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Human TNFRSF1B ELISA Kit for Sandwich ELISA - ABIN625090
Cuchacovich, Hagan, Khan, Richert, Espinoza: Tumor necrosis factor-alpha (TNF-α)-blockade-induced hepatic sarcoidosis in psoriatic arthritis (PsA): case report and review of the literature. in Clinical rheumatology 2011
These results show an unclear effect of considered T>C polymorphism on TNF-RII gene expression in bovine leukocytes and they suggest the involvement of BLV in modifying the TNF-RII expression in BLV-infected cows.
These results suggest that the endometrium might lower the TNF (show TNF ELISA Kits) concentration in the blastocyst by (1) regulating TNF (show TNF ELISA Kits) secretion into the uterine fluid and (2) inducing decreased TNF (show TNF ELISA Kits) and TNFR2 mRNA transcription in the embryo.
These results suggest that TNF-alpha (show TNF ELISA Kits) sources include immune cells, as well as large and small luteal cells, and that TNF-RI (show TNFRSF1A ELISA Kits) and TNF-RII are present in the luteal cells of the bovine corpus luteum.
The expression and cellular localization of tumor necrosis factor-alpha (TNF (show TNF ELISA Kits)) and its receptors (TNFRI (show TNFRSF1A ELISA Kits) and TNFRII) mRNAs and proteins, were determined.[TNFRII]
The TNFRSF1Brs3397 variant may play a role in modulating the risk of rheumatoid arthritis, but does not provide strong evidence of an impact of TNFRSF1B variants in determining response to anti-TNF (show TNF ELISA Kits) drugs.
atopic dermatits patients had increased TNFR2 expression on immune cells
the blocking of tumor necrosis factor receptor 2 (TNFR2) decreased TL1A (show TNFSF15 ELISA Kits)-stimulated IL-6 (show IL6 ELISA Kits) production by rheumatoid arthritis fibroblast-like synoviocytes.
Data suggest that, by targeting tumor cells and immunosuppressive tumor-associated Tregs, antagonistic tumor necrosis factor receptor 2 (TNFR2) antibodies may be an effective treatment for cancers positive for TNFR2.
p75 neurotrophin receptor (show NGFR ELISA Kits) (p75TNFR) was the first NGFR (nerve growth factor receptor (show NGFR ELISA Kits)) to be cloned and the founding member of the TNFR (show TNFRSF1A ELISA Kits) (tumor necrosis factor (show TNF ELISA Kits) receptor) superfamily. However, data suggest that p75TNF is an atypical TNFR (show TNFRSF1A ELISA Kits) superfamily protein; p75TNFR forms dimers (activated by dimeric neurotrophins) that are structurally unrelated to TNFR (show TNFRSF1A ELISA Kits) superfamily proteins. [REVIEW]
High plasma levels of TNFR2 and TNFR1 (show TNFRSF1A ELISA Kits) were associated with incident intracerebral hemorrhage.
this study shows that TNFalpha (show TNF ELISA Kits) significantly augments IFNgamma production from IL-2 (show IL2 ELISA Kits)-/IL-12 (show IL12A ELISA Kits)-treated NK cells in vitro, an effect mimicked by a TNFR2 agonistic antibodies
TRIM28 (show TRIM28 ELISA Kits) acts as a central factor in controlling endothelial inflammatory responses and angiogenic activities by retaining expression of TNFR-1 (show TNFRSF1A ELISA Kits) and -2 and VEGF receptor 2 in endothelial cells
Serum TNFR2 levels were elevated in lupus nephritis patients versus controls.
Adalimumab bound to monocyte membrane TNF (show TNF ELISA Kits) from rheumatoid arthritis patients and unexpectedly enhanced its expression and its binding to TNF-RII expressed on T reg (show EXTL3 ELISA Kits) cells.
TNFR2 sensitizes macrophages for endogenous TNF (show TNF ELISA Kits)-induced TNFR1 (show TNFRSF1A ELISA Kits)-mediated necroptosis
study reports an important role for TNFR2 on low-affinity-primed secondary effector CD8 (show CD8A ELISA Kits)+ T cells; results demonstrate the importance of TNF (show TNF ELISA Kits) signaling in low-affinity, cross-reactive CD8 (show CD8A ELISA Kits)+ T cell responses during heterologous immunity
TNF (show TNF ELISA Kits) plays an inhibitory role in modulating myocardial SDF-1 (show CXCL12 ELISA Kits) production and blockade of TNF (show TNF ELISA Kits) signaling by ablation of TNFR1 (show TNFRSF1A ELISA Kits) and TNFR2 genes increased SDF-1 (show CXCL12 ELISA Kits) expression in the heart. These data expand on TNF (show TNF ELISA Kits) signaling-initiated mechanisms in myocardium, which may lend a more complete understanding of SDF-1 (show CXCL12 ELISA Kits) and TNFR (show TNFRSF1A ELISA Kits)-derived actions in hopes of advancing ischemic heart injury treatments.
Results suggest that elevated TNF (show TNF ELISA Kits) in the heart, via TNFR1 (show TNFRSF1A ELISA Kits) and TNFR2, restrains cardiomyocyte differentiation of resident cardiac stem cells and may enhance adrenergic activation, both effects that would reduce the effectiveness of endogenous cardiac repair and the response to exogenous stem cell therapy, while promoting adverse cardiac remodeling.
Report chronic inflammatory multiorgan hepatobiliary pancreatitis, along with fibrosis and calculi formation induced by oral dibutyltin administration in TNFR1 (show TNFRSF1A ELISA Kits)/R2 deficient mice.
These findings implicate TNF (show TNF ELISA Kits)-receptor signaling cascades in the regulation of homeostatic plasticity of denervated networks and suggest an important role for TNFalpha (show TNF ELISA Kits)-signaling in the course of neurological diseases accompanied by deafferentation.
TNF (show TNF ELISA Kits) receptor 1 (TnfR1 (show TNFRSF1A ELISA Kits)) signaling is believed to be a major mediator of the cytotoxicity of Tnf-a (show TNF ELISA Kits) through activation of caspases.
our findings implicate TNFR2 in supporting myeloid-derived suppressor cells -mediated immune suppression and metastasis in the liver
TNFR2 blockade appears to disrupt commensal bacteria-host immune symbiosis to reveal autoimmune demyelination in genetically susceptible mice.
TNFR2 protects mice from colitis by inhibiting the expansion of colonic CD8 (show CD8A ELISA Kits)(+) T cells.
Retinal ischemia results in increased expression of TNF-alpha (show TNF ELISA Kits) and its receptors (TNF-R1 (show TNFRSF1A ELISA Kits) and TNF-R2).
Porcine TNFR2 (1,125 bp, 375 amino acid residues), which contains specific amino acid region of transmembrane, indicated high identities with human and murine TNFR2
The protein encoded by this gene is a member of the TNF-receptor superfamily. This protein and TNF-receptor 1 form a heterocomplex that mediates the recruitment of two anti-apoptotic proteins, c-IAP1 and c-IAP2, which possess E3 ubiquitin ligase activity. The function of IAPs in TNF-receptor signalling is unknown, however, c-IAP1 is thought to potentiate TNF-induced apoptosis by the ubiquitination and degradation of TNF-receptor-associated factor 2, which mediates anti-apoptotic signals. Knockout studies in mice also suggest a role of this protein in protecting neurons from apoptosis by stimulating antioxidative pathways.
TNF receptor-associated factor 2
, E3 ubiquitin-protein ligase TRAF6
, TNF receptor-associated factor 6
, TNF receptor-associated factor 3
, TRAF family member-associated NFKB activator
, TNF receptor-associated factor 2-like
, tumor necrosis factor receptor superfamily member 1B
, tumor necrosis factor receptor 2
, tumor necrosis factor receptor superfamily, member 1B
, p75 TNF receptor
, p80 TNF-alpha receptor
, soluble TNFR1B variant 1
, tumor necrosis factor beta receptor
, tumor necrosis factor binding protein 2
, tumor necrosis factor receptor type II
, TNF receptor beta chain
, p75 TNFR
, tumor necrosis factor receptor-II