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Human GSTM1 ELISA Kit for Sandwich ELISA - ABIN417506
Singh, Prasad, Singh, Singh, Gupta, Paliwal, Pandey, Gupta: Human Glutathione S-Transferase Enzyme Gene Polymorphisms and Their Association With Neurocysticercosis. in Molecular neurobiology 2016
Human GSTM1 ELISA Kit for Sandwich ELISA - ABIN840218
Wu, Lu, Tang, Zhang, Yuan, Li, Di Wu, Sun, Lu, Xia, Chen, Sha, Wang: GSTM1 and GSTT1 null polymorphisms and male infertility risk: an updated meta-analysis encompassing 6934 subjects. in Scientific reports 2013
Polymorphisms of GSTM1 is associated with lung cancer.
The loss of GSTM1 was significantly associated with incident kidney and heart failure, independent of traditional risk factors. These results suggest GSTM1 function is a potential treatment target for the prevention of kidney and heart failure
results suggest that combined deletion polymorphisms of GSTT1 (show GSTT1 ELISA Kits) and GSTM1 can have implications in the prediction of the clinical course of the disease
On comparing the prevalence of GSTM1 null polymorphism among the group with subjects with tobacco habits and no oral lesions, oral leukoplakia, and oral squamous cell carcinoma, it was observed that there was a statistically significant association between GSTM1 null polymorphism and the different groups (P < 0.01).
The present case-control study found that GSTM1 and GSTT1 (show GSTT1 ELISA Kits) polymorphism are not associated with JOAG (show MYOC ELISA Kits) risk in North Indian population. The present meta-analysis suggested that there might be a significant association of GSTM1 null genotype with glaucoma (JOAG (show MYOC ELISA Kits) & POAG) risk.
Pesticide-exposed individuals with inherited susceptible metabolic genotypes (particularly, null genotype for GSTM1 and the PON1 (show PON1 ELISA Kits) 192R allele) appear to have an increased risk of genotoxic DNA damage.
182 Hungarian bladder cancer cases and 78 cancer-free controls were investigated. It was not possible to establish a particular impact of NAT2 (show SLC38A1 ELISA Kits)*6A and *7B genotypes (15 cases, 8%, 5 controls, 7%). GSTT1 (show GSTT1 ELISA Kits) exerted no marked influence on bladder cancer (negative 21% cases vs. 22% controls). The portion of GSTM1 negative bladder cancer patients was increased (63% cases vs. 54% controls).
double-null genotype of the GSTM1 gene is not associated with ALL development or its progression.
We assessed the effect of allelic deletions in the GSTM1 and GSTT1 (show GSTT1 ELISA Kits) genotypes on lung cancer overall survival through a systematic review of the scientific literature after applying predefined inclusion and exclusion criteria
GSTM1 genetic polymorphisms are not associated with the development of graft-versus-host disease after allogeneic hematopoietic stem cell transplantation.
Six proteins were regulated at both basal and inducible levels exhibiting the largest dynamic range of Nrf2 (show NFE2L2 ELISA Kits) regulation: cytochrome CYP2A5, GSTM3 (show glutathione S-transferase mu 3 (brain) ELISA Kits), GSTM1, ENTPD5 (show ENTPD5 ELISA Kits),UDPGDH (show UGDH ELISA Kits), and EPHX1 (show EPHX1 ELISA Kits).
mitochondrial Gstb1 is induced under oxidative stress
the gene (GSTM1) encoding the detoxification enzyme glutathione S-transferase M1 is transcriptionally upregulated by Myb (show MYB ELISA Kits)
The Gstm1 gene was represented by two EST (show MAP3K8 ELISA Kits) clones with similar levels of downregulation.
We overexpressed Hoxa9 (show HOXA9 ELISA Kits) and Meis1 (show MEIS1 ELISA Kits) in primary hematopoietic cells. Arrays identified c-Myb (show MYB ELISA Kits) and a c-Myb (show MYB ELISA Kits) target (Gstm1) among the genes with the strongest response to Hoxa9 (show HOXA9 ELISA Kits)/Meis1 (show MEIS1 ELISA Kits).
Genetic variants that cause a decremental change in expression of Gstm1 may permit an environment of exaggerated oxidative stress, leading to susceptibility to vascular remodeling and atherosclerosis
The role of polymorphisms of glutathione S-transferases GSTM1, M3, P1, T1 and A1 in susceptibility to alcoholic liver disease. In addition, oxidant stress is proposed to be an important pathogenic factor in liver damage related to alcohol.
Cytosolic and membrane-bound forms of glutathione S-transferase are encoded by two distinct supergene families. At present, eight distinct classes of the soluble cytoplasmic mammalian glutathione S-transferases have been identified: alpha, kappa, mu, omega, pi, sigma, theta and zeta. This gene encodes a glutathione S-transferase that belongs to the mu class. The mu class of enzymes functions in the detoxification of electrophilic compounds, including carcinogens, therapeutic drugs, environmental toxins and products of oxidative stress, by conjugation with glutathione. The genes encoding the mu class of enzymes are organized in a gene cluster on chromosome 1p13.3 and are known to be highly polymorphic. These genetic variations can change an individual's susceptibility to carcinogens and toxins as well as affect the toxicity and efficacy of certain drugs.
GST class-mu 1
, glutathione S-transferase Mu 1
, glutathione S-transferase mu 1
, glutathione S-transferase M1
, GST HB subunit 4
, HB subunit 4
, S-(hydroxyalkyl)glutathione lyase
, glutathione S-alkyltransferase
, glutathione S-aralkyltransferase
, glutathione S-aryltransferase
, glutathione S-transferase M4
, glutathione S-transferase mu 4
, GST 3-3
, GST Yb1
, glutathione S-transferase Yb-1 subunit
, glutathione-S-transferase mu type 1 (Yb1)
, glutathione-S-transferase, mu type 1 (Yb1)
, GST class-mu 2
, GST, muscle
, S-(hydroxyalkyl)glutathione lyase M2
, glutathione S-alkyltransferase M2
, glutathione S-aralkyltransferase M2
, glutathione S-aryltransferase M2
, glutathione S-transferase 4
, glutathione S-transferase M2 (muscle)
, glutathione S-transferase Mu 2
, GST 1-1
, glutathione S-transferase GT8.7
, glutathione-S-transferase, mu 1