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Human Polyclonal NR1H2/3 Primary Antibody for ELISA, WB - ABIN250297
Tangirala, Bischoff, Joseph, Wagner, Walczak, Laffitte, Daige, Thomas, Heyman, Mangelsdorf, Wang, Lusis, Tontonoz, Schulman: Identification of macrophage liver X receptors as inhibitors of atherosclerosis. in Proceedings of the National Academy of Sciences of the United States of America 2002
Show all 2 references for ABIN250297
this study shows that the anti-inflammatory effect of platycodin D in IL-1beta (show IL1B Antibodies)-stimulated chondrocytes is mediated by activating LXR-a
results indicated that down-regulation of LXRalpha (show NR1H3 Antibodies) was shown to suppress invasion and EMT (show ITK Antibodies) of gastric cancer cells
this study shows that show that H2O2 exerts a dual regulation on mRNA expression of LXRalpha (show NR1H3 Antibodies) and its target genes
combined PPARgamma (show PPARG Antibodies) C1431T, PGC-1alpha (show PPARGC1A Antibodies) G482S, and LXRalpha (show NR1H3 Antibodies) -115G/A polymorphisms increase the risk of coronary artery disease and predicted the severity of coronary atherosclerosis in Thais
Distinct gene regulatory programs define the inhibitory effects of liver X receptors, NR1H2 (show NR1H2 Antibodies)/NR1H3 (show NR1H3 Antibodies) and PPARG (show PPARG Antibodies) on cancer cell proliferation.
these data show that YXS is effective in mitigating MI/R injury by suppressing mitochondrial mediated apoptosis and oxidative stress and by upregulating LXRalpha (show NR1H3 Antibodies), thereby providing a rationale for future clinical trials and clinical applications
decreased expression of miR (show MLXIP Antibodies)-155 in the peripheral blood may be utilized as a potential novel biomarker for non-alcoholic fatty liver disease screening mainly by targeting LXRalpha (show NR1H3 Antibodies).
HDAC5 inhibits hepatic lipogenic genes expression by attenuating the transcriptional activity of liver X receptor.
The anti-inflammatory effects of platelet-derived microparticles in human plasmacytoid dendritic cells involve liver X receptor activation.
these data identify a new mechanism of LXR regulation that involves TIPARP, ADP-ribosylation and MACROD1.
The protein encoded by this gene belongs to the NR1 subfamily of the nuclear receptor superfamily. The NR1 family members are key regulators of macrophage function, controlling transcriptional programs involved in lipid homeostasis and inflammation. This protein is highly expressed in visceral organs, including liver, kidney and intestine. It forms a heterodimer with retinoid X receptor (RXR), and regulates expression of target genes containing retinoid response elements. Studies in mice lacking this gene suggest that it may play an important role in the regulation of cholesterol homeostasis. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
liver X nuclear receptor alpha variant 1
, oxysterols receptor LXR-alpha