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Human CSF1 ELISA Kit for Sandwich ELISA - ABIN625043
Penolazzi, Pocaterra, Tavanti, Lambertini, Vesce, Gambari, Piva: Human osteoclasts differentiated from umbilical cord blood precursors are less prone to apoptotic stimuli than osteoclasts from peripheral blood. in Apoptosis : an international journal on programmed cell death 2008
Show all 6 references for ABIN625043
Rat (Rattus) CSF1 ELISA Kit for Sandwich ELISA - ABIN416355
Deng, Lu, Ling, Kaur: Microglia-derived macrophage colony stimulating factor promotes generation of proinflammatory cytokines by astrocytes in the periventricular white matter in the hypoxic neonatal brain. in Brain pathology (Zurich, Switzerland) 2010
Mouse (Murine) CSF1 ELISA Kit for Sandwich ELISA - ABIN625417
Chitu, Gokhan, Gulinello, Branch, Patil, Basu, Stoddart, Mehler, Stanley: Phenotypic characterization of a Csf1r haploinsufficient mouse model of adult-onset leukodystrophy with axonal spheroids and pigmented glia (ALSP). in Neurobiology of disease 2015
the high level of colony-stimulating factor 1 expression during bovine pregnancy in uteroplacental tissues is consistent with its proposed role in placental physiology
results suggest that leukemia inhibitory factor (show LIF ELISA Kits) and macrophage-colony stimulating factor (show CSF2 ELISA Kits) are produced in the endometrium and may play different roles in early and mid-pregnancy
These results indicate that BARF1 blockade of CSF-1 signaling is an important immune evasion strategy for efficient acute EBV infection and a significant determinant for virus setpoint during persistent EBV infection.
the complete sequencing of M-CSF (show CSF1R ELISA Kits) genes, gene organizaztion,splice varaiants, gene expression, and M-CSF (show CSF1R ELISA Kits) role in the immune system
M-CSF has been shown to be comparable to CA15-3 and VEGF, specificity, and AUC values only in stages III and IV of BC.
M-CSF macrophage conversion into foam cells reduces their proinflammatory responses to classical M1-polarizing activation
This can be achieved by either blocking the EGF (show EGF ELISA Kits) or CSF-1 receptors or supressing the EGF (show EGF ELISA Kits) or CSF-1 signal.
findings demonstrated that M-CSF binds to IL-34 (show IL34 ELISA Kits); molecular docking studies predicted the formation of a heteromeric M-CSF/IL-34 (show IL34 ELISA Kits) cytokine
RGC-32 (show C13orf15 ELISA Kits) expression on M2-polarized and tumor-associated macrophages is M-CSF-dependent and enhanced by tumor-derived IL-4 (show IL4 ELISA Kits).
Data show crystal structures of CSF1-CSF1R (show CSF1R ELISA Kits) ternary complexes, and propose a mechanism for their cooperative action that relies on the adoption by dimeric CSF-1 of an active conformational state and homotypic receptor interactions.
In patients with CLE (show EIF2B5 ELISA Kits), 100 and 150 mg PD-0360324 (monoclonal antibody against MCSF) every 2 weeks for 3 months suppressed a subset of circulating monocytes.
peripheral nerve injury induced de novo expression of colony-stimulating factor 1 (CSF1) in injured sensory neurons. CSF1 was transported to the spinal cord, where it targeted the microglial CSF1 receptor (CSF1R (show CSF1R ELISA Kits)).
These findings highlight an essential role for PRKAA1 (show PRKAA1 ELISA Kits)-mediated autophagy during differentiation of human monocytes.
These findings suggest a high usefulness of M-CSF in diagnosing the serous sub-type of epithelial ovarian cancer and in discriminating between cancer and non-carcinoma lesions
study shows that PLCgamma1 (show PLCG1 ELISA Kits) controls osteoclast numbers via a CSF-1-dependent DAG/beta-catenin (show CTNNB1 ELISA Kits)/cyclinD1 pathway.
study concludes that Langerhans cells require IL-34 (show IL34 ELISA Kits) when residing in fully differentiated and anatomically intact skin epidermis, but rely on neutrophil-derived CSF1 during inflamma
Proteomic Analysis Reveals Distinct Metabolic Differences Between Granulocyte-Macrophage Colony Stimulating Factor (GM-CSF (show CSF2 ELISA Kits)) and Macrophage Colony Stimulating Factor (show CSF2 ELISA Kits) (M-CSF (show CSF1R ELISA Kits)) Grown Macrophages Derived from Murine Bone Marrow Cells
Hematopoietic cells can be induced by M-CSF (show CSF1R ELISA Kits) to dedifferentiate to multipotent stem cells.
M-CSF (show CSF1R ELISA Kits) promotes macrophagic over granulocytic differentiation by inducing ERK (show EPHB2 ELISA Kits) activation but also PKCd (show PKCd ELISA Kits) expression, which in turn, down-regulates Fli-1 (show FLI1 ELISA Kits) expression and prevents granulocytic differentiation.
Results identify CSF-1-activated macrophages as crucial mediators of detrimental Schwann cell dedifferentiation in Cx32 (show GJB1 ELISA Kits)-deficient mice
Ceramide production in M-CSF (show CSF1R ELISA Kits)-deprived macrophages arises from a combination of ASMase (show SMPD1 ELISA Kits) activity and de novo synthesis.
CSF-1 did not rescue the growth and lung defects associated with hyperoxia in this model; however, an increase in CSF-1R (show CSF1R ELISA Kits)+ macrophages was not associated with an exacerbation of lung injury.
CSF-1 neutralization led to a relatively uniform reduction in all inflammatory cell populations; GM-CSF (show CSF2 ELISA Kits) neutralization resulted in the preferential loss among the monocyte/macrophage populations.
The results of this study indicated that the CSF1 overexpression observed in CNS pathologies likely has pleiotropic influences on microglia.
The protein encoded by this gene is a cytokine that controls the production, differentiation, and function of macrophages. The active form of the protein is found extracellularly as a disulfide-linked homodimer, and is thought to be produced by proteolytic cleavage of membrane-bound precursors. The encoded protein may be involved in development of the placenta. Alternate splicing results in multiple transcript variants.
macrophage colony-stimulating factor 1
, colony stimulating factor 1 (macrophage)
, macrophage colony-stimulating factor 1-like
, colony-stimulating factor 1