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anti-Human ZBTB16 Antibodies:
anti-Mouse (Murine) ZBTB16 Antibodies:
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Human Monoclonal ZBTB16 Primary Antibody for ChIP, ICC - ABIN2668927
Koken, Reid, Quignon, Chelbi-Alix, Davies, Kabarowski, Zhu, Dong, Chen, Chen, Tan, Licht, Waxman, de Thé, Zelent: Leukemia-associated retinoic acid receptor alpha fusion partners, PML and PLZF, heterodimerize and colocalize to nuclear bodies. in Proceedings of the National Academy of Sciences of the United States of America 1997
Show all 3 Pubmed References
Human Monoclonal ZBTB16 Primary Antibody for FACS - ABIN4895342
Gérart, Sibéril, Martin, Lenoir, Aguilar, Picard, Lantz, Fischer, Latour: Human iNKT and MAIT cells exhibit a PLZF-dependent proapoptotic propensity that is counterbalanced by XIAP. in Blood 2013
Show all 2 Pubmed References
Human Monoclonal ZBTB16 Primary Antibody for ELISA (Capture), PLA - ABIN2773728
Narimatu, Nagata, Sugizaki, Hamasaki: [A new method for immunological detection of fecal blood, using colloidal gold labeled with anti-human hemoglobin monoclonal antibody]. in Rinsho byori. The Japanese journal of clinical pathology 1992
Human Polyclonal ZBTB16 Primary Antibody for ICC, IF - ABIN4346320
Wasim, Carlet, Mansha, Greil, Ploner, Trockenbacher, Rainer, Kofler: PLZF/ZBTB16, a glucocorticoid response gene in acute lymphoblastic leukemia, interferes with glucocorticoid-induced apoptosis. in The Journal of steroid biochemistry and molecular biology 2010
Human Monoclonal ZBTB16 Primary Antibody for ICC, ELISA - ABIN969465
Felicetti, Errico, Bottero, Segnalini, Stoppacciaro, Biffoni, Felli, Mattia, Petrini, Colombo, Peschle, Carè: The promyelocytic leukemia zinc finger-microRNA-221/-222 pathway controls melanoma progression through multiple oncogenic mechanisms. in Cancer research 2008
Human Monoclonal ZBTB16 Primary Antibody for CyTOF, FACS - ABIN4899031
Hsieh, Botta, Gao, Li, Van Allen, Treacy, Cai, He, Sweeney, Brown, Balk, Nelson, Garraway, Kantoff: PLZF, a tumor suppressor genetically lost in metastatic castration-resistant prostate cancer, is a mediator of resistance to androgen deprivation therapy. in Cancer research 2015
Cow (Bovine) Polyclonal ZBTB16 Primary Antibody for WB - ABIN2780737
Deng, Zhang, Kang, Liu, Zhao, Gao, Li, Pan, Tang, Wang, Niu, Yang, Zeng: An unusual haplotype structure on human chromosome 8p23 derived from the inversion polymorphism. in Human mutation 2008
Show all 2 Pubmed References
Low ZBTB16 expression is associated with tongue squamous cell carcinoma.
High PLZF expression is associated with acute myeloid leukemia (show BCL11A Antibodies).
Emerging evidence shows that PLZF regulates the balance of self-renewal and differentiation in stem cells.
integration of PLZF ChIP-Seq and RNA Pol II RNA-Seq datasets revealed that the early growth response 1 (EGR1 (show EGR1 Antibodies)) transcription factor is a PLZF target for which its level of expression must be reduced to enable progesterone dependent hESC decidualization
NLRP7 (show NLRP7 Antibodies) is involved in Hydatidiform Molar Pregnancy (HYDM1) and interacts with the transcriptional repressor ZBTB16
These results support a prognostic value for loss of cytoplasmic PLZF expression in the stratification of non-small cell lung cancer and a possible role of cytoplasmic shift and down-regulation of PLZF in the pathogenesis of NSCLC.
down-regulation of PLZF is an important molecular process for prostate tumor progression
Down-regulation of PLZF in human hepatocellular carcinoma.PLZF expression was positively correlated with the alkaline phosphatase level in hepatocellular carcinoma patients.
In dental follicle cells, a Runx2 (show RUNX2 Antibodies)-independent differentiation mechanism exists that is regulated by ZBTB16.
PLZF loss enhances castration-resistant prostate cancer tumor growth in a xenograft model.
these data reveal the full profile of PLZF and SALL4 (show SALL4 Antibodies) regulatory targets in undifferentiated spermatogonia.
Several layers of regulation of the innate-like NKT (show CTSL1 Antibodies) effector program: First, PLZF bound and regulated genes encoding cytokine receptors as well as homing and adhesion receptors; second, PLZF bound and activated T-helper-specific transcription factor genes that in turn control T-helper-specific programs; finally, PLZF bound and suppressed the transcription of Bach2 (show BACH2 Antibodies), a potent general repressor of effector differentiation
Together, these results suggest a novel mechanism regulating endothelial PATZ1 (show ZNF278 Antibodies) expression based on the down-regulation of miR (show MLXIP Antibodies)-24 expression caused by hyperglycemia. Interfering with PATZ1 (show ZNF278 Antibodies) expression via miRNAs or miRNA mimics could potentially represent a new way to target endothelial PATZ1 (show ZNF278 Antibodies)-dependent signaling of vascular dysfunction in diabetes.
Results clearly indicate that PLZF promotes cell cycle exit and cell differentiation into hypertrophic chondrocytes.
plzf has a role in regulating HSC (show FUT1 Antibodies) function that is linked to cell cycle regulation
PLZF expression is highly specific to innate T cells and cannot be induced in conventional T cells via TCR-mediated activation or inflammatory challenge
PLZF expression at the innate lymphoid cell precursor stage has a long-range effect on the functional properties of mature ILC2s and highlights the importance of these cells for innate allergic responses in otherwise immunocompetent mice.
Data suggest that age-related increase in Plzf expression represents a novel molecular signature of spermatogonia aging by functionally arresting their differentiation.
Data indicate the function of promyelocytic leukaemia zinc finger PLZF in the development of innate-like interleukin-17 (show IL17A Antibodies), TcR gamma-delta (IL-17 (show IL17A Antibodies)+Vgamma6+ gammadelta) T cells.
The data suggest that Pak2 (show PAK2 Antibodies) controls thymic Natural Killer T-cell development by providing a signal that links Egr2 (show EGR2 Antibodies) to induce PLZF, in part by regulating signaling lymphocyte activation molecule (show SLAMF1 Antibodies) 6 expression.
This gene is a member of the Krueppel C2H2-type zinc-finger protein family and encodes a zinc finger transcription factor that contains nine Kruppel-type zinc finger domains at the carboxyl terminus. This protein is located in the nucleus, is involved in cell cycle progression, and interacts with a histone deacetylase. Specific instances of aberrant gene rearrangement at this locus have been associated with acute promyelocytic leukemia (APL). Alternate transcriptional splice variants have been characterized.
zinc finger and BTB domain-containing protein 16
, zinc finger protein 145 (Kruppel-like, expressed in promyelocytic leukemia)
, zinc finger protein PLZF
, promyelocytic leukemia zinc finger protein
, Green's luxoid
, zinc finger protein 145
, Polydactyly-luxate syndrome
, promyelocytic leukemia zinc finger
, zinc finger and BTB domain containing 16 protein
, promyelocytic leukemia zinc finger protein-A
, zinc finger and BTB domain containing 16
, zinc finger and BTB domain-containing protein 16-A
, zinc finger protein PLZF-A