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A yopH mutant survived better in the absence of neutrophils, indicating that neutrophil inactivation by YopH by targeting PRAM-1/SKAP-HOM (show SKAP2 ELISA Kits) and SLP-76 (show LCP2 ELISA Kits)/Vav (show VAV1 ELISA Kits)/PLCgamma2 (show PLCG2 ELISA Kits) signaling hubs may be critical for Yersinia survival.
autophagy contributes to the anti-apoptotic function of the PML (show PML ELISA Kits)-RARalpha (show RARA ELISA Kits) protein through inhibiting the Akt (show AKT1 ELISA Kits)/mTOR (show FRAP1 ELISA Kits) pathway
Transgenic mice expressing PML-RAR alpha (show RARA ELISA Kits) develop acute promyelocytic leukemia (show PML ELISA Kits) with long latency, low penetrance, and acquired cytogenetic abnormalities.
Our results show that the pretreatment PML (show PML ELISA Kits)-RARA (show RARA ELISA Kits) molecular burden could therefore be used to improve risk stratification in order to develop more individualized treatment regimens for high-risk APL (show FASL ELISA Kits) cases.
The 3-plex RT-qPCR assay is a specific, sensitive, stable, and cost-effective method that can be used for the rapid diagnosis and treatment monitoring of acute promyelocytic leukemia (show PML ELISA Kits) with PML (show PML ELISA Kits)-RARa (show RARA ELISA Kits)
The MIR125B1-mediated blockade of PML (show PML ELISA Kits)-RARA (show RARA ELISA Kits) proteolysis was regulated via an autophagy-lysosomal pathway, contributing to the inhibition of acute promyelocytic leukemia (show PML ELISA Kits) differentiation.
PRAM-1 is required for optimal integrin-dependent neutrophil function.
Caspase 3 (show CASP3 ELISA Kits)-cleaved SH3 domain (show ITSN1 ELISA Kits) of HIP-55 (show DBNL ELISA Kits) is likely involved in PRAM-1-mediated JNK (show MAPK8 ELISA Kits) activation upon arsenic trioxide-induced differentiation of NB4 cells.
The use of reverse-transcription polymerase chain reaction for the detection of the PML (show PML ELISA Kits)-RARA (show RARA ELISA Kits) and RARA (show RARA ELISA Kits)-PML (show PML ELISA Kits) fusion genes that allow for monitoring of acute myelocytic leukemia.
PML (show PML ELISA Kits)-RARalpha (show RARA ELISA Kits) fusion transcripts have been shown to be useful markers for establishing the diagnosis and for monitoring the response to treatment of acute promyelocytic leukemia (show PML ELISA Kits).
Interaction of PRAM-1 and hSH3 domains of adhesion and degranulation promoting adapter protein (show TOLLIP ELISA Kits) (ADAP (show FYB ELISA Kits)) with phosphatidylcholine (show SGMS2 ELISA Kits)-containing liposomes is observed upon incorporation of phosphatidylserine or phosphoinositides into the membrane bilayer.
hidden abnormalities and novel disease-related genomic changes occur in t(15;17)translocation in acute promyelocytic leukemia (show PML ELISA Kits) formation of PML (show PML ELISA Kits)-RARA (show RARA ELISA Kits) gene
The protein encoded by this gene is similar to FYN binding protein (FYB/SLAP-130), an adaptor protein involved in T cell receptor mediated signaling. This gene is expressed and regulated during normal myelopoiesis. The expression of this gene is induced by retinoic acid and is inhibited by the expression of PML-RARalpha, a fusion protein of promyelocytic leukemia (PML) and the retinoic acid receptor-alpha (RARalpha).
PML-RARA regulated adaptor molecule 1
, PML-RARA-regulated adapter molecule 1-like
, PML-RARA-regulated adapter molecule 1
, PML-RARA target gene encoding an Adaptor Molecule-1
, PML-RAR alpha-regulated adaptor molecule 1