Hydroxysteroid (17-Beta) Dehydrogenase 10 (HSD17B10) (C-Term) Peptide

Details for Product No. ABIN695772
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Protein Name
Synonyms
hadh2, im:6899159, wu:fj61f03, zgc:101605, abad, camr, erab, hcd2, hsd10, mhbd, mrpp2, mrx17, mrx31, mrxs10, schad, sdr5c1, HADH2, HSD17B10, 17b-HSD10, ABAD, CAMR, DUPXp11.22, ERAB, HCD2, MHBD, MRPP2, ... show more
hadh2, im:6899159, wu:fj61f03, zgc:101605, abad, camr, erab, hcd2, hsd10, mhbd, mrpp2, mrx17, mrx31, mrxs10, schad, sdr5c1, HADH2, HSD17B10, 17b-HSD10, ABAD, CAMR, DUPXp11.22, ERAB, HCD2, MHBD, MRPP2, MRX17, MRX31, MRXS10, SCHAD, SDR5C1, 17bHSD10, Ads9, Hadh2 show less
Protein Region
C-Term
Source
Synthetic
Antibody Type Synthetic
Application
Blocking Peptide (BP)
Pubmed 2 references available
Quantity 0.1 mg
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Catalog No. ABIN695772
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Immunogen Synthetic peptide
Specificity The synthetic peptide sequence used to generate the antibody AP6307b was selected from the C-term region of human ABAD. A 10 to 100 fold molar excess to antibody is recommended. Precise conditions should be optimized for a particular assay.
Alternative Name ABAD
Background Amyloid b-peptide-binding alcohol dehydrogenase (ABAD) is a member of the family of short chain dehydrogenase/reductases, unique among this family, it binds amyloid b-peptide and exhibits enzymatic activity toward a wide variety of substrates including linear alcohols. In an amyloid beta-abundant environment, ABAD appears to trigger cell stress induced by the amyloid peptide.
Restrictions For Research Use only
Storage 4
Storage Comment Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles
Expiry Date 6 months
Background publications He, Merz, Mehta et al.: "Human brain short chain L-3-hydroxyacyl coenzyme A dehydrogenase is a single-domain multifunctional enzyme. Characterization of a novel 17beta-hydroxysteroid dehydrogenase." in: The Journal of biological chemistry, Vol. 274, Issue 21, pp. 15014-9, 1999 (PubMed).

Takuma, Yao, Huang et al.: "ABAD enhances Abeta-induced cell stress via mitochondrial dysfunction." in: FASEB journal : official publication of the Federation of American Societies for Experimental Biology, Vol. 19, Issue 6, pp. 597-8, 2005 (PubMed).

Validation Images
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