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Human Oncostatin M Primary Antibody for FACS - ABIN2666535
Beigel, Friedrich, Probst, Sotlar, Göke, Diegelmann, Brand: Oncostatin M mediates STAT3-dependent intestinal epithelial restitution via increased cell proliferation, decreased apoptosis and upregulation of SERPIN family members. in PLoS ONE 2014
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Human Oncostatin M Primary Antibody for WB - ABIN2667567
Chen, Benveniste: Oncostatin M: a pleiotropic cytokine in the central nervous system. in Cytokine & growth factor reviews 2004
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Human Oncostatin M Primary Antibody for - ABIN2181568
Tanaka, Miyajima: Oncostatin M, a multifunctional cytokine. in Reviews of physiology, biochemistry and pharmacology 2003
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Human Oncostatin M Primary Antibody for Func, SDS - ABIN2039658
Yin, Chain: Suppression of lymphokine production in anti-minor histocompatibility antigen responses. in Cytokine 1991
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Human Oncostatin M Primary Antibody for HPLC, SDS - ABIN413690
Sen, Che, Rajamani, Zerboni, Sung, Ptacek, Arvin: Signal transducer and activator of transcription 3 (STAT3) and survivin induction by varicella-zoster virus promote replication and skin pathogenesis. in Proceedings of the National Academy of Sciences of the United States of America 2012
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Human Oncostatin M Primary Antibody for SDS - ABIN667495
Nair, DeVico, Nakamura, Copeland, Chen, Patel, ONeil, Oroszlan, Gallo, Sarngadharan: Identification of a major growth factor for AIDS-Kaposi's sarcoma cells as oncostatin M. in Science (New York, N.Y.) 1992
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This result demonstrates that HPV16 oncoproteins upregulate oncostatin M and play an important role to promote oral squamous cell carcinoma development
The identification of the OSM inflammatory pathway as an important mediator of epithelial mesenchymal transition in triple-negative breast cancer (TNBC) may provide a novel potential opportunity to improve therapeutic strategies.
Nucleolin stabilizes oncostatin-M mRNA by binding to a GC-rich element in its 3'UTR.
Oncostatin M and interleukin-31 (show IL31 Proteins): Cytokines, receptors, signal transduction and physiology.
Oncostatin M can regulate airway smooth muscle responses alone or in synergy with IL-17A (show IL17A Proteins).
we demonstrated that recombinant human OSM (rhOSM) promoted tumor angiogenesis in EC cell lines by activating STAT3 (signal transducer and activator of transcription 3 (show STAT3 Proteins)) and enhanced both cell migration and cell inva
OSM expression in osteoblasts increases in response to Osteopontin (show SPP1 Proteins)-induced inflammation in vitro.
Data suggest that OSM promotes osteoblastic differentiation of vascular smooth muscle cells through JAK3 (show JAK3 Proteins)/STAT3 (show STAT3 Proteins) pathway and may contribute to the development of atherosclerotic calcification.
The Notch (show NOTCH1 Proteins) signaling pathway was found to be one of the signaling pathways that inhibit OSM-induced MC3T3-E1 cell proliferation and differentiation.
administration of Fstl1 (show FSTL1 Proteins) induced airway remodeling and increased OSM, whereas administration of an anti-OSM Ab blocked the effect of Fstl1 (show FSTL1 Proteins) on inducing airway remodeling, eosinophilic airway inflammation
Loss of Oncostatin M Signaling in Adipocytes Induces Insulin (show INS Proteins) Resistance and Adipose Tissue Inflammation in Vivo.
mechanism of OSM-induced cardiomyocyte dedifferentiation is associated with B-Raf (show SNRPE Proteins)/Mek (show MDK Proteins)/Erk (show EPHB2 Proteins) signaling pathway through the OSM receptor Obeta
OSM plays multiple critical roles in the maintenance and development of the hematopoietic microenvironment in the bone marrow at a steady state as well as after injury.
OSM treatment preserved cardiac function, inhibited apoptosis and fibrosis, and stimulated angiogenesis via upregulating VEGF (show VEGFA Proteins) and bFGF (show FGF2 Proteins) in infarct border zone of ischemic myocardium, indicating that OSM could be a novel therapeutic target for MI.
The mechanism of Oncostatin M on cardiac ischemia/reperfusion injury is partly mediated by the Notch3 (show NOTCH3 Proteins)/Akt (show AKT1 Proteins) pathway.
OSM-induced osteogenesis and upregulation of osteogenic gene products require activity of PKCdelta (show PKCd Proteins).
A novel role for OSM during pneumonia as an important signal to epithelial cells for chemokine (show CCL1 Proteins) induction mediating neutrophil recruitment.
Protein kinase R plays a pivotal role in oncostatin M and interleukin-1 signalling in bovine articular cartilage chondrocytes.
Oncostatin M is a member of a cytokine family that includes leukemia-inhibitory factor, granulocyte colony-stimulating factor, and interleukin 6. This gene encodes a growth regulator which inhibits the proliferation of a number of tumor cell lines. It regulates cytokine production, including IL-6, G-CSF and GM-CSF from endothelial cells.