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Ceruloplasmin Protein

CP Origin: Human Host: Human Native > 95 % pure
Catalog No. ABIN934757
  • Target See all Ceruloplasmin (CP) Proteins
    Ceruloplasmin (CP) (Ceruloplasmin (Ferroxidase) (CP))
    Protein Type
    Native
    Origin
    • 3
    • 2
    • 1
    Human
    Source
    • 3
    • 2
    • 1
    Human
    Characteristics
    Purified native Human Ceruloplasmin protein
    Protein Source: Human plasma
    Purity
    > 95 % pure
    Top Product
    Discover our top product CP Protein
  • Application Notes
    Each Investigator should determine their own optimal working dilution for specific applications.
    Restrictions
    For Research Use only
  • Format
    Lyophilized
    Reconstitution
    Reconstitute with deionized water to the pre-lyophilization volume.
    Buffer
    Lyophilized from 50 mM KH0 O4, pH 6.8, with 100 M KCl, 20 M E-amino caproic acid and 0 M EDTA.
    Precaution of Use
    Donor samples were tested and found to be negative for HBsAg, anti-HCV, anti-HBc, and negative for anti-HIV 1 & 2. Nonetheless caution should be used when handling this material as there is a margin of error in all tests.
    Handling Advice
    Avoid repeated freeze/thaw cycles.
    Storage
    -20 °C
    Storage Comment
    Aliquot and store at -20 °C.
  • Target
    Ceruloplasmin (CP) (Ceruloplasmin (Ferroxidase) (CP))
    Alternative Name
    Ceruloplasmin (CP Products)
    Synonyms
    CP-2 Protein, fi23f10 Protein, wu:fi23f10 Protein, D3Ertd555e Protein, CERP Protein, CP Protein, ceruloplasmin Protein, CP Protein, cp Protein, Cp Protein, LOC100533122 Protein
    Background
    An acute-phase reactant. Increased levels are associated with normal pregnancy, rheumatoid arthritis, and cirrhosis. Decreased levels are associated with hepatolenticular degeneration (Wilson's Disease). An elevated level of Cp is found in patients with progressive tumors. Additionally, as Cp is a prooxidant, an elevated level is a sign of cardiovascular disease.
    Description: Human plasma.
    Molecular Weight
    132 kDa
    Pathways
    Transition Metal Ion Homeostasis
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