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Mouse (Murine) IFNA1 ELISA Kit for Sandwich ELISA - ABIN1446139
Nagill, Kaur, Joshi, Kaur: Immunogenicity and efficacy of recombinant 78 kDa antigen of Leishmania donovani formulated in various adjuvants against murine visceral leishmaniasis. in Asian Pacific journal of tropical medicine 2015
Taken together, this work suggests that IFNa (show IFNA ELISA Kits) provides protection of salmon against SAV3 locally in an infected area while IFNb (show IFNB1 ELISA Kits) and IFNc provides systemic protection against the virus.
IFNa (show IFNA ELISA Kits) is the main IFN subtype induced through salmon RIG-I (show DDX58 ELISA Kits)/viral RNA receptor MDA5 (show IFIH1 ELISA Kits) pathway in lymphoid tissues.
These findings also identify STAT3 (show STAT3 ELISA Kits) as a therapeutic target against viral infection and highlight it as an essential pathway component for endogenous and therapeutic IFN-alpha (show IFNA ELISA Kits) responsiveness.
the close association between the increased proportion of CD180 (show CD180 ELISA Kits)-negative B cells and the activation of IFN-alpha (show IFNA ELISA Kits) signaling in Systemic lupus erythematosus, is reported.
IL-12 (show IL12A ELISA Kits) and IFN-alpha (show IFNA ELISA Kits) differentially program CD8 (show CD8A ELISA Kits) T cells to re-express distinct levels of PD-1 (show PDCD1 ELISA Kits) upon re-encountering Ag, resulting in IL-12 (show IL12A ELISA Kits)-stimulated cells being less susceptible to exhaustion in the face of sustained tumor Ag.
The expansion of CD8 (show CD8A ELISA Kits) T cells depends on type I IFN, type I IFN and IL-12 (show IL12A ELISA Kits) or is largely independent of the two cytokines.
A spontaneous genomic duplication and frameshift mutation in the guanine exchange factor dedicator of cytokinesis 2 (Dock2 (show DOCK2 ELISA Kits)) that has arisen in at least a subset of circulating Irf5 (show IRF5 ELISA Kits)(-/-) mice and inadvertently been bred to homozygosity.
Knockdown of endogenous guanylate binding protein (GBP)4 (show GBP4 ELISA Kits) increases IRF7 (show IRF7 ELISA Kits)-mediated IFN-alpha (show IFNA ELISA Kits) production, whereas overexpression of GBP4 (show GBP4 ELISA Kits) has the opposite effect.
Data show that similar T cell expansion and serum IgG responses were observed in adenovirus (Adv (show AVIL ELISA Kits))-IFN-treated WT and BAFF (show TNFSF13B ELISA Kits)-deficient mice despite their disparate pathological and clinical responses.
Studies in both mice and humans have demonstrated a role for IFN-alpha (show IFNA ELISA Kits)/beta in directly influencing the fate of both CD4 (show CD4 ELISA Kits)(+) and CD8 (show CD8A ELISA Kits)(+) T cells during the initial phases of antigen recognition.
IFN-alpha (show IFNA ELISA Kits) enhances both the induction and maintenance of programmed cell death (PD)-1 (show PDCD1 ELISA Kits) expression on T cell receptor-engaged primary mouse T cells through association of IFN-responsive factor 9 (IRF9 (show IRF9 ELISA Kits)) with the IFN stimulation response element.
This article reports the production of interferon alpha (show IFNA ELISA Kits)/beta (IFN-alpha (show IFNA ELISA Kits)/beta) by SJL/J mouse brain astrocyte cultures infected with Theiler's murine encephalomyelitis virus (TMEV).
results demonstrate that Sphingosine 1-phosphate lyase (SPL (show SGPL1 ELISA Kits)) is a host factor that augments type I IFN responses during influenza A virus infection; study delineates the relationship between IKKepsilon (show IKBKE ELISA Kits) and SPL (show SGPL1 ELISA Kits), which provides a mechanistic understanding of the pro-IFN activity of SPL (show SGPL1 ELISA Kits)
These data suggest that plasmacytoid dendritic cells producing IFN-alpha (show IFNA ELISA Kits) and IL-33 (show IL33 ELISA Kits) play a pivotal role in the chronic fibro-inflammatory responses underlying murine autoimmune pancreatitis and human IgG4-related autoimmune pancreatitis.
Mothers of children affected by autoimmune congenital heart block had a significantly higher expression IFN-alpha (show IFNA ELISA Kits).
Suppression of the exogenous Type I IFN-induced Jak (show JAK3 ELISA Kits)/STAT (show STAT1 ELISA Kits) signaling by NSs might be one of the mechanisms of Severe fever with thrombocytopenia syndrome (SFTS) to evade host immune surveillance.
High Type I Interferon (show IFNA ELISA Kits) expression due to hypomethylation is associated with Systemic Lupus Erythematosus.
these studies identify phosphorylation of S734-STAT2 (show STAT2 ELISA Kits) as a new regulatory mechanism that negatively controls the type I IFN-antiviral response.
The synthetic IFNP molecule exerted its antitumor activity by upregulating the downstream genes involved in the STAT1 (show STAT1 ELISA Kits) pathway and in apoptosis. Using a cell receptor binding assay, we showed that this Jurkat-binding peptide (show HP ELISA Kits) facilitated the binding affinity of IFNalpha to the cell surface type I IFN receptor
KSHV-encoded viral IRF4 (show IRF4 ELISA Kits) interacts with the host IRF7 (show IRF7 ELISA Kits) and inhibits interferon-alpha (show IFNA ELISA Kits) production.
Data indicate ARID3a (show ARID3A ELISA Kits)(+) B cells as a type of effector B cell, and link ARID3a (show ARID3A ELISA Kits) expression in B lymphocytes to interferon alpha (IFNa (show IFNA ELISA Kits))-associated inflammatory responses in systemic lupus erythematosus (SLE).
Collectively, these data show that porcine epidemic diarrhea virus is capable of subverting the type I interferon (show IFNA ELISA Kits) response by inducing STAT1 (show STAT1 ELISA Kits) degradation.
Amino acid residues in the N-terminal domain of Npro are involved in the stability of Npro, in interaction of Npro with IRF-3 (show IRF3 ELISA Kits) and subsequent degradation of IRF-3 (show IRF3 ELISA Kits), leading to downregulation of IFN-alpha (show IFNA ELISA Kits)/beta production.
Pregnane X receptor is required for interferon-alpha-mediated CYP3A29 expression, and its expression before CYP3A29.
Overall, data provide evidence for the possible role of PI3K in the activation of the transcription of IFN-alpha (show IFNA ELISA Kits)/beta by PRRSV; study concludes that PRRSV inhibits the induction of IFN-alpha (show IFNA ELISA Kits) in monocyte-derived dendritic cells by as yet undefined post-transcriptional mechanisms.
Nsp1beta inhibits interferon-activated (show MNDA ELISA Kits) STAT1 (show STAT1 ELISA Kits)/STAT2 (show STAT2 ELISA Kits) signal transduction by inducing karyopherin-alpha1 degradation.
Expression of Mx protein (show MX2 ELISA Kits) and interferon-alpha (IFN-alpha (show IFNA ELISA Kits)) was examined by immunohistochemistry in pigs experimentally infected with swine influenza virus.
Foot-and-Mouth Disease Virus inhibits IFN-alpha (show IFNA ELISA Kits) expression in infected cells by blocking cap-dependent translation.
The protein encoded by this gene is produced by macrophages and has antiviral activity. This gene is intronless and the encoded protein is secreted.
, interferon alpha 1
, interferon alpha 13
, interferon alpha family gene 1
, interferon alpha-1
, IFN-alpha 1
, interferon-alpha 1
, IFN-alpha 1b
, interferon alpha 1b
, interferon alpha-D
, leIF D