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anti-Mouse (Murine) IFNAR1 Antibodies:
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Human Polyclonal IFNAR1 Primary Antibody for CyTOF, FACS - ABIN4900075
Lin, Liao, Lin, Lin: Blocking of the alpha interferon-induced Jak-Stat signaling pathway by Japanese encephalitis virus infection. in Journal of virology 2004
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Human Monoclonal IFNAR1 Primary Antibody for FACS - ABIN4897145
Stone, Giugliano, Schnell, Cheng, Leahy, Golden-Mason, Gale, Rosen: Hepatitis C virus pathogen associated molecular pattern (PAMP) triggers production of lambda-interferons by human plasmacytoid dendritic cells. in PLoS pathogens 2013
Show all 3 Pubmed References
Human Monoclonal IFNAR1 Primary Antibody for FACS - ABIN4897147
Ramachandran, Yu, Dyczynski, Baskaran, Zhang, Lulla, Lulla, Saul, Nelander, Dimberg, Merits, Leja-Jarblad, Essand: Safe and Effective Treatment of Experimental Neuroblastoma and Glioblastoma Using Systemically Delivered Triple MicroRNA-Detargeted Oncolytic Semliki Forest Virus. in Clinical cancer research : an official journal of the American Association for Cancer Research 2016
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Human Monoclonal IFNAR1 Primary Antibody for CyTOF, FACS - ABIN4900074
Zhou, Huang, Poon, Chen, Chan, Ng, Guan, Watt, Lu, Yuen, Zheng: Functional dissection of an IFN-alpha/beta receptor 1 promoter variant that confers higher risk to chronic hepatitis B virus infection. in Journal of hepatology 2009
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Human Monoclonal IFNAR1 Primary Antibody for CyTOF, FACS - ABIN4900073
Madrid, Ganem: Kaposi's sarcoma-associated herpesvirus ORF54/dUTPase downregulates a ligand for the NK activating receptor NKp44. in Journal of virology 2012
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Rat (Rattus) Polyclonal IFNAR1 Primary Antibody for IHC, WB - ABIN152597
Kumar, Krolewski, Fuchs: Phosphorylation and specific ubiquitin acceptor sites are required for ubiquitination and degradation of the IFNAR1 subunit of type I interferon receptor. in The Journal of biological chemistry 2004
Human Monoclonal IFNAR1 Primary Antibody for FACS - ABIN4897143
Fuchs, Kaiser-Labusch, Bank, Ammann, Kolb-Kokocinski, Edelbusch, Omran, Ehl: Tyrosine kinase 2 is not limiting human antiviral type III interferon responses. in European journal of immunology 2016
transfusion-induced differentiation of IFNAR1(-/-) B cells into germinal center B cells and plasma cells was significantly reduced, compared to WT B cells. This study demonstrates that B cells require signaling from IFN-alpha (show IFNA Antibodies)/beta to produce alloantibodies to the human KEL glycoprotein in mice.
These data suggest that plasmacytoid dendritic cells producing IFN-alpha (show IFNA Antibodies) and IL-33 (show IL33 Antibodies) play a pivotal role in the chronic fibro-inflammatory responses underlying murine autoimmune pancreatitis and human IgG4-related autoimmune pancreatitis.
type I interferons, besides their known antiviral properties, can initiate the recruitment and activation of leukocytes via induction of chemokine (show CCL1 Antibodies) expression including CCL2 (show CCL2 Antibodies).
this study shows that the presence of IFN-alpha (show IFNA Antibodies) at antigen sensitization activates an IDO1 (show IDO1 Antibodies)/TGF-beta (show TGFB1 Antibodies)-dependent anti-inflammatory program that upon antigenic rechallenge prevents inflammation via plasmacytoid dendritic cells
these studies demonstrate an important role for type I IFN in skin fibrosis, and they provide a rationale for IFNAR1 inhibition in scleroderma
These results identify a key interface created by IFNAR1 residues Tyr (show TYR Antibodies)(240) and Tyr (show TYR Antibodies)(274) interacting with IFN-beta (show IFNB1 Antibodies) residues Phe(63), Leu(64), Glu (show GCG Antibodies)(77), Thr (show TRH Antibodies)(78), Val(81), and Arg(82) that underlie IFN-beta (show IFNB1 Antibodies)-IFNAR1-mediated signaling and biological processes.
IFNAR1-deficiency accelerated humoral immune responses and parasite control by boosting ICOS (show ICOS Antibodies)-signalling in two non-lethal murine models of malaria
reduced type I interferon (show IFNA Antibodies) production in obesity is caused by SOCS3 (show SOCS3 Antibodies) overexpression as well as tolerance induced by leptin (show LEP Antibodies)
Downregulation of IFNAR1 promotes melanoma development and progression. IFNAR1 mutation, which is partially resistant to downregulation, delays melanoma development.
The study provides evidence for the importance of brain endothelial and epithelial cells in the communication between the Central Nervous System and the immune system, and demonstrates tissue specific IFNAR1 engagement during sickness behavior.
Low IFNAR1 expression is associated with peritoneal metastasis in gastric cancer.
the level of IFNAR1, IFNAR2, and CCR5 mRNA expression was found to be significantly lower in the responders than nonresponders.Our results highlighted the significance of IFNAR and CCR5 genes in multiple sclerosis risk and the response to IFN-b therapy.
Downregulation of IFNAR1 in tumor stroma stimulated colorectal cancer development and growth, played a key role in formation of the immune-privileged niche.
These results suggest that miR-29a, upregulated during RSV infection, is a negative regulator of IFNAR1 and is critical for respiratory syncytial virus NS1-induced virus replication.
On one hand, hepatitis B virus activates MMP-9 (show MMP9 Antibodies) in infected patients and leukocytes. On the other hand, MMP-9 (show MMP9 Antibodies) facilitates hepatitis B virus replication through repressing IFN/JAK (show JAK3 Antibodies)/STAT (show STAT1 Antibodies) signaling, IFNAR1 function, and IFN-alpha (show IFNA Antibodies) action.
this study shows that single nucleotide polymorphism in IFNAR1 gene is associated with female vitiligo (show MITF Antibodies) in Estonian patients
A small proportion of both pancreatic and periampullary tumors showed a strong expression of the IFNAR-1.
rs2843710 of IFNAR1 was associated with the susceptibility and severity of EV71 HFMD in Chinese Han populations.
Genetic polymorphisms of the promoter of INFAR gene represent important factors associated with the clinical phase of HBeAg-negative chronic HBV infection.
data illustrate a lipid G-protein coupled receptor (GPCR (show TAS1R3 Antibodies))-IFNAR1 regulatory loop that balances effective and detrimental immune responses and elevated endogenous S1PR1 (show S1PR1 Antibodies) signaling
Mutation of the IFNAR-1 receptor binding site of human IFN-alpha2 (show IFNA2 Antibodies) generates type I IFN competitive antagonists.
The complete 168,835 bp insert sequence of a porcine BAC clone harboring the IFNAR1 gene was determined.
The protein encoded by this gene is a type I membrane protein that forms one of the two chains of a receptor for interferons alpha and beta. Binding and activation of the receptor stimulates Janus protein kinases, which in turn phosphorylate several proteins, including STAT1 and STAT2. The encoded protein also functions as an antiviral factor.
interferon alpha/beta receptor 1
, interferon receptor
, soluble receptor
, interferon-alpha receptor 1
, putative interferon-alpha/beta receptor alpha chain
, interferon receptor 1
, interferon (alpha, beta and omega) receptor 1
, interferon alpha/beta receptor 1-like
, IFN-alpha/beta receptor 1
, INF-a receptor
, interferon-alpha/beta receptor 1
, type I interferon receptor 1
, alpha-type antiviral protein
, beta-type antiviral protein
, cytokine receptor class-II member 1
, cytokine receptor family 2 member 1
, interferon-alpha/beta receptor alpha chain
, interferon-beta receptor 1
, interferon (alpha and beta) receptor 1
, interferon, alpha; receptor
, Interferon-alpha/beta receptor alpha chain