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Data show that cytochrome P450 (show CYP ELISA Kits) enzymes Cyp17 (show CYP17A1 ELISA Kits)-I, Cyp11c1 (show CYP11B2 ELISA Kits), Cyp19a1a and Cyp19a1b and one of their regulators forkhead protein (show FOXO4 ELISA Kits) Foxl2a were detected both in the testis and ovary.
Data indicate taht monocrotophos (MCP) exposure modulated gene expression of forkhead transcription factor gene L2 (foxl2), doublesex/mab-3 related transcription factor 1 (dmrt1), gonadal aromatase (cyp19a1a) and brain aromatase (cyp19a1b).
Effects of sexual steroids on the expression of foxl2 in Gobiocypris rarus
Xenopus W-linked DM-W induces foxl2 expression during ovary formation.
evidence that FOXL2 modulates Col1a2 (show COL1A2 ELISA Kits) transcription through interaction with a response element 65 Kb upstream of the transcription start site
Data indicate that a balance between supporting cell (show PTPRJ ELISA Kits) self-renewal and differentiation is maintained in the developing ovary by relative Wnt4 (show WNT4 ELISA Kits) protein/beta-catenin (show CTNNB1 ELISA Kits) and p27Kip1 (show CDKN1B ELISA Kits) protein (p27 (show CDKN1B ELISA Kits))/Forkhead box L2 (FOXL2) activities.
Data indicate that the hypothalamic-pituitary-gonadal axis controls expression of Foxl2 in pituitary gonadotropes primarily through positive feedback from ovarian hormones.
FOXL2 mobilizes estrogen signaling to establish a coherent feed-forward loop repressing Sox9.
Foxl2 has a crucial role in postnatal uterine maturation and could help to understand sub-fertility predisposition in women.
Results support FOXL2 as a master transcription factor in a spectrum of developmental processes, including growth, cartilage and bone formation. Its action overlaps that of SOX9 (show SOX9 ELISA Kits), though they are antagonistic in female vs male gonadal sex determination but conjoint in cartilage and skeletal development.
Microarray expression profiling of whole adrenal mRNA from ovariectomized vs. intact mice demonstrated selective upregulation of gonadal-like genes including Spinlw1 and Insl3 (show INSL3 ELISA Kits) in GDX (show UBL4A ELISA Kits)-induced adrenocortical tumors of the mouse.
our results demonstrate the necessity of FOXL2 for proper FSH (show BRD2 ELISA Kits) production in mice and implicate FOXL2 in integration of transcription factors at the level of the FSHbeta promoter.
Results show that a piggyBac insertion ~160 kb upstream of the transcription start site of Foxl2 partially disrupted its expression resulting in BPES (Blepharophimosis, ptosis, epicanthus inversus syndrome)-like conditions.
AMH (show AMH ELISA Kits) is an endogenous target gene of FOXL2. AMH (show AMH ELISA Kits) and FOXL2 collaboratively work to reserve ovarian follicles.
The de novo mutation rate in FOXL2 is exceptionally high compared with other dominant disorders manifesting with an ocular phenotype.
SUMOylation of FOXL2 and PML (show PML ELISA Kits) Bodies
Ten novel protein partners of FOXL2
FOXL2 mobilizes estrogen signaling to maintain the identity of ovarian granulosa cells
The absence of FOXL2 and DICER1 (show DICER1 ELISA Kits) gene mutation observed in 3 patients, along with strong FOXL2 immunoreactivity provides additional evidence to place microcystic stromal tumor within pure gonadal stromal rather than sex cord ovarian tumors.
genetic association study in two families with blepharophimosis-ptosis-epicanthus inversus syndrome type 1: Two distinct mutations, a missense p.H104R change and an in-frame p.A222_A231dup10 duplication in FOXL2 gene are identified in three women.
suggests the potential of pS33 FOXL2 to serve as a new biomarker for the diagnosis of adult-type GCT (show QPCT ELISA Kits)
Our combined analysis identifies potential candidate genes, whose alterations might contribute to adult-type Ovarian granulosa cell tumors formation/progression together with the recurrent FOXL2 somatic mutation.
Describe microcystic stromal tumor as a distinctive ovarian sex cord-stromal neoplasm characterized by FOXL2, SF-1 (show NR5A1 ELISA Kits), WT-1 (show WT1 ELISA Kits), Cyclin D1 (show CCND1 ELISA Kits), and beta-catenin (show CTNNB1 ELISA Kits) nuclear expression and CTNNB1 (show CTNNB1 ELISA Kits) mutations.
Foxl2 deletion in either Cranial Neural Crest Cells (CNCCs) or Cranial Mesodermal Cells (CMCs (show GPSM2 ELISA Kits)) prevents eyelid closure and induces subtle skeletal developmental defects.
This gene encodes a forkhead transcription factor. The protein contains a fork-head DNA-binding domain and may play a role in ovarian development and function. Mutations in this gene are a cause of blepharophimosis syndrome and premature ovarian failure 3.
forkhead box L2
, forkhead box protein L2
, transcription factor FOXL2
, forkhead transcription factor L2
, foxl2 protein
, fork-head box L2 transcription factor
, pituitary forkhead factor
, putative transcription factor foxl2
, forkhead transcription factor FOXL2
, Forkhead box protein L2