Browse our C-JUN (JUN) ELISA Kits

On www.antibodies-online.com are 102 Jun Proto-Oncogene (JUN) ELISA Kits from 15 different suppliers available. Additionally we are shipping C-JUN Antibodies (876) and C-JUN Proteins (24) and many more products for this protein. A total of 1063 C-JUN products are currently listed.
Synonyms:
AP-1, AP1, B230310J22Rik, Bsk, BSK/DJNK, c-jun, CG2275, CG5680, cJun, D-JNK, d-JRA, d-jun, D-jun/Jra, D-junk, dAP-1, DBSK/JNK, dJNK, DJNK/bsk, dJra, djun, dm-Jun, Dmel\\CG2275, Dmel\\CG5680, fj36h07, jnk, JNK/SAPK, jra, jun, Junc, Junk, l(2)46Ef, l(2)IA109, l(2R)IA109, p39, SAPKa, V, wu:fj36h07, zgc:65863
list all ELISA KIts Gene Name GeneID UniProt
JUN 12570 O35926
JUN 3725 P05412
JUN 24516 P17325

C-JUN (JUN) ELISA Kits by Reactivity

More ELISA Kits for C-JUN Interaction Partners

Xenopus laevis Jun Proto-Oncogene (JUN) interaction partners

  1. AP-1(c-Jun/FosB (show FOSB ELISA Kits)) may play a role in neurogenesis via the induction of FoxD5b expression during early vertebrate development

  2. The cJun transcription factor bound to a variant cAMP response element in the promoter region of tlx3 (show TLX3 ELISA Kits) and modulated transcription and regulated neurotransmitter phenotype via its transactivation domain

Fruit Fly (Drosophila melanogaster) Jun Proto-Oncogene (JUN) interaction partners

  1. Mir (show MYLIP ELISA Kits)-8 modulates Drosophila C virus replication by negative regulation of dJun.

  2. Here we uncover a cell non-autonomous requirement for the Epidermal growth factor receptor (Egfr (show EGFR ELISA Kits)) pathway in the lateral epidermis for sustained dpp (show TGFb ELISA Kits) expression in the LE. Specifically, we demonstrate that Egfr (show EGFR ELISA Kits) pathway activity in the lateral epidermis prevents expression of the gene scarface (scaf), encoding a secreted antagonist of JNK (show MAPK8 ELISA Kits) signaling

  3. Tau and spectraplakin promote synapse formation and maintenance through Jun kinase (show MAPK9 ELISA Kits) and neuronal trafficking.

  4. n addition to significantly increasing the number of JNK (show MAPK8 ELISA Kits) target genes identified so far, our results reveal that the LE is a highly heterogeneous morphogenetic organizer, sculpted through crosstalk between JNK (show MAPK8 ELISA Kits), segmental and AP signalling. This fine-tuning regulatory mechanism is essential to coordinate morphogenesis and dynamics of tissue sealing

  5. malignant transformation of the ras(V12)scrib(1 (show SCRIB ELISA Kits)) tumors requires bZIP protein Fos, the ETS (show ETS1 ELISA Kits)-domain factor Ets21c and the nuclear receptor Ftz-F1 (show NR5A2 ELISA Kits), all acting downstream of Jun-N-terminal kinase (show MAPK8 ELISA Kits).

  6. Diminished MTORC1-dependent JNK (show MAPK8 ELISA Kits) activation underlies the neurodevelopmental defects associated with lysosomal dysfunction.

  7. ROS (show ROS1 ELISA Kits)/JNK (show MAPK8 ELISA Kits)/p38 (show MAPK14 ELISA Kits)/Upd (show UROD ELISA Kits) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  8. Significantly, the JNK (show MAPK8 ELISA Kits) pathway is responsible for the majority of the phenotypes and transcriptional changes downstream of Notch (show NOTCH1 ELISA Kits)-Src (show SRC ELISA Kits) synergy.

  9. This study demonstrated that the mechanism by which Bsk (show FRK ELISA Kits) is required for pruning is through reducing the membrane levels of the adhesion molecule (show NCAM1 ELISA Kits) Fasciclin II (show NCAM2 ELISA Kits) (FasII)

  10. Jra recruits the HP1a (show CBX5 ELISA Kits)/KDM4A (show KDM4A ELISA Kits) complex to its gene body region upon osmotic stress to reduce H3K36 methylation levels and disrupt H3K36 methylation-dependent histone deacetylation

Pig (Porcine) Jun Proto-Oncogene (JUN) interaction partners

  1. Porcine reproductive and respiratory syndrome virus -activated TAK-1 (show NR2C2 ELISA Kits) was essential for the activation of JNK (show MAPK8 ELISA Kits) and NF-kappaB (show NFKB1 ELISA Kits) pathways and IL-8 (show IL8 ELISA Kits) expression.

  2. The effects of prostaglandin F2alpha administration on transcription factor AP-1 expression and the expression of downstream genes involved in luteolysis are reported.

Rabbit Jun Proto-Oncogene (JUN) interaction partners

  1. ICAM1 (show ICAM1 ELISA Kits) and IL10 (show IL10 ELISA Kits) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 may be responsible for this upregulation.

Mouse (Murine) Jun Proto-Oncogene (JUN) interaction partners

  1. AP1 factors are important regulators of adult taste cell renewal and their downregulation negatively impacts taste maintenance.

  2. these data indicate that MEF2 (show MEF2C ELISA Kits) and AP-1 confer antagonistic regulation of Hspb7 (show HSPB7 ELISA Kits) gene expression in skeletal muscle, with implications for autophagy and muscle atrophy.

  3. results suggest that fibroblasts, c-Jun, and IGF-1 (show IGF1 ELISA Kits) play key roles in mediating stromal-epithelial interactions that are required for the therapeutic effects of finasteride in benign prostate epithelial cells

  4. These findings highlight a key role of the TLR4 (show TLR4 ELISA Kits)-NOS1 (show NOS1 ELISA Kits)-AP1 signaling axis in regulating macrophage polarization

  5. Data suggest that Sf1 and c-jun interact and cooperate to activate the Fdx1 promoter in MA-10 (tumorigenic cell line) and TM3 (non-tumorigenic cell line) Leydig cells; such activation requires different regulatory elements located between -124 and -306 bp of Fdx1 promoter and involves recruitment of Sf1 to this region. (Sf1 = splicing factor 1; c-jun = proto-oncogene c-jun; Fdx1 = ferredoxin 1)

  6. In this study, we discovered that selective upregulation of p39 (show ATP6V0D1 ELISA Kits) is the underlying mechanism that accommodates the increased functional requirement of Cdk5 (show CDK5 ELISA Kits) activation during neuronal differentiation. In addition, we demonstrated that p39 (show ATP6V0D1 ELISA Kits) selectively directs Cdk5 (show CDK5 ELISA Kits) to phosphorylate protein substrates essential for axonal development, dendritic spine formation, and synaptogenesis. Moreover, our studies suggest opposing roles o

  7. NF-kappaB (show NFKB1 ELISA Kits) and c-Jun coregulate lipopolysaccharide-induced Fra-1 (show FOSL1 ELISA Kits) transcription.

  8. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  9. this study shows that c-Jun regulates the activation state of macrophages and promotes arthritis via differentially regulating cyclooxygenase-2 (show PTGS2 ELISA Kits) and arginase-1 (show ARG1 ELISA Kits) levels

  10. PLIO treatment inhibited nuclear factorkappaB (NFkappaB) nuclear translocation in B16F10 cells. In addition, PLIO treatment inhibited the phosphorylation of c-Jun Nterminal kinases and AKT (show AKT1 ELISA Kits)

Human Jun Proto-Oncogene (JUN) interaction partners

  1. Our results indicate that assessing AP1 (show FOSB ELISA Kits) and PEA3 (show ETV4 ELISA Kits) transcription factor status might be a good indicator of OAC status. However, we could not detect any associations with disease stage or patient treatment regime. This suggests that the PEA3 (show ETV4 ELISA Kits)-AP1 (show FOSB ELISA Kits) regulatory module more likely contributes more generally to the cancer phenotype. In keeping with this observation, depletion of ETV1 (show ETV1 ELISA Kits) and/or ETV4 (show ETV4 ELISA Kits) causes an OAC cell growth defect

  2. shRNA-mediated inhibition of JUN decreases AML (show RUNX1 ELISA Kits) cell survival and propagation in vivo. These data uncover a previously unrecognized role of JUN as a regulator of the unfolded protein response

  3. These findings demonstrate an essential role for the ERK pathway together with c-JUN and c-FOS in the differentiation activity of LukS-PV.

  4. The present study defines the minimal TIM-3 (show HAVCR2 ELISA Kits) promoter region and demonstrates its interaction with c-Jun during TIM-3 (show HAVCR2 ELISA Kits) transcription in CD4 (show CD4 ELISA Kits)(+) T cells.

  5. Taken together, our data demonstrate that JNK (show MAPK8 ELISA Kits) regulates triple-negative breast cancer (TNBC)tumorigenesis by promoting CSC phenotype through Notch1 (show NOTCH1 ELISA Kits) signaling via activation of c-Jun and indicate that JNK/c-Jun/Notch1 (show NOTCH1 ELISA Kits) signaling is a potential therapeutic target for TNBC

  6. Regulation of osteosarcoma cell lung metastasis by the c-Fos/AP-1 target FGFR1

  7. c-jun promoted FOXK1 (show FOXK2 ELISA Kits)-mediated proliferation and metastasis via orthotopic implantation.

  8. Data provide evidence that AP-1 (show FOSB ELISA Kits) is a key determinant of endocrine resistance of breast cancer cells by mediating a global shift in the estrogen receptor (show ESR1 ELISA Kits) transcriptional program.

  9. Comparison of how AP-1 (Jun/Jun dimer) and Epstein-Barr virus Zta recognize methyl groups within their cognate response elements

  10. this study revealed the functional and mechanistic links between CDK5 (show CDK5 ELISA Kits) and the oncogenic ERK5 (show MAPK7 ELISA Kits)-AP-1 (show FOSB ELISA Kits) signaling pathway in the pathogenesis of colorectal cancer.

Zebrafish Jun Proto-Oncogene (JUN) interaction partners

  1. These results support a role for trim69 (show TRIM69 ELISA Kits) in the development of the zebrafish brain through ap-1 pathway.

  2. CPEB-1 (show CPEB1 ELISA Kits) control of c-Jun mRNA translation regulates GH gene expression and resulting downstream signaling events (e.g., synaptic plasticity) in the mouse hippocampus.

Cow (Bovine) Jun Proto-Oncogene (JUN) interaction partners

  1. The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 DNA-binding activity and expression of several transcriptions factors in breast cancer cells.

  2. dynamic compression stimulates cell proliferation and proteoglycan (show Vcan ELISA Kits) synthesis in the presence of IL-1beta (show IL1B ELISA Kits) and/or inhibitors of the MAPKs and NFkappaB and AP-1 signalling pathways

C-JUN (JUN) Antigen Profile

Antigen Summary

This gene is the putative transforming gene of avian sarcoma virus 17. It encodes a protein which is highly similar to the viral protein, and which interacts directly with specific target DNA sequences to regulate gene expression. This gene is intronless and is mapped to 1p32-p31, a chromosomal region involved in both translocations and deletions in human malignancies.

Alternative names and synonyms associated with C-JUN (JUN)

  • jun oncogene (jun) Elisa Kit
  • basket (bsk) Elisa Kit
  • C-JUN protein (C-JUN) Elisa Kit
  • c-Jun protein (C-JUN) Elisa Kit
  • c-jun transcription factor (C-JUN) Elisa Kit
  • cyclin-dependent kinase 5, regulatory subunit 2 (p39) (Cdk5r2) Elisa Kit
  • Jun oncogene (Jun) Elisa Kit
  • jun proto-oncogene (JUN) Elisa Kit
  • jun proto-oncogene (jun) Elisa Kit
  • jun proto-oncogene (Jun) Elisa Kit
  • Jun-related antigen (Jra) Elisa Kit
  • AP-1 Elisa Kit
  • AP1 Elisa Kit
  • B230310J22Rik Elisa Kit
  • Bsk Elisa Kit
  • BSK/DJNK Elisa Kit
  • c-jun Elisa Kit
  • CG2275 Elisa Kit
  • CG5680 Elisa Kit
  • cJun Elisa Kit
  • D-JNK Elisa Kit
  • d-JRA Elisa Kit
  • d-jun Elisa Kit
  • D-jun/Jra Elisa Kit
  • D-junk Elisa Kit
  • dAP-1 Elisa Kit
  • DBSK/JNK Elisa Kit
  • dJNK Elisa Kit
  • DJNK/bsk Elisa Kit
  • dJra Elisa Kit
  • djun Elisa Kit
  • dm-Jun Elisa Kit
  • Dmel\\CG2275 Elisa Kit
  • Dmel\\CG5680 Elisa Kit
  • fj36h07 Elisa Kit
  • jnk Elisa Kit
  • JNK/SAPK Elisa Kit
  • jra Elisa Kit
  • jun Elisa Kit
  • Junc Elisa Kit
  • Junk Elisa Kit
  • l(2)46Ef Elisa Kit
  • l(2)IA109 Elisa Kit
  • l(2R)IA109 Elisa Kit
  • p39 Elisa Kit
  • SAPKa Elisa Kit
  • V Elisa Kit
  • wu:fj36h07 Elisa Kit
  • zgc:65863 Elisa Kit

Protein level used designations for Jun Proto-Oncogene (JUN) ELISA Kits

v-jun sarcoma virus 17 oncogene homolog , CG5680-PB , CG5680-PE , CG5680-PF , JNK , JUN kinase , Jun N-terminal kinase , Jun NH2-terminal kinase , Jun-N-terminal kinase , Jun-kinase , bsk-PB , bsk-PE , bsk-PF , c-Jun N-terminal kinase , c-Jun aminoterminal kinase , c-Jun-N-terminal kinase , drosophila JNK , AP1 , V-jun avian sarcoma virus 17 oncogene homolog , activator protein 1 , proto-oncogene c-jun , transcription factor AP-1 , CDK5 activator 2 , cyclin-dependent kinase 5 activator 2 , cyclin-dependent kinase 5 regulatory subunit 2 , p39I , AH119 , immediate early , jun A , Jun activation domain binding protein , enhancer-binding protein AP1 , jun oncogene , p39 , proto-oncogene c-Jun , v-jun avian sarcoma virus 17 oncogene homolog , Avian sarcoma virus 17 (v-jun) oncogene homolog , Jun oncogene , v-jun sarcoma virus 17 oncogene , CG2275-PA , CG2275-PB , CG2275-PC , Jra-PA , Jra-PB , Jra-PC , Jun related antigen , complementation group V , lethal(2)1A109 , lethal(2)IA109

GENE ID SPECIES
379050 Xenopus laevis
44801 Drosophila melanogaster
396913 Sus scrofa
443219 Ovis aries
100008856 Oryctolagus cuniculus
12570 Mus musculus
16476 Mus musculus
3725 Homo sapiens
335916 Danio rerio
24516 Rattus norvegicus
399400 Xenopus laevis
424673 Gallus gallus
609429 Canis lupus familiaris
280831 Bos taurus
100170668 Ovis aries
36057 Drosophila melanogaster
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