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Rabbit BDNF ELISA Kit for Sandwich ELISA - ABIN572355
Chung, Kim, Im, Yoo, Choi, Hwang, Won, Chang, Cho, Hwang, Moon: Neuroprotective effects of adipose-derived stem cells against ischemic neuronal damage in the rabbit spinal cord. in Journal of the neurological sciences 2012
Show all 4 references for ABIN572355
Rat (Rattus) BDNF ELISA Kit for Sandwich ELISA - ABIN416070
Wang, Liu, Jin, Dai, Zhao, Xie, Liu, Yu, Ma: Interaction of PFOS and BDE-47 co-exposure on thyroid hormone levels and TH-related gene and protein expression in developing rat brains. in Toxicological sciences : an official journal of the Society of Toxicology 2011
Show all 2 references for ABIN416070
Human BDNF ELISA Kit for Sandwich ELISA - ABIN414355
Li, Chen, Wang, Lü, Huan, Fang, Han, Ge, Chen: Changes in growth factor levels in the cerebrospinal fluid of autism patients after transplantation of human umbilical cord blood mononuclear cells and umbilical cord-derived mesenchymal stem cells. in Genetics and molecular research : GMR 2016
Mouse (Murine) BDNF ELISA Kit for Sandwich ELISA - ABIN415358
Yan, Zhang, Jia, Sun, Liu: Chronic stress impairs learning and hippocampal cell proliferation in senescence-accelerated prone mice. in Neuroscience letters 2011
These results suggest an involvement of the BDNF/TrkB (show NTRK2 ELISA Kits) system in the regulation of food intake and energy balance in zebrafish, as in mammals
BDNF-TrkB (show NTRK2 ELISA Kits) influences the expression level of components of chemokine (show CCL1 ELISA Kits) signaling including Cxcr4b, and the generation of progenitors of mechanoreceptors, at the level of expression of Atoh1a-Atp2b1a.
Light regulates the expression of the BDNF/TrkB2 (show NTRK2 ELISA Kits) system in the adult zebrafish retina.
Brain-derived neurotrophic factor (BDNF) induces polarized signaling of small GTPase (show RACGAP1 ELISA Kits) (Rac1) protein at the onset of Schwann cell myelination through partitioning-defective 3 (Par3 (show PARD3 ELISA Kits)) protein.
the results demonstrate that bdnf mediates non-cell-autonomous maintenance of position and thereby the identity of differentiated neurons
The present results demonstrate that there is a parallel time-related decline in the expression of BDNF and TrkB (show NTRK2 ELISA Kits) in zebrafish.
The cloning and analysis of three additional zebrafish (Danio rerio) BDNF gene exons and two associated promoters, is reported. Among them are two exons that generate a novel tripartite mature transcript
Loss of BDNF is a major cause of the developmental abnormalities seen with huntingtin (show HTT ELISA Kits) knockdown in zebrafish.
Together these results suggest an important role of BDNF in the maintenance and regeneration of the olfactory system.
BDNF suppresses neuromuscular junction maturation through cAMP-PKA signaling pathway
Findings demonstrate the neurotrophin, BDNF-dependent formation of integrin beta1-based adhesions in the growth cone and reveal how a positive regulator of substrate adhesions can block the negative remodeling and growth inhibitory effects of myelin-associated glycoprotein (show MAG ELISA Kits) .
These results indicate that brief sensory stimulation, by initiating nuclear transcription and de novo protein synthesis of BDNF, can facilitate the refinement of response properties in the developing visual system.
In the Xenopus melanotrope, BDNF biosynthesis and processing occur along the secretory granule maturation axis, together with that of POMC (show POMC ELISA Kits)-derived alphaMSH (show POMC ELISA Kits), and that the light controls the biosynthesis and secretion of BDNF and of POMC (show POMC ELISA Kits) end-products.
BDNF released from the neural lobe of the pituitary gland acts as a neurohormone stimulating the secretory activity of the melanotrope cells in the intermediate pituitary lobe.
BDNF influences synaptic connectivity in multiple ways, promoting not only the morphological maturation of axonal arbors but also their stabilization, but also their stabilization.
BDNF, in addition to its neural and hormonal roles, can be released as a neurohormone from the neural pituitary lobe of X. laevis
BDNF induces glial cell proliferation as well as axonal outgrowth and myelination in vivo.
PACAP stimulates the expression of BDNF transcript IV.
Study showed that complex relationships exist between BDNF/TrkB (show NTRK2 ELISA Kits) gene expression and interneuron marker gene expression that appear to be dependent on the presence of testosterone at adolescence
In a monkey model, cortical BDNF and activity regulated cytoskeletal-associated protein ARC (show Arc ELISA Kits) expressions are strongly correlated with spontaneous physical activity.
A SNP is present in rhesus macaques and is able to affect BDNF peripheral levels, thus making this primate model a fundamental tool to study gene by environment interactions involving the BDNF gene.
In monkey the decline of the BDNF protein level started earlier in the sensory and motor neocortical areas than in the association neocortical areas.
Results imply that oleanolic acid ameliorates scopolamine-induced memory impairment by modulating the brain-derived neurotrophic factor -ERK1/2 (show MAPK1/3 ELISA Kits)-cAMP response element-binding protein (show CREB ELISA Kits) pathway through tropomyosin receptor kinase (show NTRK1 ELISA Kits) B activation in mice, suggesting that oleanolic acid would be a potential therapeutic agent for the treatment of cognitive deficits.
The decrease in ARC (show NOL3 ELISA Kits) BDNF may support increased feeding in food-deprived mice, whereas the increase in the VMH may moderate overeating in WD-fed mice.
The results of the present study identify BDNF expression within mesocorticolimbic brain structures as a reasonable target for further investigations into the neural mechanisms underlying ABA.
BDNF triggers multiple effects including neuronal intrinsic growth capacity improvement and distal portion atrophy protection to promote behavioral recovery following sciatic nerve crush injury in mouse.
the nicotine-induced decrease in BDNF was inhibited in the presence of mecamylamine. We also detected a3 and b4 mRNA in intestinal mucosal cells and a3 protein expression in intestinal enteroendocrine cells. We conclude that STC-1 (show STC1 ELISA Kits) cells and intestinal enteroendocrine cells express nAChRs.
loss of DTNBP1 (show DTNBP1 ELISA Kits) in pyramidal neurons diminished activity-dependent secretion of brain-derived neurotrophic factor (BDNF).
FTY720-treated Tg mice had reduced aSyn aggregation and less constipation, occurring in part by increasing both pro-BDNF and mature BDNF levels.
Expression levels of brain derived neurotrophic factor, postsynaptic density 95 (show DLGAP2 ELISA Kits), and p-cyclic-AMP response element binding protein (show CREB ELISA Kits) levels were significantly elevated in the testosterone (T) group, but flutamide reduced the T-induced effects in these biomarkers to control levels.
BDNF-overexpression may not only facilitate the recovery of locomotor function via AKT (show AKT1 ELISA Kits) pathway, but also contributed simultaneously to thermal hyperalgesia after spinal cord injury
findings reveal a spine-autonomous, autocrine signalling mechanism involving NMDAR (show GRIN1 ELISA Kits)-CaMKII (show CAMK2G ELISA Kits)-dependent BDNF release from stimulated dendritic spines and subsequent TrkB (show NTRK2 ELISA Kits) activation on these same spines that is crucial for structural and functional plasticity
genetic association/nutrigenomic studies in population in South Korea: Data suggest that an SNP in BDNF (rs6265) is negatively associated with type 2 diabetes; BDNF Val/Met and Met/Met variants (rs6265) decrease risk for glucose intolerance and type 2 diabetes. Middle-aged individuals with BDNF Val/Val are prone to developing type 2 diabetes even with low energy intake and low protein intake.
The results of the present study show that the BDNF Val66Met polymorphism is an important moderator of the relation between child maltreatment and reappraisal ability, and that this G x E interaction fits a strong differential susceptibility model.
Significant associations were identified at 3.5 years old for TMEM18 (show TMM18 ELISA Kits) rs6548238, NEGR1 (show NEGR1 ELISA Kits) rs2815752, BDNF rs10767664 and rs6265 (1 year old and 3.5 years old) with anthropometric phenotypes
Role of BDNF gene variants in susceptibility to schizophrenia and in some aspects of cognitive function.
Significant differences of serum BDNF levels were found between male patients with schizophrenia with and without metabolic syndrome, but not in females. This finding suggested the gender difference behind the mechanism of BDNF in metabolic syndrome in schizophrenia.
BDNF mutations are associated with Obesity.
TPH2 (show TDO2 ELISA Kits) SNP rs4570625 interaction with BDNF Val66Met predicts a high-risk emotional phenotype
Results indicated that phosphorylation of glycogen synthase kinase 3beta at Ser9 might be involved in Human endogenous retrovirus W family envelope (HERV-W env)-induced brain-derived neurotrophic factor expression, and will hopefully improve our understanding of the role of HERV-W env in neurological and psychiatric diseases (schizophrenia, etc).
the prevalence of phantosmia in the general older population is not negligible and that some factors that are beneficial for preserved olfactory function, such as female gender and the BDNF met allele, are also associated with the occurrence of phantom smells.
BDNF 66Met may play unfavorable roles in blood pressure and lipid profiles in the general population in Hongshui River area
These observations provided evidence that brain-derived naeurotrophic factor(BDNF) and its receptor (BDNF receptor) secreted by bovine sperm was important in regulation of insulin (show INS ELISA Kits) and leptin (show LEP ELISA Kits).
Expressed in ganglionic neuron-like tumor cells, which may activate an embryonic pathway involving BDNF.
Lf upregulated several canonical signaling pathways associated with neurodevelopment and cognition and influenced ~10 genes involved in the brain-derived neurotrophin factor (BDNF) signaling pathway.
Taken together, these data indicate that recurrent tethering stress in sows over 4.5 years results in a loss of neurotrophic support by BDNF, mediated by an overactive neuroendocrine system.
FiO2 used for resuscitation affects matrix metalloproteinases MMP-9 (show MMP9 ELISA Kits) and MMP-2 (show MMP2 ELISA Kits), caspase-3 (show CASP3 ELISA Kits) and BDNF
The protein encoded by this gene is a member of the nerve growth factor family. It is induced by cortical neurons, and is necessary for survival of striatal neurons in the brain. Expression of this gene is reduced in both Alzheimer's and Huntington disease patients. This gene may play a role in the regulation of stress response and in the biology of mood disorders. Multiple transcript variants encoding distinct isoforms have been described for this gene.
brain-derived neurotrophic factor
, brain derived neurotrophic factor
, anorexia BDNF
, brain derived neurothrophic factor
, neurotrophic factor