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Human TNFRSF1B ELISA Kit for Sandwich ELISA - ABIN625090
Cuchacovich, Hagan, Khan, Richert, Espinoza: Tumor necrosis factor-alpha (TNF-α)-blockade-induced hepatic sarcoidosis in psoriatic arthritis (PsA): case report and review of the literature. in Clinical rheumatology 2011
These results show an unclear effect of considered T>C polymorphism on TNF-RII gene expression in bovine leukocytes and they suggest the involvement of BLV in modifying the TNF-RII expression in BLV-infected cows.
These results suggest that the endometrium might lower the TNF (show TNF ELISA Kits) concentration in the blastocyst by (1) regulating TNF (show TNF ELISA Kits) secretion into the uterine fluid and (2) inducing decreased TNF (show TNF ELISA Kits) and TNFR2 mRNA transcription in the embryo.
These results suggest that TNF-alpha (show TNF ELISA Kits) sources include immune cells, as well as large and small luteal cells, and that TNF-RI (show TNFRSF1A ELISA Kits) and TNF-RII are present in the luteal cells of the bovine corpus luteum.
The expression and cellular localization of tumor necrosis factor-alpha (TNF (show TNF ELISA Kits)) and its receptors (TNFRI (show TNFRSF1A ELISA Kits) and TNFRII) mRNAs and proteins, were determined.[TNFRII]
High plasma levels of TNFR2 and TNFR1 (show TNFRSF1A ELISA Kits) were associated with incident intracerebral hemorrhage.
this study shows that TNFalpha (show TNF ELISA Kits) significantly augments IFNgamma production from IL-2 (show IL2 ELISA Kits)-/IL-12 (show IL12A ELISA Kits)-treated NK cells in vitro, an effect mimicked by a TNFR2 agonistic antibodies
TRIM28 (show TRIM28 ELISA Kits) acts as a central factor in controlling endothelial inflammatory responses and angiogenic activities by retaining expression of TNFR-1 (show TNFRSF1A ELISA Kits) and -2 and VEGF receptor 2 in endothelial cells
Serum TNFR2 levels were elevated in lupus nephritis patients versus controls.
Adalimumab bound to monocyte membrane TNF (show TNF ELISA Kits) from rheumatoid arthritis patients and unexpectedly enhanced its expression and its binding to TNF-RII expressed on T reg (show EXTL3 ELISA Kits) cells.
Voxel-based morphometry was used to analyse the associations between TNFRSF1B (rs1061624) genotypes and grey matter structure. Analysis of the TNFRSF1B SNP rs1061624 yielded a significant association with hippocampal but not with striatal volume, whereby G homozygotes were associated with increased volumes relative to A homozygotes and heterozygotes.
Circulating TNFR1 (show TNFRSF1A ELISA Kits) and 2 were shown to be associated with cardiovascular disease, independent of age, sex, inflammatory markers, and other cardiovascular disease risk factors, in the chronic kidney disease patients
Data suggest that TRAF2 (TNF receptor-associated factor 2 (show TRAF2 ELISA Kits)) negatively regulates (1) TNFR1- (tumor necrosis factor binding protein 1 (show TNFRSF1A ELISA Kits))-induced apoptosis, (2) TNFR2- (tumor necrosis factor (show TNF ELISA Kits) receptor type 2)-induced non-canonical NFkappaB (show NFKB1 ELISA Kits) signaling, and (3) TNF- (tumor necrosis factor (show TNF ELISA Kits))-induced necroptosis. [REVIEW]
Our results demonstrated that Treg frequencies and TNFR2 expression on Tregs are increased in sarcoidosis, followed by a decline during infliximab therapy, suggesting a pathophysiological role of this T cell subset.
Data indicate that plasma concentrations of TNFR1 (show TNFRSF1A ELISA Kits) and TNFR2 are elevated in pediatric lupus nephritis.
TNFR2 sensitizes macrophages for endogenous TNF (show TNF ELISA Kits)-induced TNFR1 (show TNFRSF1A ELISA Kits)-mediated necroptosis
study reports an important role for TNFR2 on low-affinity-primed secondary effector CD8 (show CD8A ELISA Kits)+ T cells; results demonstrate the importance of TNF (show TNF ELISA Kits) signaling in low-affinity, cross-reactive CD8 (show CD8A ELISA Kits)+ T cell responses during heterologous immunity
TNF (show TNF ELISA Kits) plays an inhibitory role in modulating myocardial SDF-1 (show CXCL12 ELISA Kits) production and blockade of TNF (show TNF ELISA Kits) signaling by ablation of TNFR1 (show TNFRSF1A ELISA Kits) and TNFR2 genes increased SDF-1 (show CXCL12 ELISA Kits) expression in the heart. These data expand on TNF (show TNF ELISA Kits) signaling-initiated mechanisms in myocardium, which may lend a more complete understanding of SDF-1 (show CXCL12 ELISA Kits) and TNFR (show TNFRSF1A ELISA Kits)-derived actions in hopes of advancing ischemic heart injury treatments.
Results suggest that elevated TNF (show TNF ELISA Kits) in the heart, via TNFR1 (show TNFRSF1A ELISA Kits) and TNFR2, restrains cardiomyocyte differentiation of resident cardiac stem cells and may enhance adrenergic activation, both effects that would reduce the effectiveness of endogenous cardiac repair and the response to exogenous stem cell therapy, while promoting adverse cardiac remodeling.
Report chronic inflammatory multiorgan hepatobiliary pancreatitis, along with fibrosis and calculi formation induced by oral dibutyltin administration in TNFR1 (show TNFRSF1A ELISA Kits)/R2 deficient mice.
These findings implicate TNF (show TNF ELISA Kits)-receptor signaling cascades in the regulation of homeostatic plasticity of denervated networks and suggest an important role for TNFalpha (show TNF ELISA Kits)-signaling in the course of neurological diseases accompanied by deafferentation.
TNF (show TNF ELISA Kits) receptor 1 (TnfR1 (show TNFRSF1A ELISA Kits)) signaling is believed to be a major mediator of the cytotoxicity of Tnf-a (show TNF ELISA Kits) through activation of caspases.
our findings implicate TNFR2 in supporting myeloid-derived suppressor cells -mediated immune suppression and metastasis in the liver
TNFR2 blockade appears to disrupt commensal bacteria-host immune symbiosis to reveal autoimmune demyelination in genetically susceptible mice.
TNFR2 protects mice from colitis by inhibiting the expansion of colonic CD8 (show CD8A ELISA Kits)(+) T cells.
Retinal ischemia results in increased expression of TNF-alpha (show TNF ELISA Kits) and its receptors (TNF-R1 (show TNFRSF1A ELISA Kits) and TNF-R2).
Porcine TNFR2 (1,125 bp, 375 amino acid residues), which contains specific amino acid region of transmembrane, indicated high identities with human and murine TNFR2
The protein encoded by this gene is a member of the TNF-receptor superfamily. This protein and TNF-receptor 1 form a heterocomplex that mediates the recruitment of two anti-apoptotic proteins, c-IAP1 and c-IAP2, which possess E3 ubiquitin ligase activity. The function of IAPs in TNF-receptor signalling is unknown, however, c-IAP1 is thought to potentiate TNF-induced apoptosis by the ubiquitination and degradation of TNF-receptor-associated factor 2, which mediates anti-apoptotic signals. Knockout studies in mice also suggest a role of this protein in protecting neurons from apoptosis by stimulating antioxidative pathways.
TNF receptor-associated factor 2
, E3 ubiquitin-protein ligase TRAF6
, TNF receptor-associated factor 6
, TNF receptor-associated factor 3
, TRAF family member-associated NFKB activator
, TNF receptor-associated factor 2-like
, tumor necrosis factor receptor superfamily member 1B
, tumor necrosis factor receptor 2
, tumor necrosis factor receptor superfamily, member 1B
, p75 TNF receptor
, p80 TNF-alpha receptor
, soluble TNFR1B variant 1
, tumor necrosis factor beta receptor
, tumor necrosis factor binding protein 2
, tumor necrosis factor receptor type II
, TNF receptor beta chain
, p75 TNFR
, tumor necrosis factor receptor-II