Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
ANT1 can induce autoimmune myocarditis in A/J mice by generating autoreactive T cells.
miR (show MLXIP ELISA Kits)-2861 and ANT1 are regulators of cardiomyocyte necrosis and myocardial infarction.
These results suggest that follicular helper T cells cells and IL-21 (show IL21 ELISA Kits) might be involved in the pathogenesis of viral myocarditis and play an important role in anti-ANT autoantibody production.
Data indicate that palmitoyl-carnitine (PC) increases the type 2 ryanodine receptor (RyR2 (show RYR2 ELISA Kits)) oxidation and reduces the ATP/ADP translocase (ANT) activity.
Downregulation of adenine nucleotide translocator 1 exacerbates tumor necrosis factor-alpha (show TNF ELISA Kits)-mediated cardiac inflammatory responses.
Plasma membrane-localized ANT1 and ANT2 (show SLC25A5 ELISA Kits) regulate L1-mediated neurite outgrowth in conjunction with MMP14 (show MMP14 ELISA Kits).
Although the molecular mechanism linking ANT1-Cys (show DNAJC5 ELISA Kits)(57) nitroalkylation and uncoupling is not yet known, these data suggest that ANT1-mediated uncoupling may be a mechanism for nitroalkene-induced cardioprotection.
The attenuation of ANT-1 in the presence of PGC-1alpha (show PPARGC1A ELISA Kits) overexpression preserves the mitochondrial membrane potential in response to hydrogen-peroxide stress.
Data demonstrate that ANT1 protein level (show YY1 ELISA Kits)s are upregulated in Mecp2-null mice.
uncoupling protein-3 (show UCP3 ELISA Kits) can minimize the induction of the adenine nucleotide translocase (show SLC25A5 ELISA Kits)-mediated 'energy-wasting' process during Calorie restriction (CR).
Yeast aac2 (show SLC25A5 ELISA Kits) R96H and aac2 (show SLC25A5 ELISA Kits) R252G mutations are equivalent to R80H and R235G human ANT1 pathological mutations. mtDNA instability induced by aac2R96H and aac2R252G is rescued by N-acetylcysteine.
identification by whole-exome sequencing of seven probands harboring dominant, de novo SLC25A4 mutations; all affected individuals presented at birth, were ventilator dependent and, where tested, revealed severe combined mitochondrial respiratory chain deficiencies associated with a marked loss of mitochondrial DNA copy number in skeletal muscle
A directed proteomic approach discovered the novel interaction of BKCa (show KCNMA1 ELISA Kits) with Tom22 (show TOMM22 ELISA Kits), a component of the mitochondrion outer membrane import system, and the adenine nucleotide translocator (ANT).
Identification of ZNF555 as a putative transcriptional factor that impacts ANT1 promoter activity in facioscapulohumeral dystrophy myoblasts.
elevated ANT1 expression supports EV infection and is associated with EV persistence, a condition with adverse prognosis.
Data suggest acetylation of ANT1 at lysines 10/23/92 has dramatic physiological effects on ADP-ATP exchange; extent of acetylation of lysine 23 decreases following physical activity; this change is highly dependent on insulin (show INS ELISA Kits) sensitivity/resistance.
Expression of ANT1 were lower in inclusion body myositis samples versus both polymyositis and controls
Compares and contrasts all the known human SLC25A (show SLC25A25 ELISA Kits)* genes and includes functional information.
A 13-generation Mennonite pedigree with autosomal recessive myopathy and cardiomyopathy due to an SLC25A4 frameshift null mutation (c.523delC, p.Q175RfsX38), which codes for the heart-muscle isoform of the adenine nucleotide translocator-1, was studied.
This report expands the clinical spectrum of ANT1-related human diseases, and emphasises the crucial role of the mitochondrial ADP/ATP carriers in muscle function and pathophysiology of human myopathies.
Data show that the binding sites on adenine nucleotide translocator 1 (ANT1) have an affinity for cardiolipin (CL).
This gene is a member of the mitochondrial carrier subfamily of solute carrier protein genes. The product of this gene functions as a gated pore that translocates ADP from the mitochondrial matrix into the cytoplasm. The protein forms a homodimer embedded in the inner mitochondria membrane. Mutations in this gene have been shown to result in autosomal dominant progressive external opthalmoplegia and familial hypertrophic cardiomyopathy.
adenine nucleotide translocator 1
, ADP,ATP carrier protein 1, mitochondrial
, ANT 1
, ADP/ATP translocase 1
, Adenine nucleotide translocator 1
, ADP,ATP carrier protein 1
, ADP,ATP carrier protein, heart/skeletal muscle
, adenine nucleotide translocase-1
, adenine nucleotide translocator 1, skeletal muscle
, solute carrier family 25 member 4
, mitochondrial adenine nucleotide translocator
, solute carrier family 25 (mitochondrial adenine nucleotide translocator) member 4
, adenine nucleotide translocator 1 (skeletal muscle)
, heart/skeletal muscle ATP/ADP translocator
, ADP,ATP carrier protein, heart
, ADP/ATP carrier
, ADP/ATP translocase T1
, 30 kDa CSQ-binding protein
, 30 kDa calsequestrin-binding protein
, CSQ-binding 30 kDa protein
, solute carrier family 25 (mitochondrial carrier; adenine nucleotide translocator), member 5