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TGFbeta2 regulates hypothalamic TRH expression through TIEG1 during fetal development.
co-regulation of the enzyme glutaminyl cyclase (show QPCT Proteins) and its substrate TRH was reflected by a co-induction of both proteins in reactive astrocytes in proximity of Abeta (show APP Proteins) deposits
RXRA (show RXRA Proteins) and RXRB (show RXRB Proteins) display specific roles in modulating T3-dependent regulation of thyrotropin releasing hormone.
Reversal of hypothyroidism in female patient after treatment with thyrotropin releasing hormone.
Data suggest that expression of TRH is up-regulated by CREM (cAMP responsive element modulator (show CREM Proteins) protein) in hypothalamus; however, regulation of TRH by triiodothyronine appears independent of CREM (show CREM Proteins) or CREB (cAMP responsive element binding protein (show CREB1 Proteins)).
Thyrotropin-releasing hormone altered the basal expression of the TSHbeta (show TSHB Proteins) gene and the responsiveness of the gene to thyroid hormone (show PTH Proteins).
Klf4 (show KLF4 Proteins) plays a key role in the maturation of TRH expression in hypothalamic neurons.
Data demonstrate that the distribution of hypophysiotropic TRH neurons in mice is vastly different from the pattern in rats, with a dominant occurrence of these neurosecretory cells in the compact part and adjacent regions at the mid-level of the PVN.
These demonstrations of PPARgamma (show PPARG Proteins) effects on hypothalamic Trh transcription in vivo consolidate the role of the TRH neuron as a central integrator of energy homeostasis.
study provides functional in vivo data showing that both thyroid hormone receptor (show THRA Proteins) beta1 and beta2 isoforms have specific roles in regulating thyrotropin releasing hormone transcription
TRH rs13097335 may have a protective role toward the development of breast cancer.
Data indicate that decrease of cholesterol level impaired the functional coupling between the thyrotropin-releasing hormone receptor and the cognate G proteins.
TRH, LH-RH and substance P (show TAC1 Proteins) are not affected in Alzheimer disease and Down's syndrome.
Cpe (show CPE Proteins) mutations in mouse may affect processing of prothyrotropin releasing hormone into thyrotropin releasing hormone.
TRH inhibits melanin-concentrating hormone (MCH (show PMCH Proteins)) neurons by increasing synaptic inhibition from local GABA neurons.
This study introduces TRH as a novel, potent, selective, and evolutionarily highly conserved neuroendocrine factor controlling human pigmentation in situ.
Case Report: Isolated idiopathic central hypothyroidism in an adult, possibly caused by thyrotropin releasing hormone (TRH) deficiency.
Thus, human scalp hair follicles are both a source and a target of TRH, which operates as a potent hair-growth stimulator
TRH mRNA levels in "mute" thyrotropinomas: cause-effect or coincidence?
results show that that leptin (show LEP Proteins) can regulate trh gene expression via activation of intracellular signal transducer and activator of transcription 3 (STAT3 (show STAT3 Proteins)) proteins in TRH neurons in the hypothalamus
Distribution of the mRNAs encoding proTRH and the three TRH receptors in the central nervous system and pituitary of X. laevis is described.
This hormone is responsible for the regulation and release of thyroid-stimulating hormone, as well as prolactin. Deficiency of this hormone has been associated with hypothalamic hypothyroidism.
, pro-thyrotropin-releasing hormone
, thyrotropin releasing hormone
, thyrotropin-releasing factor
, prepro-thyrotropin-releasing hormone
, thyrotropin-releasing hormone
, pro-thyrotropin-releasing hormone-B
, prothyroliberin type B
, pro-thyrotropin-releasing hormone-A
, prothyroliberin type A
, thyroliberin type A