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anti-Human VEGF Antibodies:
anti-Mouse (Murine) VEGF Antibodies:
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Human Polyclonal VEGF Primary Antibody for IHC (p), WB - ABIN3044494
Jiang, Han, Li, Yang, Liu: Carboxymethyl chitosan represses tumor angiogenesis in vitro and in vivo. in Carbohydrate polymers 2015
Show all 62 Pubmed References
Human Polyclonal VEGF Primary Antibody for IHC (p), WB - ABIN3042324
Song, Yue, Li, Li, Zhao, Zhang: Study of the mechanism of sonodynamic therapy in a rat glioma model. in OncoTargets and therapy 2014
Show all 61 Pubmed References
Guinea Pig Polyclonal VEGF Primary Antibody for IF (p), IHC (p) - ABIN707186
Wang, Liao, Wang, Deng, Yu: Transplantation of bone marrow stromal cells overexpressing human vascular endothelial growth factor 165 enhances tissue repair in a rat model of radiation-induced injury. in Chinese medical journal 2014
Show all 3 Pubmed References
Human Monoclonal VEGF Primary Antibody for Neut, ELISA - ABIN1003427
Kim, Li, Houck, Winer, Ferrara: The vascular endothelial growth factor proteins: identification of biologically relevant regions by neutralizing monoclonal antibodies. in Growth factors (Chur, Switzerland) 1992
Show all 2 Pubmed References
Human Monoclonal VEGF Primary Antibody for FACS - ABIN4898939
Lee, Myers, Kim: Vascular endothelial growth factor drives autocrine epithelial cell proliferation and survival in chronic rhinosinusitis with nasal polyposis. in American journal of respiratory and critical care medicine 2009
Rat (Rattus) Polyclonal VEGF Primary Antibody for IHC, ELISA - ABIN802829
Zhang, Kong, Chen, Zhang, Lian, Zhu, Lu, Zheng: Peroxisome proliferator-activated receptor-? interrupts angiogenic signal transduction by transrepression of platelet-derived growth factor-? receptor in hepatic stellate cells. in Journal of cell science 2014
Human Polyclonal VEGF Primary Antibody for IF (p), IHC (p) - ABIN675893
Ryzhov, Goldstein, Novitskiy, Blackburn, Biaggioni, Feoktistov: Role of A2B adenosine receptors in regulation of paracrine functions of stem cell antigen 1-positive cardiac stromal cells. in The Journal of pharmacology and experimental therapeutics 2012
Human Monoclonal VEGF Primary Antibody for PLA, ELISA - ABIN521322
Arai, Kawachi, Setiawan, Kobayashi: Hypoxia-selective growth inhibition of cancer cells by furospinosulin-1, a furanosesterterpene isolated from an Indonesian marine sponge. in ChemMedChem 2010
Human Monoclonal VEGF Primary Antibody for CyTOF, FACS - ABIN4900874
Banerjee, Lin, Skinner, Schiffhauer, Peacock, Hicks, Redmond, Morrow, Huston, Shayne, Langstein, Miller-Graziano, Strickland, ODonoghue, De: Heat shock protein 27 differentiates tolerogenic macrophages that may support human breast cancer progression. in Cancer research 2011
Rspo1 (show RSPO1 Antibodies) is required for hematopoietic stem cell specification through control of parallel signaling pathways controlling HSC (show FUT1 Antibodies) specification: Wnt16 (show WNT16 Antibodies)/DeltaC/DeltaD and Vegfa/Tgfbeta1 (show TGFB1 Antibodies)
Tmem2 regulates hyaluronic acid turnover to promote normal Vegf signaling during developmental angiogenesis.
low levels of Vegf signaling promote overall vascular endothelial differentiation and cell survival by upregulating etv2 expression, while high levels of Vegf signaling promote arterial and inhibit venous specification.
altering the amount of VEGF signaling in endothelial cells by stimulating them with different VEGF concentrations triggered distinct and mutually exclusive dynamic Ca(2 (show CA2 Antibodies)+) signaling responses that correlated with different cellular behaviors.
Bis(2,3-dibromo-4,5-dihydroxybenzyl) ether downregulate the expression of VEGF and VEGFR, and simultaneously regulates VEGF signaling pathway to prevent angiogenesis.
noncanonical function of tars (show TARS Antibodies) regulates vascular development presumably by modulating the expression of vegfa
Methylglyoxal acts on smaller blood vessels in zebrafish via the VEGF receptor (show FLT1 Antibodies) signaling cascade, thereby describing a new mechanism that can explain vascular complications under hyperglycemia and elevated MG concentrations.
The translation initiation factor eIF3i (show EIF3I Antibodies) up-regulates VEGF-A, accelerates cell proliferation, and promotes angiogenesis in embryonic development.
lack of SULF1 (show SULF1 Antibodies) expression downregulates VEGFA-mediated arterial marker expression, confirming that Sulf1 (show SULF1 Antibodies) mediates arterial specification by regulating VegfA165 activity.
yolk-derived 17beta-estradiol sets the ventral boundary of hemogenic vascular niche specification by antagonizing the dorsal-ventral regulatory limits of VEGF.
VEGFA produced in the somites is required to initiate adult haemangioblast programming in the adjacent dorsal lateral plate mesoderm.
sequential, isoform-specific VEGFA signaling successively induces the endothelial, arterial, and hematopoietic stem cell programs in the dorsal aorta.
VEGF induces stromal cell migration or recruitment that are required for blood vessel formation.
increased VEGF(170) levels disturb Hand-1 (show HAND1 Antibodies) expression in the region required for normal heart morphogenesis
data for the first time demonstrate a calpain/PTP1B/VEGFR2 negative feedback loop in the regulation of VEGF-induced angiogenesis. Modulation of local PTP1B and/or calpain activities may prove beneficial in the treatment of impaired wound healing in diabetes.
Studied the effect of cyclic uniaxial stretch (20%, 1 Hz), with and without the stimulation of vascular endothelial growth factor (VEGF), on sprouting angiogenesis by employing a stretchable three-dimensional cell culture model.
Study shows that VEGFA mRNA in mammalian endothelial cells undergoes programmed translational readthrough (PTR) generating VEGF-Ax, an isoform containing a unique 22-amino-acid C terminus extension.
Data indicate that superoxide dismutase (SOD) inhibited high glucose (HG)-induced expression of uPAR and VEGF in bovine retinal microvascular endothelial cell (REC).
Exposure of endothelial cells to VEGF, high glucose, or hydrogen peroxide up-regulated the XBP1 (show XBP1 Antibodies)/IRE1 alpha (show ERN1 Antibodies) and ATF6 (show ATF6 Antibodies) arms of the unfolded protein response compared with untreated cells.
Microvascular endothelial cells are more susceptible than aortic cells to advanced glycation end products-enhanced permeability and that AGE-enhanced permeability is dependent on VEGF expression induced by reactive oxygen species.
VEGF supports germ cell survival and sperm production in bulls.
analysis of polymorphisms of bovine VEGF gene and their associations with growth traits in Chinese cattle
VEGF mRNA expression at estrus was higher than at the early I, early II and late luteal stages (P<0.05), whereas VEGF protein content was greatest at the early I luteal stage and decreased thereafter
Alterations in the expression of VEGF-A and bFGF (show FGF2 Antibodies) systems suggest that angiogenic factors are involved in abnormal placental development in cloned gestations, contributing to impaired fetal development and poor survival rates.
Corticotrophin-releasing hormone accelerated tumor angiogenesis by upregulating VEGF expression and secretion in colon cancer cells.
In multivariate analysis, only serum VEGF-A correlated to diabetes duration, whereas VEGF-C (show VEGFC Antibodies) only correlated to HbA1c and fasting blood glucose.
The study aimed to assess the usefulness of the determination of cytokines: IL-8, VEGF and its soluble receptors: VEGF-R1, VEGF-R2 in patients with endometrial cancer. The increased levels of VEGF may be useful in the preoperative assessment of the status of para-aortic lymph nodes.
It is likely that these neuronal factors, under the influence of estrogen, could induce physiological inflammation during cervical remodeling by promoting the expression of vascular endothelial growth factor, among other factors
Our results reveal that aflibercept significantly lowers the amount of unbound VEGF as well as the proliferation rate of HUVEC. Moreover, in contrast to specifications given by the manufacturer, aflibercept retains its full inhibitory effect up to at least 120h after transference from the original vial into the injection syringe.
Since VEGF contributes to disturbed vasculature in chronic obstructive pulmonary disease (COPD (show ARCN1 Antibodies)), altered miR (show MLXIP Antibodies)-503 production might play a role in modulating fibroblast-mediated vascular homeostasis in COPD (show ARCN1 Antibodies).
results showed that fascaplysin inhibited ovarian cancer cell proliferation, invasion and migration, as well as inducing S arrest and cell apoptosis. Treatment with fascaplysin also suppressed CDK4 (show CDK4 Antibodies), cyclin D1 (show CCND1 Antibodies), Bcl-2 (show BCL2 Antibodies), and VEGFA expression at protein levels
Patients with mixed dyslipidaemia have higher plasma VEGF levels. Rosuvastatin monotherapy at high dose reduces VEGF values, whereas a significant increase is observed in patients receiving the combination of low rosuvastatin dose with omega-3 PUFAs.
The variation in physical adaptation to exercise training in diabetes mellitus type 2 was associated with changes in expression of vascular endothelial growth factor A in muscle.
Serum VEGF is a useful marker for prediction of ovarian cancer microvessel density and tumor VEGF expression.
VEGF protein levels were also higher in the ipsilateral hemisphere of WT mice compared to Par-1 (show MARK2 Antibodies) KO mice after glioma cell implantation.
the importance of VEGF derived from tumor-infiltrating myeloid cells for initiating vascularization in gliomas
Low VEGF expression is associated with liver fibrosis.
We conclude that HIF-1 (show HIF1A Antibodies) is not a major regulator of Vegfa expression during wound healing; rather, it serves to maintain basal levels of expression of Vegfa and its target genes in intact skin, which are required for optimal granulation tissue formation in response to wounding.
Mechanical strain stimulates vasculogenesis of embryonic stem cells by the intracellular messengers ROS (show ROS1 Antibodies), NO and calcium as well as by upregulation of angiogenesis guidance molecules and the angiogenic growth factors VEGF, FGF-2 (show FGF2 Antibodies) and PDGF (show PDGFA Antibodies)-BB.
This study identifies YAP/TAZ as central mediators of VEGF signaling
Ectopic midline vascularisation in endothelial Nrp1 (show NRP1 Antibodies) and Vegfa(188/188) mutants caused additional axonal exclusion zones within the chiasm.
RUNX1T1 (show RUNX1T1 Antibodies) serves as a common angiogenic driver for vaculogenesis and functionality of endothelial lineage cells
VEGF inhibition decreases local CFH (show CFH Antibodies) and other complement regulators in the eye and kidney through reduced VEGFR2 (show KDR Antibodies)/PKC-alpha (show PKCa Antibodies)/CREB (show CREB1 Antibodies) signaling.
Vascular endothelial growth factor (VEFG)-A, originally described as an angiogenic factor, belongs to a super-family of glycoproteins, and signals through tyrosine kinase (show TYRO3 Antibodies) receptors VEGF receptor (show FLT1 Antibodies) 1 (VEGFR1 (show FLT1 Antibodies)) and VEGF receptor 2 (VEGFR2) and coreceptors, neuropilin (NP) 1 (show NRP1 Antibodies) and 2.
It was concluded that VEGF and factor VIII are important growth factors associated with fetal development in pigs and are identified in all uterine segments.
results are consistent with a participation of (VEFG) in the regulation of the dynamics of oviductal fluid secretion and the oviduct contractibility
Here we demonstrate that VEGF-165 mediates MSC (show MSC Antibodies) differentiation into ECs via VEGFR-2 (show KDR Antibodies)-dependent induction of Sox18 (show SOX18 Antibodies), which ultimately coordinates the transcriptional upregulation of specific markers of the EC phenotype.
VEGF was significantly downregulated 7 days after cryotherapy of the sclera and stayed at that level until day 14. It returned to baseline by day 21.
VEGF production, blood vessel network and follicle remodeling are impaired by the antiprogesterone RU486.
findings suggest that the augmented neovascular response seen with VEGF administration was through the VEGF-induced upregulation of Notch (show NOTCH1 Antibodies) signaling.
interleukin-1beta-induced vascular endothelial growth factor in airway smooth muscle cells
Upregulation of VEGF during hypoxia in chondrocyte is mediated partially through HIF-1alpha (show HIF1A Antibodies).
data shows that members of the VEGF-VEGFR (show KDR Antibodies) system are temporally and spatially well localized for playing key roles during umbilical cord formation and its intensive growth observed after day 75 of pregnancy
cardiac-specific and hypoxia-induced coexpression of VEGF and Ang1 (show ANGPT1 Antibodies) improves the perfusion and function of porcine MI heart through the induction of angiogenesis and cardiomyocyte proliferation
Peripheral Blood-Derived Mesenchymal Stromal Cells Promote Angiogenesis via Paracrine Stimulation of Vascular Endothelial Growth Factor Secretion
TNF (show TNF Antibodies) may up-regulate VEGF and stimulate angiogenesis in the mare (show C16orf35 Antibodies) early corpus luteum.
After acoustic trauma, vascular endothelial growth factor was up-regulated both in the cochlea and vestibule. Expression in both structures suggests a reparative role with potentially therapeutic implications.
Studied the role of T help 17 cells (Th17) and STAT3-VEGF pathway in pathogenesis of psoriasis.
VEGF mRNA abundance was present at low levels at 16 h post-ovulation and remained low at 72 h, but the level increased at 144 h in uterus.
correlation was observed between CT perfusion parameters (BF, BV, PS, and MAV (show KLHL2 Antibodies)) and expression of VEGF and MVD (show MVD Antibodies) in tumor tissue
Physiologic ischemic training stimulates VEGF-mediated mobilization of endothelial progenitor cells as well as angiogenesis.
Suggest supplemental oxygen inhibits HIF-1alpha/VEGF signaling to reduce smooth muscle proliferation in rabbit arteriovenous fistula model.
Therefore, it was reasonable to speculate that the increased expression of VEGF and MMP-9 (show MMP9 Antibodies) in residual hepatic tumor cells and tumor angiogenesis post-embolization would be responsible for the increased metastatic potentiality and invasiveness.
VEGF expression peaked at 8 weeks after meniscus transplantation
Studied the biocompatibility and neovascularization of the PLGA nanospheres wrapped with vascular endothelial growth factor (VEGF).
VEGF induces TGF-beta1 (show TGFB1 Antibodies) expression and myofibroblast transformation after glaucoma surgery.
The results of this study are promising concerning the role of VEGF as a diagnostic marker in moderate osteoarthritis
The overexpression of VEGF increased tumor growth and vascularization, favored cyst formation, and reduced tumor necrosis.
VEGF plays an important role in choroid-retinal endothelial cell proliferation and tube formation.
These findings suggest that FBLN5 (show FBLN5 Antibodies) may interfere with choroidal neovascularization by downregulating VEGF, CXCR4 (show CXCR4 Antibodies), and TGFB1 (show TGFB1 Antibodies) expression and inhibiting choroidl endothelial cell proliferation.
Apelin may play a role in the development of central retinal vein occlusion (CRVO). Apelin has a unique upstream signaling pathway, independent of the VEGF pathway.
Dll4 play an important role in choroidal neovascularization (CNV) angiogenesis, which appears to be regulated by HIF-1alpha and VEGF during the progression of CNV under hypoxic conditions.
A functional network involving VEGF, IGF2, and MMP9 (show MMP9 Antibodies) in early placental trophoblast cells and maternal endometrium appears to be important for normal placentation.
This gene is a member of the PDGF/VEGF growth factor family and encodes a protein that is often found as a disulfide linked homodimer. This protein is a glycosylated mitogen that specifically acts on endothelial cells and has various effects, including mediating increased vascular permeability, inducing angiogenesis, vasculogenesis and endothelial cell growth, promoting cell migration, and inhibiting apoptosis. Elevated levels of this protein is linked to POEMS syndrome, also known as Crow-Fukase syndrome. Mutations in this gene have been associated with proliferative and nonproliferative diabetic retinopathy. Alternatively spliced transcript variants, encoding either freely secreted or cell-associated isoforms, have been characterized. There is also evidence for the use of non-AUG (CUG) translation initiation sites upstream of, and in-frame with the first AUG, leading to additional isoforms.
, vascular endothelial growth factor A
, vascular endothelial growth factor A-A
, vascular endothelial growth factor A-a
, vascular endothelial growth factor 164
, vascular permeability factor
, vascular endothelial growth factor 188
, angiogenic factor
, Vascular permeability factor
, endothelial growth factor-164
, vascular endothelial growth factor VEGF