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Results identify a key molecular mechanism by which the BTG2-PRMT1 (show PRMT1 ELISA Kits) module regulates pre-B cell differentiation and inhibits pre-B cell leukemogenesis.
Results suggest that Btg2 is an anti-adipogenic factor and its expression is controlled by the Stat3 (show STAT3 ELISA Kits) signaling pathway.
these data show that both Btg1 (show BTG1 ELISA Kits) and Btg2 are required for normal vertebral patterning of the axial skeleton, but each gene contributes differently in specifying the identity along the anterior-posterior axis of the skeleton.
Btg2 overexpression in vivo and in vitro induced all the observed changes during interdigit regression, including oxidative stress, arrest of cell cycle progression, transcriptional regulation of senescence markers, and caspase (show CASP3 ELISA Kits)-mediated apoptosis.
study provides a novel molecular mechanism of BTG2-mediated induction of hepcidin (show HAMP ELISA Kits) gene expression, thereby contributing to a better understanding of the hepatic hepcidin (show HAMP ELISA Kits) production involved in iron homeostasis
TIS21 is required for development of spiral ganglion cells
we show that the putative tumor suppressor gene Btg2 is consistently downregulated in high grade gliomas and that its downregulation is necessary for a cell to maintain the malignant phenotype.
Mice lacking the Tis21 3' UTR (show UTS2R ELISA Kits) develop a microcephalic neocortex with fewer neurons.
BTG2 regulates hepatic glucose homeostasis via induction of orphan nuclear receptor (show NR1D1 ELISA Kits) Nur77 (show NR4A1 ELISA Kits) in diabetic mouse model.
pro-apoptotic pathway of CRP (show CRP ELISA Kits)-CD32-Nox2 (show CYBB ELISA Kits)-p53 (show TP53 ELISA Kits)-BTG2 may contribute to the retardation of the atherogenic process
Effect of TIS21 on the regulation of p53 (show TP53 ELISA Kits) activity was confirmed by knockdown of TIS21 expression by RNA interference.
TIS21 attenuated Doxorubicin-induced cancer cell senescence by inhibiting linear actin nucleation via Nox4 (show NOX4 ELISA Kits)-ROS (show ROS1 ELISA Kits)-ABI2-DRF (show MPO ELISA Kits) signal cascade
Data indicate that BTG2, MAP3K11 (show MAP3K11 ELISA Kits), RPS6KA1 (show RPS6KA1 ELISA Kits) and PRDM1 (show PRDM1 ELISA Kits) as putative targets of microRNA miR (show MLXIP ELISA Kits)-125b.
The results indicated that BTG2 expression was downregulated in skin cancer cell lines. Overexpression of BTG2 significantly inhibited cell proliferation, cell cycle progression, and the invasion and migration of skin cancer cells.
Overexpression of BTG2 inhibited the proliferation and migration/invasion of human osteosarcoma cells in vitro
clinical data support conclusions generated from patient-derived xenograft models and indicate that BTG2 expression is a candidate prognostic biomarker for TNBC.
miR (show MLXIP ELISA Kits)-21 was found to be overexpressed and BTG2 was found to to be downregulated in HepG2 liver cancer cells.
BTG2 stimulates mRNA deadenylation via CAF1 (show CHAF1B ELISA Kits) activation through interaction with PABPC1 (show PABPC1 ELISA Kits). Interaction of BTG2 with the first RRM domain of PABPC1 (show PABPC1 ELISA Kits) is required for BTG2 to control cell proliferation.
SETD1A (show SETD1A ELISA Kits) suppresses BTG2 expression through its induction of several BTG2-targeting miRNAs.
Results indicate that miR27a and BTG2 promote the migration and the growth of glioblastoma (GBM) cells and miR27a directly targets BTG2 in GBM cells.
The full length sequences of cDNA and genomic DNA of BTG2 gene from the porcine skeletal muscle, was cloned.
The protein encoded by this gene is a member of the BTG/Tob family. This family has structurally related proteins that appear to have antiproliferative properties. This encoded protein is involved in the regulation of the G1/S transition of the cell cycle.
B-cell translocation gene 2, anti-proliferative
, BTG family, member 2
, protein BTG2
, B-cell translocation gene 2
, B cell translocation protein 2
, BTG family member 2
, NGF-inducible protein TIS21
, NGF-inducible anti-proliferative protein PC3
, nerve growth factor-inducible anti-proliferative
, pheochromacytoma cell-3
, B-cell translocation protein 2
, Early induced gene B-cell translocation gene 2
, NGF-inducible anti-proliferative putative secreted protein
, cell surface alloantigen