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mRNA Expression of Raldh3 during zebrafish early development.
Melanoma treatment with the novel irreversible isoform-specific ALDH1 (show ALDH1A1 ELISA Kits) inhibitor [4-dimethylamino-4-methyl-pent (show PNMT ELISA Kits)-2-ynthioic acid-S methylester] di-methyl-ampal-thio (show ACAA1 ELISA Kits)-ester (DIMATE) or depletion of ALDH1A1 (show ALDH1A1 ELISA Kits) and/or ALDH1A3, promoted the accumulation of apoptogenic aldehydes leading to apoptosis and tumor growth inhibition in immunocompetent, immunosuppressed and patient-derived xenograft mouse models.
these findings ascribe a novel function for ALDH1A3 in an aggressive glioma stem cells phenotype via the up-regulation of tissue transglutaminase (show TGM2 ELISA Kits)
our findings define a FOXD1 (show FOXD1 ELISA Kits)-ALDH1A3 pathway in controling the clonogenic and tumorigenic potential of mesenchymal glioma stem-like cells in glioblastoma tumors
we looked up our single center primary prostate cancer post-operative follow-up data and suggested that the high level ALDH1A3 expression could predict the poor progression-free survival in a 158-patient cohort. We concluded that ALDH1A3, localized in luminal layer in prostate epithelium, is highly expressed in prostate cancer
this report brings new information for the phenotype-genotype correlation of ALDH1A3 mutations and raises important questions, especially in terms of genetic counselling given to the patients and their families
ALDH1A3 was most relevant to extracellular matrix organization and cell adhesion biological process, and the ability of tumor invasion was suppressed after ALDH1A3 knockdown in vitro.
the prevalence of ALDH1A3(+)/CD44 (show CD44 ELISA Kits)(+) tumor cells in breast cancer is significantly associated with worse prognostic factors and favors a poor prognosis
Whole-exome sequencing in a South American coh (show RARB ELISA Kits)ort links ALDH1A3, FOXN1 and RARB/retinoic acid regulation pathways to autism spectrum di (show FOXN1 ELISA Kits)sorders.
we demonstrated that a specific ALDH isoform, namely ALDH1A3, is enriched in chemoresistant mesothelioma cell subpopulations
ALDH1A3 induces differential RA signaling in breast cancer cells which affects the rate of breast cancer progression.
Chromatin immunoprecipitation assay using RarB (show RARB ELISA Kits) as the immunoprecipitation target suggests retinoic acid regulation of Aldh1a3 and Foxn1 (show FOXN2 ELISA Kits) in mice.
Data suggest that expression of Aldh1a3/Raldh3 in placenta plays role in differentiation/placentation of glycogen (show GYS1 ELISA Kits) trophoblast cells (junctional zone cells) via production of a local source of retinoic acid.
Raldh1 (show ALDH1A1 ELISA Kits) and Raldh3 influence enteric nervous system structure and function and heterozygosity for Raldh2 (show ALDH1A2 ELISA Kits) causes ENS defects
ALDH1A3 and ALDH2 (show ALDH2 ELISA Kits) expression was detectable in ALDH (show ALDH3A1 ELISA Kits)(very-br) and ALDH (show ALDH3A1 ELISA Kits)(br) cells, unlike ALDH (show ALDH3A1 ELISA Kits)(dim) cells, albeit at lower levels compared with ALDH1A1 (show ALDH1A1 ELISA Kits) and ALDH1A2 (show ALDH1A1 ELISA Kits).
regulation of fat depots through the concerted action of Aldh1 (show ALDH1A1 ELISA Kits) enzymes establishes retinoic acid-dependent tandem regulation of transcription factors ZFP423 (show 104125 ELISA Kits) and PPARgamma (show PPARG ELISA Kits) in a depot-specific manner
Kinetic characterization of RALDH3 and 4, providing their specificities for retinal isomer substrates.
Raldh2 (show ALDH1A2 ELISA Kits) and Raldh3 are selectively expressed in cortical stroma and in the ureteric bud during kidney development.
Three retinaldehyde dehydrogenases (RALDH1 (show ALDH1A1 ELISA Kits), RALDH2 (show ALDH1A2 ELISA Kits) and RALDH3), show differential expression patterns throughout later mouse organogenesis
RALDH3 is transiently expressed during early postnatal cortical development, suggesting an important role in cortical neuronal development.
RA-generating activities in Raldh2 (show ALDH1A2 ELISA Kits)-null mouse embryos could be ascribed to RALDH3 in eye, nasal, and inner ear epithelia, and within the mesonephric area that expresses RALDH3.
Aldehyde dehydrogenase isozymes are thought to play a major role in the detoxification of aldehydes generated by alcohol metabolism and lipid peroxidation. The enzyme encoded by this gene uses retinal as a substrate, either in a free or cellular retinol-binding protein form.
aldehyde dehydrogenase 1A3
, aldehyde dehydrogenase 1 family, member A3
, aldehyde dehydrogenase family 1 subfamily A3-like
, aldehyde dehydrogenase family 1 member A3
, retinaldehyde dehydrogenase family 1 subfamily A3
, aldehyde dehydrogenase family 1 member A3-like
, acetaldehyde dehydrogenase 6
, aldehyde dehydrogenase 6
, retinaldehyde dehydrogenase 3
, aldehyde dehydrogenase family 1, subfamily A3
, aldehyde dehydrogenase-6