Browse our MAPK14 (MAPK14) ELISA Kits

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Mitogen-Activated Protein Kinase 14 ELISA Kits (MAPK14)
On www.antibodies-online.com are 48 Mitogen-Activated Protein Kinase 14 (MAPK14) ELISA Kits from 10 different suppliers available. Additionally we are shipping MAPK14 Antibodies (195) and MAPK14 Proteins (38) and many more products for this protein. A total of 312 MAPK14 products are currently listed.
Synonyms:
186F5S, anon-sts23, AP22.98, AP22_98, ATMPK14, BG:DS00797.3, CG7393, CRK1, csbp, Csbp1, Csbp2, CSPB1, D-p38, D-p38 MAPK, D-p38b, Dmel\\CG7393, Dmp38b, Dm p38b, Dp38, dp38b, ESTS:186F5S, Exip, Hog, mapk14a, mitogen-activated protein kinase 14, Mpk34C, mxi2, p38, p38 beta, p38 MAPK, p38-alpha, p38a, p38alpha, p38B, p38beta, p38Hog, p38Kb, p38MAPK, Prkm14, Prkm15, RK, sapk2, sapk2a
list all ELISA KIts Gene Name GeneID UniProt
MAPK14 1432 Q16539
MAPK14 26416 P47811
MAPK14 81649  

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More ELISA Kits for MAPK14 Interaction Partners

Cow (Bovine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. results suggest that ET-1 (show EDN1 ELISA Kits)-induced activation of proMMP-2 is mediated via cross-talk between NADPH oxidase (show NOX1 ELISA Kits)-PKCalpha (show PKCa ELISA Kits)-p(38)MAPK (show MAPK1 ELISA Kits) and NFkappaB-MT1MMP (show MMP14 ELISA Kits) signaling pathways along with a marked decrease in TIMP-2 (show TIMP2 ELISA Kits) expression in the cells

  2. cross-talk between p(38)MAPK (show MAPK1 ELISA Kits) and Gialpha play a pivotal role for full activation of cPLA2 (show PLA2G4A ELISA Kits) during ET-1 (show EDN1 ELISA Kits) stimulation of pulmonary artery smooth muscle cells.

  3. MAPK14 signalling pathway is largely involved in heat-induced sperm damage.

  4. p38 MAPK is an early redox sensor in the laminar shear stress with hydrogen peroxide being a signaling mediator.

  5. Blockade of p38 enhances chondrocyte phenotype in monolayer culture and may promote more efficient cartilage tissue regeneration for cell-based therapies.

  6. p38 phosphorylation and MMP13 (show MMP13 ELISA Kits) expression are regulated by Rho/ROCK activation, and support the potential novel pathway that Rho/ROCK is in the upper part of the mechanical stress-induced matrix degeneration cascade in cartilage.

  7. These data suggest that the p38 and JNK (show MAPK8 ELISA Kits) signaling pathways play pivotal roles in PRRSV replication and may regulate immune responses during virus infection.

  8. findings support the hypothesis that ischemic factor stimulation of the blood-brain barrier Na-K-Cl cotransporter (show SLC12A1 ELISA Kits) involves activation of p38 and JNK (show MAPK8 ELISA Kits) MAPKs

  9. These data suggest a differential requirement of JNK1 (show MAPK8 ELISA Kits) and p38 MAPK in TNF (show TNF ELISA Kits) regulation of E2F1 (show E2F1 ELISA Kits). Targeted inactivation of JNK1 (show MAPK8 ELISA Kits) at arterial injury sites may represent a potential therapeutic intervention for ameliorating TNF (show TNF ELISA Kits)-mediated EC dysfunction.

  10. p38 MAPK (MAPK14) is redox-regulated in reactive oxygen species-dependent endothelial barrier dysfunction.

Fruit Fly (Drosophila melanogaster) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. ROS (show ROS1 ELISA Kits)/JNK (show MAPK8 ELISA Kits)/p38/Upd (show UROD ELISA Kits) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  2. Taken together, our findings indicate that the p38 MAP Kinase is an integral component of the core circadian clock of Drosophila in addition to playing a role in stress-input pathways.

  3. Data show that the genetic interaction between p38b MAPK (show MAPK1 ELISA Kits) and Rack1 (show GNB2L1 ELISA Kits) controls muscle aggregate formation, locomotor function, and longevity.

  4. The interaction of any of several Drosophila Delta class glutathione transferases and p38b mitogen-activated protein kinase (show MAPK1 ELISA Kits) can affect the substrate specificity of either enzyme, which suggests induced conformational changes affecting catalysis.

  5. found a correlation between the depth of integration of individual p38 kinases into the protein interaction network and their functional significance; propose a central role of p38b in the p38 signaling module with p38a and p38c playing more peripheral auxiliary roles

  6. Loss of p38 MAPK causes early lethality and precipitates age-related motor dysfunction and stress sensitivity.

  7. The p38 pathway-mediated stress response contribute to Drosophila host defense against microbial infection.

  8. p38b MAPK (show MAPK1 ELISA Kits) plays a crucial role in the balance between intestinal stem cell proliferation and proper differentiation in the adult Drosophila midgut.

  9. the D-p38b gene is regulated by the DREF pathway and DREF is involved in the regulation of proliferation and differentiation of Drosophila ISCs (show NFS1 ELISA Kits) and progenitors

Horse (Equine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. p38 mitogen-activated protein kinase is crucial for bovine papillomavirus type-1 transformation of equine fibroblasts.

  2. p38 Mitogen-activated protein kinase (MAPK (show MAPK1 ELISA Kits)) is essential for drug-induced COX-2 (show PTGS2 ELISA Kits) expression in leukocytes, suggesting that p38 MAPK is a potential target for anti-inflammatory therapy.

  3. These findings support a function for p38 MAPK in equine neutrophil migration and suggest the potential for the ability of p38 MAPK inhibition to limit neutrophilic inflammation in the laminae during acute laminitis.

  4. Cultured equine digital vein endothelial cells were exposed to lipopolysaccharide and phosphorylation of p38 MAPK was assessed by Western blotting using phospho-specific antibodies.

Human Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. findings show that endothelial MAPKs ERK (show EPHB2 ELISA Kits), p38 (show CRK ELISA Kits), and JNK (show MAPK8 ELISA Kits) mediate diapedesis-related and diapedesis-unrelated functions of ICAM-1 (show ICAM1 ELISA Kits) in cerebral and dermal microvascular endothelial cells

  2. Tetraarsenic hexoxide (As4O6) induced G2/M arrest, apoptosis and autophagic cell death through PI3K (show PIK3CA ELISA Kits)/Akt (show AKT1 ELISA Kits) and p38 MAPK pathways alteration in SW620 colon cancer cells.

  3. The N-Terminal phosphorylation of RB by p38 (show CRK ELISA Kits) bypasses its inactivation by cyclin (show PCNA ELISA Kits)-dependent kinases and prevents proliferation in cancer cells.

  4. Inhibition of MAPK14 conclusively facilitates elucidation of the impact of the complex network of p38 MAPK signaling on atherogenesis.

  5. Collectively, this study provides more insights into RELT (show RELT ELISA Kits) expression, RELT (show RELT ELISA Kits) family member function, and the mechanism of RELT (show RELT ELISA Kits)-induced death.

  6. Data, including data from studies conducted in cells from transgenic/knockout mice, suggest that p38alpha MAPK (show MAPK1 ELISA Kits) (MAPK14) activity is required for hypoxia-induced pro-angiogenic activity involving cardiomyocytes and vascular endothelial cells; p38 MAPK activation in cardiomyocyte is sufficient to promote paracrine signaling-mediated, pro-angiogenic activity/myocardial revascularization.

  7. The findings indicate that p38alpha and GADD45alpha (show GADD45A ELISA Kits) are involved in an enhanced vitamin D signaling on TRPV6 (show TRPV6 ELISA Kits) expression.

  8. These results suggest that the p38/NPM/PP2A complex acts as a dynamic sensor, allowing endothelial cells to react rapidly to acute oxidative stress.

  9. Inhibition of the inflammatory signaling intermediate p38 MAPK reduced tissue factor (show F3 ELISA Kits) (TF) mRNA by one third but increased tumor necrosis factor (TNF (show TNF ELISA Kits)) and interleukin-1 beta (IL-1beta (show IL1B ELISA Kits)) mRNA.

  10. These results suggest that the activation of endogenous levels of SFK renders the endothelial barrier more susceptible to low, physiologic doses of TNF-alpha (show TNF ELISA Kits) through activation of p38 (show CRK ELISA Kits) which leads to a loss of endothelial tight junctions.

Mouse (Murine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. Soluble epoxide hydrolase (show EPHX2 ELISA Kits) inhibitor AUDA decreases bleomycin-induced pulmonary toxicity in mice by inhibiting the p38 (show CRK ELISA Kits)/Smad3 (show SMAD3 ELISA Kits) signaling pathway.

  2. These results provide evidence supporting a key role for the p38 MAPK signaling pathway which is involved in the regulation of Abeta1-42 internalization in the parietal cortex and hippocampus of mouse through LRP1 (show LRP1 ELISA Kits) in vivo.

  3. The N-Terminal phosphorylation of RB by p38 (show CRK ELISA Kits) bypasses its inactivation by cyclin (show PCNA ELISA Kits)-dependent kinases and prevents proliferation in cancer cells.

  4. Quantitative real-time polymerase chain reaction (qRT-PCR) and Western blotting were used to detect the mRNA and protein expressions of p-p38MAPK, AAT, signal transducer and activator of transcription 1 (STAT1 (show STAT1 ELISA Kits)) and activating transcription factor2 (ATF2 (show ATF2 ELISA Kits))

  5. p38alpha is essential to maintain in actin dynamics with age in hepatocytes.

  6. Data, including data from studies conducted in cells from transgenic/knockout mice, suggest that p38alpha MAPK (show MAPK1 ELISA Kits) (Mapk14) activity is required for hypoxia-induced pro-angiogenic activity involving cardiomyocytes and vascular endothelial cells; p38 MAPK activation in cardiomyocyte is sufficient to promote paracrine signaling-mediated, pro-angiogenic activity/myocardial revascularization.

  7. blockage of NF-kappaB (show NFKB1 ELISA Kits) p65 (show NFkBP65 ELISA Kits) and/or MAPK p38 (show MAPK1 ELISA Kits) with their specific inhibitors strongly attenuated B7-H3 (show CD276 ELISA Kits)-amplified inflammatory response with significantly reduced proinflammatory cytokine and chemokine (show CCL1 ELISA Kits) production, and markedly ameliorated B7-H3 (show CD276 ELISA Kits)-exacerbated disruption of blood-brain barrier and severity of disease status in S. pneumoniae-infected mice.

  8. Data suggest that single muscle immobilization induces a shift of myosin heavy chain (MHC) isoforms composition toward a faster contractile phenotype and decreases the polymorphic profile of single fibres, and that activation of p38 and JNK could be a potential mechanism involved in these contractile phenotype modifications during muscle immobilization.

  9. The kinase TPL2 (show MAP3K8 ELISA Kits) activates ERK (show EPHB2 ELISA Kits) and p38 (show CRK ELISA Kits) signaling to promote neutrophilic inflammation

  10. studies have therefore uncovered a p38alpha-mediated pathway that alters Hematopoietic Stem/Progenitor Cell metabolism to respond to stress and promote recovery.

Rabbit Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. These findings suggest that the TQ-induced production of ROS (show ROS1 ELISA Kits) causes dedifferentiation through the ERK (show MAPK1 ELISA Kits) pathway and inflammation through the PI3K and p38 pathways in rabbit articular chondrocytes.

  2. These results suggest that p38 MAPK signal transduction pathway is critical to NO-induced chondrocyte apoptosis, and p38 plays a role by way of stimulating NF-kappaB (show NFKB1 ELISA Kits), p53 (show TP53 ELISA Kits) and caspase-3 (show CASP3 ELISA Kits) activation.

Pig (Porcine) Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. Porcine reproductive and respiratory syndrome virus strain CH-1a could significantly up-regulate IL-10 (show IL10 ELISA Kits) production through p38 MAPK activation.

  2. JNK (show MAPK8 ELISA Kits) plays an active role in fragmentation of pig oocytes and p38 MAPK is not involved in this process.[p38MAPK]

  3. Retinal ischemia-reperfusion alters expression of mitogen-activated protein kinases, particularly ERK1/2 (show MAPK1/3 ELISA Kits), in the neuroretina and retinal arteries.

Xenopus laevis Mitogen-Activated Protein Kinase 14 (MAPK14) interaction partners

  1. cytochrome c (show CYCS ELISA Kits) microinjection induces p38 phosphorylation through caspase-3 (show CASP3 ELISA Kits) activation, and caspase (show CASP3 ELISA Kits) inhibition reduces p38 activation induced by osmostress, indicating that a positive feedback loop is engaged by hyperosmotic shock

MAPK14 Antigen Profile

Antigen Summary

The protein encoded by this gene is a member of the MAP kinase family. MAP kinases act as an integration point for multiple biochemical signals, and are involved in a wide variety of cellular processes such as proliferation, differentiation, transcription regulation and development. This kinase is activated by various environmental stresses and proinflammatory cytokines. The activation requires its phosphorylation by MAP kinase kinases (MKKs), or its autophosphorylation triggered by the interaction of MAP3K7IP1/TAB1 protein with this kinase. The substrates of this kinase include transcription regulator ATF2, MEF2C, and MAX, cell cycle regulator CDC25B, and tumor suppressor p53, which suggest the roles of this kinase in stress related transcription and cell cycle regulation, as well as in genotoxic stress response. Four alternatively spliced transcript variants of this gene encoding distinct isoforms have been reported.

Alternative names and synonyms associated with MAPK14

  • mitogen-activated protein kinase 14 (MPK14) Elisa Kit
  • mitogen-activated protein kinase 14 (MAPK14) Elisa Kit
  • mitogen-activated protein kinase 14 (Mapk14) Elisa Kit
  • CG7393 gene product from transcript CG7393-RA (p38b) Elisa Kit
  • mitogen activated protein kinase 14 (Mapk14) Elisa Kit
  • mitogen-activated protein kinase 14 (mapk14) Elisa Kit
  • 186F5S Elisa Kit
  • anon-sts23 Elisa Kit
  • AP22.98 Elisa Kit
  • AP22_98 Elisa Kit
  • ATMPK14 Elisa Kit
  • BG:DS00797.3 Elisa Kit
  • CG7393 Elisa Kit
  • CRK1 Elisa Kit
  • csbp Elisa Kit
  • Csbp1 Elisa Kit
  • Csbp2 Elisa Kit
  • CSPB1 Elisa Kit
  • D-p38 Elisa Kit
  • D-p38 MAPK Elisa Kit
  • D-p38b Elisa Kit
  • Dmel\\CG7393 Elisa Kit
  • Dmp38b Elisa Kit
  • Dm p38b Elisa Kit
  • Dp38 Elisa Kit
  • dp38b Elisa Kit
  • ESTS:186F5S Elisa Kit
  • Exip Elisa Kit
  • Hog Elisa Kit
  • mapk14a Elisa Kit
  • mitogen-activated protein kinase 14 Elisa Kit
  • Mpk34C Elisa Kit
  • mxi2 Elisa Kit
  • p38 Elisa Kit
  • p38 beta Elisa Kit
  • p38 MAPK Elisa Kit
  • p38-alpha Elisa Kit
  • p38a Elisa Kit
  • p38alpha Elisa Kit
  • p38B Elisa Kit
  • p38beta Elisa Kit
  • p38Hog Elisa Kit
  • p38Kb Elisa Kit
  • p38MAPK Elisa Kit
  • Prkm14 Elisa Kit
  • Prkm15 Elisa Kit
  • RK Elisa Kit
  • sapk2 Elisa Kit
  • sapk2a Elisa Kit

Protein level used designations for MAPK14

MAP kinase 14 , MAP kinase p38 alpha , MAPK 14 , mitogen-activated protein kinase p38 alpha , p38 mitogen activated protein kinase , CG7393-PA , p38 mitogen-activated protein kinase , p38b-PA , stress-activated p38b MAP kinase , p38 mitogen-activated kinase , cytokine suppressive anti-inflammatory drug binding protein 1 , mitogen activated protein kinase 14 , p38 MAP kinase alpha , p38 MAPK , p38 alpha , tRNA synthetase cofactor p38 , CSAIDS-binding protein 1 , mitogen-activated protein kinase 14A , stress-activated protein kinase 2a , Csaids binding protein , MAP kinase 2 , MAP kinase Mxi2 , MAX-interacting protein 2 , cytokine suppressive anti-inflammatory drug binding protein , cytokine-supressive anti-inflammatory drug binding protein , mitogen-activated protein kinase 14 , p38 MAP kinase , p38alpha Exip , reactive kinase , stress-activated protein kinase 2A , MAPK p38 , MPK2 , Mitogen-activated protein kinase 2 , mitogen-activated Mitogen-activated protein kinase 2

GENE ID SPECIES
829797 Arabidopsis thaliana
534492 Bos taurus
403856 Canis lupus familiaris
100723285 Cavia porcellus
34780 Drosophila melanogaster
100063532 Equus caballus
421183 Gallus gallus
1432 Homo sapiens
26416 Mus musculus
100341695 Oryctolagus cuniculus
450161 Pan troglodytes
81649 Rattus norvegicus
100156630 Sus scrofa
379992 Xenopus laevis
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