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Human DEFB103A ELISA Kit for Sandwich ELISA - ABIN419779
Berger, McClellan, Vistisen, Hazlett: HIF-1? is essential for effective PMN bacterial killing, antimicrobial peptide production and apoptosis in Pseudomonas aeruginosa keratitis. in PLoS pathogens 2013
Show all 2 Pubmed References
hBD3 exhibits potent anti-periodontitis properties both in vitro and in vivo, and this effect may be correlated to inhibition of the nuclear factor-kappaB pathway and macrophage polarization.
Different dynamics and pathway of disulfide bonds reduction has been demonstrated for two human defensins, HD5 (show DEFA5 ELISA Kits) and HBD-3.
In this study, the results showed genetically modified (GM) milk containing human beta-defensin-3 (HBD3) was easy to be digested in simulated gastric fluid, and it did not have adverse effects on general and gastrointestinal (GI) tract health compared to conventional milk.
this study shows that hBD3 amplifies the response to bacterial DNA in both mouse and human immune cells in a TLR9 (show TLR9 ELISA Kits)-dependent manner
Direct interaction between SP-A or SAP01 and human BD-3 seemed to be responsible for the inhibitory effects on chemotaxis of mast cells.
Expression of hBD-1, hBD-2, hBD-3, and hBD-4 in healthy and chronic periodontitis gingiva.
Per duplication of mRNA in whole blood, the risk of being a carrier increased by 93% for IL17A (show IL17A ELISA Kits) and decreased by 35% for IFN-gamma (show IFNG ELISA Kits). Independent of carriage and DEFB promotor haplotype, a 1-unit increase in IFN-gamma (show IFNG ELISA Kits)/IL-17A (show IL17A ELISA Kits) mRNA ratio led to a 24% increase in HBD3 mRNA in wounded skin. A low Th1 (show TH1L ELISA Kits) to Th17 mRNA ratio increases the risk of colonization, with HBD3 being a potential link.
Analysis of TLR4 (show TLR4 ELISA Kits) and HBD3 expression in epithelial cells demonstrates the key role of the innate immune factors in the pathogenesis of inflammatory and destructive periodontal lesions.
study indicated that the expression of HBD-2 (show DEFB4 ELISA Kits) and HBD-3 mRNA and protein in lesions of cutaneous tuberculosis may be not consistent with that of tuberculids
epidermal growth factor receptor (EGFR (show EGFR ELISA Kits))activation via phosphorylation of EGFR (show EGFR ELISA Kits) via TAK1 (show MAP3K7 ELISA Kits)-p38alpha (show MAPK14 ELISA Kits) pathway, and subsequent internalization of EGFR (show EGFR ELISA Kits) are functionally linked to the induction of hBD3 in gastric epithelial cells infected with H. pylori.
Thus, oral epithelial cells dominantly control IL-17R-dependent responses to oropharyngeal candidiasis through regulation of BD3 expression.
C/EBPbeta (show CEBPB ELISA Kits) contributes to immunity to mucosal candidiasis during cortisone immunosuppression in a manner linked to beta-defensin 3 expression, but is apparently dispensable for the IL-17 (show IL17A ELISA Kits)-dependent response.
Stress decreases mouse beta-defensin-3 expression in the esophagus of mice with diabetes.
visfatin (show NAMPT ELISA Kits) enhances CAMP, hBD-2 (show DEFB4 ELISA Kits), hBD-3, and S100A7 (show S100A7 ELISA Kits) production in human keratinocytes and their orthologs in murine imiquimod-treated psoriatic skin.
this investigation revealed an indispensable role for BD3, BD4, and CRAMP in defense against F. solani-induced keratitis
SP-A (show SFTPA1 ELISA Kits) protects lung epithelium from tissue injury caused by BD3
Mouse beta-defensin 3 was strongly up-regulated in colonic epithelium of 15 (but not 5) week-old IL-2 (show IL2 ELISA Kits)(-/-) mice and in dextran-sulfate-induced chronic (but not acute) colitis, but not in IL-2 (show IL2 ELISA Kits)(+/-) 15-week-old mice.
Epithelial and mucosal tissues expressed bd3 and recombinant bd3 exhibits antifungal activity.
Murine beta-defensins BD2 (show DEFB4 ELISA Kits) and BD3 together promote resistance against corneal infection by Pseudomonas aeruginosa.
Mycobacterial infection induces the expression of mBD3 (show MBD3 ELISA Kits) and mBD4 (show MBD4 ELISA Kits), and both might participate in the control of mycobacterial growth.
Expression of the bovine DEFB103 gene was surveyed in 27 tissues and transcript was most abundant in tissues with stratified squamous epithelium.
Defensins form a family of microbicidal and cytotoxic peptides made by neutrophils. Members of the defensin family are highly similar in protein sequence. This gene encodes defensin, beta 103B, which has broad spectrum antimicrobial activity and may play an important role in innate epithelial defense.
, beta-defensin 3
, defensin, beta 103
, defensin, beta 3
, defensin-like protein
, beta defensin-2
, beta-defensin 4
, defensin beta 3
, beta defensin 3
, beta-defensin 103A
, defensin, beta 103B
, Beta-defensin 6
, bovine neutrophil beta-defensin 3
, defensin, beta 103A
, beta-defensin 103B
, beta-defensin 300