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Rat (Rattus) STAT5B ELISA Kit for Sandwich ELISA - ABIN1117115
Sun, Wang, Yan, Lu, Shi: Effects of Er-Zhi-Wan on microarchitecture and regulation of Wnt/?-catenin signaling pathway in alveolar bone of ovariectomized rats. in Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban 2014
This study indicates that CISH (show CISH ELISA Kits) functions as a conserved in vivo target and regulator of STAT5 in the control of embryonic hematopoiesis.
study demonstrates that STAT5 in basophils is activated through both the IL-3 (show IL-3 ELISA Kits) and the FcepsilonRI (show FCER1G ELISA Kits) signaling pathway
Studies demonstrate a conserved role for SOCS1 (show SOCS1 ELISA Kits) in T cell development and suggest a novel T cell-independent function in embryonic myelopoiesis mediated, at least in part, via its effects on receptors using the Jak2 (show JAK2 ELISA Kits)-Stat5 pathway.
The stat5.1 gene lies next to the stat3 (show STAT3 ELISA Kits) gene.
This is the first report of a survival disadvantage of EBV+ patients with CLL, and the first time that STAT5b expression is correlated with survival. The correlation of STAT5 (show STAT5A ELISA Kits) expression with the presence of the virus, along with our survival correlations defines a subgroup of patients with CLL that may benefit from anti-STAT (show STAT1 ELISA Kits) agents.
STAT5 (show STAT5A ELISA Kits) signaling axis drives abnormal cell proliferation in autosomal dominant polycystic kidney disease.
p65 (show GORASP1 ELISA Kits) activation triggered by TCR signaling could promote SNRPA (show SNRPA ELISA Kits) transcription and 3' UTR (show UTS2R ELISA Kits) shortening of STAT5B. Thus we propose that the alternative polyadenylation switching of STAT5B induced by TCR activation is mediated by SNRPA (show SNRPA ELISA Kits).
this study shows that musculin (show MSC ELISA Kits) inhibits human T-helper 17 cell response to interleukin 2 (show IL2 ELISA Kits) by controlling STAT5B activity
Molecular interactions of EphA4 (show EPHA4 ELISA Kits), growth hormone receptor (show GHR ELISA Kits), Jak2 (show JAK2 ELISA Kits), and STAT5B have been described.
All merkel cell carcinoma (MCC) cases with a good outcome expressed pSTAT5B, whereas all MCC cases with a poor outcome did not express pSTAT5B; hence, pSTAT5B expression may be an indicator of positive prognosis in patients with MCC.
Data show that signal transducer and activator of transcription 5 STAT5A (show STAT5A ELISA Kits) and STAT5B have distinctive and potentially important roles in regulation of hepatic drug response genes as well as in contributing to gender-related differences in expression of hepatic cytochrome P450s (CYPs) and transcription factors (TFs).
STAT5b confers gemcitabine chemoresistance and promotes cell adherence and invasiveness in pancreatic cancer cells.
STAT5 (show STAT5A ELISA Kits) activation by EGF (show EGF ELISA Kits) constitutes an important cascade for the regulation of cell proliferation and invasion in trophoblast cells.
MSM decreased the ability of STAT5b to bind the promoter of the HER2 (show ERBB2 ELISA Kits) gene.
A study of the polymorphisms of STAT5b is presented, and the use of radiation hybrid mapping to localize the gene to chromosome 12 is presented.
SNPs in JAK2 (show JAK2 ELISA Kits) and STAT5B are associated with mastitis susceptibility in Chinese Holstein cattle
This study suggests that growth hormone (show GH1 ELISA Kits) inhibits the differentiation of bovine preadipocytes to adipocytes through STAT5b-dependent inhibition of C/EBPalpha (show CEBPA ELISA Kits) and PPARgamma (show PPARG ELISA Kits) expression.
These results suggested that the SNPs in CD4 (show CD4 ELISA Kits) and STAT5b may be potential genetic markers for SCS (show TWIST1 ELISA Kits) and milk/protein (show CSN2 ELISA Kits) yields selecting and warrant further functional research.
Instead, we find that STAT5B is dominant over STAT5A (show STAT5A ELISA Kits) in CD4 (show CD4 ELISA Kits)(+) 'helper' T cells for both effector and regulatory (Treg) responses and, therefore, uniquely necessary for immunological tolerance.
These studies establish the importance of STAT5 (show STAT5A ELISA Kits) in macrophages during ductal morphogenesis in the mammary gland and demonstrate that altering STAT5 (show STAT5A ELISA Kits) function in macrophages can affect the development of tissue-specific disease.
Data indicate that 89% of signal transducer and activator of transcription 5 (show STAT5A ELISA Kits) (Stat5b)-CA mice in which the T2/Onc transposon had been mobilized died of progenitor B-cell acute lymphoblastic leukemia (B-ALL) by 3 months of age.
STAT5B and MOF (show KAT8 ELISA Kits) work as negative regulators in adipogenesis.
Strong selective advantage for leukemic transformation in the background of Stat5 (show STAT5A ELISA Kits) deficient hematopoiesis was permissive for faster initiation of Myc (show MYC ELISA Kits)-induced transformation to B (show TDO2 ELISA Kits)-ALL.
liver STAT5b activation/masculinization occurred at puberty and suppression/feminization occurred during aging and in mutant mice with defects in growth hormone signaling.
Loss of STAT5b had no effect on either sex.
These data provide the first definitive evidence for a contribution of STAT5a (show STAT5A ELISA Kits)/b to the sex bias in pulmonary hypertension in the hypoxic mouse and implicate reduced STAT5 (show STAT5A ELISA Kits) in the pathogenesis of the human disease.
Data show that three hypotheses of dimer formation between STAT5 (show STAT5A ELISA Kits) transcription factors STAT5A (show STAT5A ELISA Kits) and STAT5B were used to explain the analytical results by a static mathematical model.
The protein encoded by this gene is a member of the STAT family of transcription factors. In response to cytokines and growth factors, STAT family members are phosphorylated by the receptor associated kinases, and then form homo- or heterodimers that translocate to the cell nucleus where they act as transcription activators. This protein mediates the signal transduction triggered by various cell ligands, such as IL2, IL4, CSF1, and different growth hormones. It has been shown to be involved in diverse biological processes, such as TCR signaling, apoptosis, adult mammary gland development, and sexual dimorphism of liver gene expression. This gene was found to fuse to retinoic acid receptor-alpha (RARA) gene in a small subset of acute promyelocytic leukemias (APLL). The dysregulation of the signaling pathways mediated by this protein may be the cause of the APLL.
transcription factor STAT5B
, Signal transducer and activator of transcription 5a
, mammary gland factor STAT5B
, signal transducer and activator of transcription 5B
, signal transducer and activator of transcription 5A
, Signal transducer and activator of transcription 5B