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Human EPOR ELISA Kit for Sandwich ELISA - ABIN414365
Villafuerte, Macarlupú, Anza-Ramírez, Corrales-Melgar, Vizcardo-Galindo, Corante, León-Velarde: Decreased plasma soluble erythropoietin receptor in high-altitude excessive erythrocytosis and Chronic Mountain Sickness. in Journal of applied physiology (Bethesda, Md. : 1985) 2014
These results identify EPOR as the secondbona fidehydroxylation-dependent substrate of VHL (show VHL ELISA Kits) that potentially influences oxygen homeostasis and contributes to the complex genotype-phenotype correlation in VHL (show VHL ELISA Kits) disease.
We report for a first time that functional EpoR is expressed in human rhabdomyosarcoma cell lines as well as by primary tumors from RMS patients.
erythrocyte lineage enforces exclusivity through upregulation of EKLF (show KLF1 ELISA Kits) and its lineage-specific cytokine receptor (show EBI3 ELISA Kits) (EpoR) while inhibiting both FLI-1 (show FLI1 ELISA Kits) and the receptor TpoR (show MPL ELISA Kits) (also known as MPL (show MPL ELISA Kits)) for the opposing megakaryocyte lineage
A new point mutation in EPOR induces a short deletion in congenital erythrocytosis.
Data show that erythropoietin receptor antagonist EMP9 suppressed hemoglobin synthesis in xenografts of HeLa cells.
Data suggest that erythropoietin receptor (EPOR) could be a target to overcome therapeutic resistance toward ionizing radiation or temozolomide.
basic mechanisms for hypoxia-inducible factor induction of EPO (show EPO ELISA Kits) expression, and within erythroid progenitors EPOR engagement of canonical Janus kinase 2 (show JAK2 ELISA Kits)/signal transducer and activator of transcription 5 (show STAT5A ELISA Kits) (JAK2 (show JAK2 ELISA Kits)/STAT5 (show STAT5A ELISA Kits))
transmembrane domain and the juxtamembrane region of the erythropoietin receptor in micelles
while EPO (show EPO ELISA Kits) can stimulate NO production, NO in turn can regulate EPOR expression in endothelial cells during hypoxia
The progressive increase of EPO (show EPO ELISA Kits) and EpoR mRNA can already be observed during the fibrotic-cirrhotic development with a peak of expression rising at tumor formation.
These data indicate that EpoR signaling is associated with cardiac remodeling following chronic iron deficiency.
We propose that the CID (show CENPA ELISA Kits)-dependent dimerization system combined with the EpoR intracellular domain and the Gata1 (show GATA1 ELISA Kits) gene regulatory region generates a novel peroral strategy for the treatment of anemia.
EpoR and its activity are downstream effectors of Klotho (show KL ELISA Kits) enabling it to function as a cytoprotective protein against oxidative injury.
Expression of EPOR in rod photoreceptors, Muller cells, and amacrine, horizontal, and ganglion cells of the peripheral retina is not required for the maturation, function, and survival of these cells in aging tissue.
Data indicate a Cbl (show CBL ELISA Kits)/p85 (show ECM1 ELISA Kits)/epsin-1 (show EPN1 ELISA Kits) pathway in erythropoietin receptor (EpoR) endocytosis.
Data from knockout mice suggest that adipose tissue-specific disruption of EPO (show EPO ELISA Kits) receptor does not alter adipose tissue expansion, adipocyte morphology, insulin (show INS ELISA Kits) resistance, inflammation, or angiogenesis.
the EPO (show EPO ELISA Kits)-EPOR system may play a role in glucose metabolism within adipocytes.
EPOR regulates transcriptome for primary progenitors, including Tnfr (show TNFRSF1A ELISA Kits)-sf13c as a novel mediator of EPO (show EPO ELISA Kits)-dependent erythroblast formation.
These findings reveal a role of endogenous EPO (show EPO ELISA Kits)/EPOR for cognition, at least in schizophrenic patients.
This gene encodes the erythropoietin receptor which is a member of the cytokine receptor family. Upon erythropoietin binding, this receptor activates Jak2 tyrosine kinase which activates different intracellular pathways including: Ras/MAP kinase, phosphatidylinositol 3-kinase and STAT transcription factors. The stimulated erythropoietin receptor appears to have a role in erythroid cell survival. Defects in the erythropoietin receptor may produce erythroleukemia and familial erythrocytosis. Dysregulation of this gene may affect the growth of certain tumors. Alternate splicing results in multiple transcript variants.
, Erythropoietin receptor
, type I single-transmembrane cytokine receptor
, erythropoietin receptor-like