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Human Oncostatin M Primary Antibody for ELISA - ABIN414913
Lee, Kil, Kwon, Kim, Yoo, Shin: Oncostatin M as a target biological molecule of preeclampsia. in The journal of obstetrics and gynaecology research 2010
Mouse (Murine) Oncostatin M Primary Antibody for ELISA - ABIN415531
Moidunny, Matos, Wesseling, Banerjee, Volsky, Cunha, Agostinho, Boddeke, Roy: Oncostatin M promotes excitotoxicity by inhibiting glutamate uptake in astrocytes: implications in HIV-associated neurotoxicity. in Journal of neuroinflammation 2016
This result demonstrates that HPV16 oncoproteins upregulate oncostatin M and play an important role to promote oral squamous cell carcinoma development
The identification of the OSM inflammatory pathway as an important mediator of epithelial mesenchymal transition in triple-negative breast cancer (TNBC) may provide a novel potential opportunity to improve therapeutic strategies.
Nucleolin (show NCL ELISA Kits) stabilizes oncostatin-M mRNA by binding to a GC-rich (show RELB ELISA Kits) element in its 3'UTR (show UTS2R ELISA Kits).
Oncostatin M and interleukin-31 (show IL31 ELISA Kits): Cytokines, receptors, signal transduction and physiology.
Oncostatin M can regulate airway smooth muscle responses alone or in synergy with IL-17A (show IL17A ELISA Kits).
we demonstrated that recombinant human OSM (rhOSM) promoted tumor angiogenesis in EC cell lines by activating STAT3 (signal transducer and activator of transcription 3 (show STAT3 ELISA Kits)) and enhanced both cell migration and cell inva
OSM expression in osteoblasts increases in response to Osteopontin (show SPP1 ELISA Kits)-induced inflammation in vitro.
Data suggest that OSM promotes osteoblastic differentiation of vascular smooth muscle cells through JAK3 (show JAK3 ELISA Kits)/STAT3 (show STAT3 ELISA Kits) pathway and may contribute to the development of atherosclerotic calcification.
The Notch (show NOTCH1 ELISA Kits) signaling pathway was found to be one of the signaling pathways that inhibit OSM-induced MC3T3-E1 cell proliferation and differentiation.
administration of Fstl1 (show FSTL1 ELISA Kits) induced airway remodeling and increased OSM, whereas administration of an anti-OSM Ab blocked the effect of Fstl1 (show FSTL1 ELISA Kits) on inducing airway remodeling, eosinophilic airway inflammation
Loss of Oncostatin M Signaling in Adipocytes Induces Insulin (show INS ELISA Kits) Resistance and Adipose Tissue Inflammation in Vivo.
mechanism of OSM-induced cardiomyocyte dedifferentiation is associated with B-Raf (show SNRPE ELISA Kits)/Mek (show MDK ELISA Kits)/Erk (show EPHB2 ELISA Kits) signaling pathway through the OSM receptor Obeta
OSM plays multiple critical roles in the maintenance and development of the hematopoietic microenvironment in the bone marrow at a steady state as well as after injury.
OSM treatment preserved cardiac function, inhibited apoptosis and fibrosis, and stimulated angiogenesis via upregulating VEGF (show VEGFA ELISA Kits) and bFGF (show FGF2 ELISA Kits) in infarct border zone of ischemic myocardium, indicating that OSM could be a novel therapeutic target for MI.
The mechanism of Oncostatin M on cardiac ischemia/reperfusion injury is partly mediated by the Notch3 (show NOTCH3 ELISA Kits)/Akt (show AKT1 ELISA Kits) pathway.
OSM-induced osteogenesis and upregulation of osteogenic gene products require activity of PKCdelta (show PKCd ELISA Kits).
A novel role for OSM during pneumonia as an important signal to epithelial cells for chemokine (show CCL1 ELISA Kits) induction mediating neutrophil recruitment.
Protein kinase R plays a pivotal role in oncostatin M and interleukin-1 signalling in bovine articular cartilage chondrocytes.
Oncostatin M is a member of a cytokine family that includes leukemia-inhibitory factor, granulocyte colony-stimulating factor, and interleukin 6. This gene encodes a growth regulator which inhibits the proliferation of a number of tumor cell lines. It regulates cytokine production, including IL-6, G-CSF and GM-CSF from endothelial cells.