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hBD3 (show DEFB103A Antibodies) exhibits potent anti-periodontitis properties both in vitro and in vivo, and this effect may be correlated to inhibition of the nuclear factor-kappaB pathway and macrophage polarization.
Different dynamics and pathway of disulfide bonds reduction has been demonstrated for two human defensins, HD5 (show DEFA5 Antibodies) and HBD-3 (show DEFB103A Antibodies).
In this study, the results showed genetically modified (GM) milk containing human beta-defensin-3 (HBD3 (show DEFB103A Antibodies)) was easy to be digested in simulated gastric fluid, and it did not have adverse effects on general and gastrointestinal (GI) tract health compared to conventional milk.
Direct interaction between SP-A or SAP01 and human BD-3 (show DEFB103A Antibodies) seemed to be responsible for the inhibitory effects on chemotaxis of mast cells.
Expression of hBD-1 (show DEFB1 Antibodies), hBD-2 (show DEFB4 Antibodies), hBD-3 (show DEFB103A Antibodies), and hBD-4 (show DEFB104A Antibodies) in healthy and chronic periodontitis gingiva.
Per duplication of mRNA in whole blood, the risk of being a carrier increased by 93% for IL17A (show IL17A Antibodies) and decreased by 35% for IFN-gamma (show IFNG Antibodies). Independent of carriage and DEFB promotor haplotype, a 1-unit increase in IFN-gamma (show IFNG Antibodies)/IL-17A (show IL17A Antibodies) mRNA ratio led to a 24% increase in HBD3 (show DEFB103A Antibodies) mRNA in wounded skin. A low Th1 (show TH1L Antibodies) to Th17 mRNA ratio increases the risk of colonization, with HBD3 (show DEFB103A Antibodies) being a potential link.
Analysis of TLR4 (show TLR4 Antibodies) and HBD3 (show DEFB103A Antibodies) expression in epithelial cells demonstrates the key role of the innate immune factors in the pathogenesis of inflammatory and destructive periodontal lesions.
study indicated that the expression of HBD-2 (show DEFB4 Antibodies) and HBD-3 (show DEFB103A Antibodies) mRNA and protein in lesions of cutaneous tuberculosis may be not consistent with that of tuberculids
epidermal growth factor receptor (EGFR (show EGFR Antibodies))activation via phosphorylation of EGFR (show EGFR Antibodies) via TAK1 (show MAP3K7 Antibodies)-p38alpha (show MAPK14 Antibodies) pathway, and subsequent internalization of EGFR (show EGFR Antibodies) are functionally linked to the induction of hBD3 (show DEFB103A Antibodies) in gastric epithelial cells infected with H. pylori.
These results demonstrate that the gamma-core within HBD3 (show DEFB103A Antibodies) is the ancestral core of the full-length molecule and is a viable HDP (show HNRNPA1 Antibodies) per se, since it is endowed with the most important biological features of HBD3 (show DEFB103A Antibodies)
Defensins form a family of microbicidal and cytotoxic peptides made by neutrophils. Members of the defensin family are highly similar in protein sequence. This gene encodes defensin, beta 103B, which has broad spectrum antimicrobial activity and may play an important role in innate epithelial defense.
, beta-defensin 3
, defensin, beta 103
, defensin-like protein
, Gal 3
, antimicrobial peptide-3
, beta-defensin 1
, beta-defensin prepropeptide
, Beta-defensin 6
, bovine neutrophil beta-defensin 3
, defensin beta 3