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Human Oncostatin M ELISA Kit for Sandwich ELISA - ABIN414913
Lee, Kil, Kwon, Kim, Yoo, Shin: Oncostatin M as a target biological molecule of preeclampsia. in The journal of obstetrics and gynaecology research 2010
Mouse (Murine) Oncostatin M ELISA Kit for Sandwich ELISA - ABIN415531
Moidunny, Matos, Wesseling, Banerjee, Volsky, Cunha, Agostinho, Boddeke, Roy: Oncostatin M promotes excitotoxicity by inhibiting glutamate uptake in astrocytes: implications in HIV-associated neurotoxicity. in Journal of neuroinflammation 2016
Study showed that in atrial fibrillation (AF) with thrombus, the atrial tissue infiltration of M1 macrophages increased significantly; the OSM expression was also found to increase simultaneously; downstream tissue factor (show F3 ELISA Kits) (TF) increased and tissue factor (show F3 ELISA Kits) pathway inhibitors (TFPI (show TFPI ELISA Kits))decreased, leading to an imbalance between TF and TFPI (show TFPI ELISA Kits) eventually. OSM might be related to thrombosis in patients with AF mediated by TF and TFPI (show TFPI ELISA Kits)
a novel STAT3 (show STAT3 ELISA Kits)/SMAD3 (show SMAD3 ELISA Kits)-signaling axis is required for OSM-mediated senescence.
This result demonstrates that HPV16 oncoproteins upregulate oncostatin M and play an important role to promote oral squamous cell carcinoma development
The identification of the OSM inflammatory pathway as an important mediator of epithelial mesenchymal transition in triple-negative breast cancer (TNBC) may provide a novel potential opportunity to improve therapeutic strategies.
Nucleolin (show NCL ELISA Kits) stabilizes oncostatin-M mRNA by binding to a GC-rich (show RELB ELISA Kits) element in its 3'UTR (show UTS2R ELISA Kits).
Oncostatin M and interleukin-31 (show IL31 ELISA Kits): Cytokines, receptors, signal transduction and physiology.
Oncostatin M can regulate airway smooth muscle responses alone or in synergy with IL-17A (show IL17A ELISA Kits).
we demonstrated that recombinant human OSM (rhOSM) promoted tumor angiogenesis in EC cell lines by activating STAT3 (signal transducer and activator of transcription 3 (show STAT3 ELISA Kits)) and enhanced both cell migration and cell inva
OSM expression in osteoblasts increases in response to Osteopontin (show SPP1 ELISA Kits)-induced inflammation in vitro.
Data suggest that OSM promotes osteoblastic differentiation of vascular smooth muscle cells through JAK3 (show JAK3 ELISA Kits)/STAT3 (show STAT3 ELISA Kits) pathway and may contribute to the development of atherosclerotic calcification.
these results support the proinflammatory role of OSM when it is overexpressed in the skin. However, OSM expression was not required in the murine model of psoriasis induced by topical application of imiquimod, as demonstrated by the inflammatory phenotype of OSM-deficient mice or wild-type mice treated with anti-OSM antibodies.
OSM (mOSM) signals mainly via an OSM receptor (OSMR (show OSMR ELISA Kits))-gp130 (show LRPPRC ELISA Kits) heterodimer and binds with only very low affinity to mLIFR.
Loss of Oncostatin M Signaling in Adipocytes Induces Insulin (show INS ELISA Kits) Resistance and Adipose Tissue Inflammation in Vivo.
mechanism of OSM-induced cardiomyocyte dedifferentiation is associated with B-Raf (show SNRPE ELISA Kits)/Mek (show MDK ELISA Kits)/Erk (show EPHB2 ELISA Kits) signaling pathway through the OSM receptor Obeta
OSM plays multiple critical roles in the maintenance and development of the hematopoietic microenvironment in the bone marrow at a steady state as well as after injury.
OSM treatment preserved cardiac function, inhibited apoptosis and fibrosis, and stimulated angiogenesis via upregulating VEGF (show VEGFA ELISA Kits) and bFGF (show FGF2 ELISA Kits) in infarct border zone of ischemic myocardium, indicating that OSM could be a novel therapeutic target for MI.
The mechanism of Oncostatin M on cardiac ischemia/reperfusion injury is partly mediated by the Notch3 (show NOTCH3 ELISA Kits)/Akt (show AKT1 ELISA Kits) pathway.
OSM-induced osteogenesis and upregulation of osteogenic gene products require activity of PKCdelta (show PKCd ELISA Kits).
The Notch (show NOTCH1 ELISA Kits) signaling pathway was found to be one of the signaling pathways that inhibit OSM-induced MC3T3-E1 cell proliferation and differentiation.
Protein kinase R plays a pivotal role in oncostatin M and interleukin-1 signalling in bovine articular cartilage chondrocytes.
Oncostatin M is a member of a cytokine family that includes leukemia-inhibitory factor, granulocyte colony-stimulating factor, and interleukin 6. This gene encodes a growth regulator which inhibits the proliferation of a number of tumor cell lines. It regulates cytokine production, including IL-6, G-CSF and GM-CSF from endothelial cells.