3 ELISA Kits))-signal transducer and activator of transcription 3 (STAT3 (show STAT3 ELISA Kits)) signalling pathway. The activated PD-L1 (show CD274 ELISA Kits)(+) neutrophils effectively suppressed normal T-cell immunity in vitro and contributed to the growth and progression of human GC in vivo.
The IL-3 (show IL-3 ELISA Kits)/ GM-CSF effected on the myofibroblastic differentiation of human adipose derive stromal cells (hASCs) as well as it did on human dermal fibroblasts (HDFs).
Obesity alters the lung neutrophil infiltration to enhance breast cancer metastasis through IL5 (show IL5 ELISA Kits) and GM-CSF.
Data suggest that the methylotrophic yeast Pichia pastoris is an effective recombinant host for heterologous granulocyte-macrophage colony-stimulating factor (rhGM-CSF) production.
Letter: Knockdown of either filaggrin (show FLG ELISA Kits) or loricrin (show LOR ELISA Kits) increases the productions of interleukin (IL)-1alpha, IL-8 (show IL8 ELISA Kits), IL-18 (show IL18 ELISA Kits) and granulocyte macrophage colony-stimulating factor in stratified human keratinocytes.
Impaired RASGRF1 (show RASGRF1 ELISA Kits)/ERK (show EPHB2 ELISA Kits)-mediated GM-CSF response characterizes CARD9 (show CARD9 ELISA Kits) deficiency in French-Canadians.
Honokiol could possess potential anti-inflammatory effects and inhibits TNF-alpha (show TNF ELISA Kits)-induced IL-1beta (show IL1B ELISA Kits), GM-CSF and IL-8 (show IL8 ELISA Kits) production in PBMCs from rheumatoid arthritis patients.
The canonical NFkappaB (show NFKB1 ELISA Kits) signaling in fibroblasts, but not in tumor cells, was shown to be responsible for the induced and constitutive CSF2 expression.
These results demonstrated that bovine colonic cells seem capable to respond to E. bovis merozoite I infection by the upregulation of CXCL10 (show CXCL10 ELISA Kits) and GM-CSF gene transcription.
Data suggest exposure to maternal CSF2 from D5-D7 of development is fundamentally different for female/male blastocysts with respect to embryo elongation, characteristics of transcriptome/methylome, and endometrial interferon tau secretion at D15 (show MRPL16 ELISA Kits).
The increase in calving rate caused by CSF2 treatment involves, in part, more extensive development of extraembryonic membranes and capacity of the conceptus to secrete IFNT2 at day 15 of pregnancy.
immunolocalization studies confirmed the presence of granulocyte-macrophage colony stimulating factor(GM-CSF) in the germ cell line in bovine and human testes and addition of GM-CSF enhances several parameters of sperm motility
the conceptus, through its secretion of IFN-tau, stimulates maternal epithelial expression of COX-2 (show PTGS2 ELISA Kits) and GM-CSF during the peri (show PLIN1 ELISA Kits)-attachment period in the cow.
CSF2 affect embryonic development and enhance embryo competence for posttransfer survival.
T-GM-CSF and -IL-3 (show IL-3 ELISA Kits) significantly, and reciprocally, blunted receptor binding and myeloid progenitor cell proliferation activity of both FL-GM-CSF and -IL-3 (show IL-3 ELISA Kits) in vitro and in vivo
Results indicate GM-CSF as both a key contributor to the pathogenesis of MI and a potential therapeutic target.
GM-CSF is required for the normal balance of leukocyte subsets, including granulocytes, B cells, and naive vs. effector T cells. There was an approximately 3-fold increase in the percentages of granulocytes in Csf2-/- PBMCs. The presence of maximal experimental autoimmune encephalomyelitis in the complete absence of GM-CSF revealed that GM-CSF is not an obligate effector molecule in all forms of EAE.
chemerin (show RARRES2 ELISA Kits) inhibited nuclear factor-kappaB activation and the expression of granulocyte-macrophage colony-stimulating factor (GM-CSF) and interleukin-2 (show IL2 ELISA Kits) (IL-6 (show IL6 ELISA Kits)) by tumor cells and tumor-associated endothelial cell, respectively, via its receptors, and consequently, MDSC induction was impaired, leading to restoration of antitumor T-cell response and decreased tumor angiogenesis.
These findings describe a novel role for GM-CSF as an essential initiating cytokine in cardiac inflammation.
Data reviewed establish that any damage to brain tissue tends to cause an increase in G-CSF (show CSF3 ELISA Kits) and/or GM-CSF (G(M)-CSF (show CSF1R ELISA Kits)) synthesized by the brain. Glioblastoma cells themselves also synthesize G(M)-CSF (show CSF1R ELISA Kits). G(M)-CSF (show CSF1R ELISA Kits) synthesized by brain due to damage by a growing tumor and by the tumor itself stimulates bone marrow to shift hematopoiesis toward granulocytic lineages away from lymphocytic lineages.
Evi1 (show MECOM ELISA Kits)(+)DA-3 cells modified to express an intracellular form of GM-CSF, acquired growth factor independence and transplantability and caused an overt leukemia in syngeneic hosts, without increasing serum GM-CSF levels.
IL-23 (show IL23A ELISA Kits)-induced GM-CSF mediates the pathogenicity of CD4 (show CD4 ELISA Kits)(+) T cells in experimental autoimmune myocarditis.
GM-CSF accelerated the G1/S phase transition in EPCs by upregulating the expression of cyclins D1 and E.
CSF2 stimulates proliferation of trophectoderm cells by activation of the PI3K-and ERK1/2 (show MAPK1/3 ELISA Kits) MAPK (show MAPK1 ELISA Kits)-dependent MTOR (show FRAP1 ELISA Kits) signal transduction cascades.
Data indicate that differentially expressed IL-17 (show IL17A ELISA Kits), IL-22 (show IL22 ELISA Kits), and IL-23 (show IL23A ELISA Kits) levels are associated with K-ras (show HRAS ELISA Kits) in a stage-specific fashion along colorectal cancer progression and an association was established between mutant K-ras (show HRAS ELISA Kits) and GM-CSF and IFN-gamma (show IFNG ELISA Kits).
The protein encoded by this gene is a cytokine that controls the production, differentiation, and function of granulocytes and macrophages. The active form of the protein is found extracellularly as a homodimer. This gene has been localized to a cluster of related genes at chromosome region 5q31, which is known to be associated with interstitial deletions in the 5q- syndrome and acute myelogenous leukemia. Other genes in the cluster include those encoding interleukins 4, 5, and 13.
granulocyte-macrophage colony-stimulating factor
, colony stimulating factor 2 (granulocyte-macrophage)
, colony-stimulating factor
, granulocyte-macrophage colony stimulating factor 2
, put. GM-CSF
, granulate-macrophage stimulating factor
, granulocyte-macrophage stimulation factor