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Human VEGFC ELISA Kit for Sandwich ELISA - ABIN411382
Zhao, Sun, Li: Expression and clinical significance of STAT3, P-STAT3, and VEGF-C in small cell lung cancer. in Asian Pacific journal of cancer prevention : APJCP 2012
Show all 11 references for ABIN411382
Mouse (Murine) VEGFC ELISA Kit for Sandwich ELISA - ABIN415601
Zhang, Wang, Gao, Guo, Chen, Wang, Gao, Rao, Chen: Alternatively activated RAW264.7 macrophages enhance tumor lymphangiogenesis in mouse lung adenocarcinoma. in Journal of cellular biochemistry 2009
Show all 2 references for ABIN415601
data not only reveal a non-canonical function of Mt2 (show MT2 ELISA Kits) in angiogenesis, but also propose Mt2 (show MT2 ELISA Kits) as a novel regulator of vegfc expression.
Vegfc signaling increases mafba (show MAFB ELISA Kits) expression to control downstream transcription
Vegfc is dispensable for facial lymphatic sprouting but not for the complete development of the facial lymphatic network.
In the embryo, phenotypes driven by increased Vegfc are suppressed in the absence of Ccbe1 (show CCBE1 ELISA Kits), and Vegfc-driven sprouting is enhanced by local Ccbe1 (show CCBE1 ELISA Kits) overexpression. Moreover, Vegfc- and Vegfr3 (show FLT4 ELISA Kits)-dependent Erk (show MAPK1 ELISA Kits) signaling is impaired in the absence of Ccbe1 (show CCBE1 ELISA Kits).
Vegfc has an essential role in lymphangiogenesis [review]
The parallel growth of motoneuron axons with the dorsal aorta depends on Vegfc/Vegfr3 (show FLT4 ELISA Kits) signaling in zebrafish.
Vegfc acts in two distinct modes during development: as a paracrine factor secreted from arteries to guide closely associated lymphatic vasculature and as an autocrine factor to drive migratory persistence during angiogenesis.
Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.
Here, we show that vascular endothelial growth factor C (Vegfc), an angiogenic as well as a lymphangiogenic factor, is unexpectedly involved in this process in zebrafish.
The development of lymphatic vessels in zebrafish embyros depends on Vegfc signaling.
VEGF-C and VEGF-C156S genes have roles in the pro-lymphangiogenic growth factor therapy of lymphedema
Transcription of the vascular endothelial growth factor C gene (VEGF-C) and translation of the corresponding protein were significantly up-regulated in swine umbilical vein endothelial cells with classical swine fever virus acute infection.
No difference in bioactivity was detected between porcine relaxin-1 (show RLN1 ELISA Kits) and recombinant human relaxin-2 (show RLN1 ELISA Kits) in either mice or rats.
During progressive ischemia, functional and metabolic benefits of intramyocardial VEGF-C gene transfer were apparent. VEGF-C-induced collateral formation occurred at the site of gene transfer
Human lung-resident macrophages express CB1 (show CNR1 ELISA Kits) and CB2 (show CNR2 ELISA Kits) receptors whose activation inhibits the release of angiogenic and lymphangiogenic factors
Immunohistochemical analysis of 122 gliomas showed that TGLI1 expression was positively correlated with VEGF-C, TEM7 (show PLXDC1 ELISA Kits) and microvessel density.
PKM2 expression was correlated with VEGF-C expression, and combination of PKM2 and VEGF-C levels had the better prognostic significance in predicting the poor outcome of patients with breast cancer.
Serum levels of VEGF-C do not appear to predict sentinel lymph node status in patients with early breast cancer who undergo SLNB.
Findings suggest that ILT4 (show LILRB2 ELISA Kits) drives NSCLC development in part on activation of ERK (show EPHB2 ELISA Kits) signaling which in turn upregulates VEGF-C.
There was evidence that HMGB1 (show HMGB1 ELISA Kits) upregulates VEGF-C by activating NF-kappaB (show NFKB1 ELISA Kits) in a colon cancer cell line.
Inhibition of COX-2 (show COX2 ELISA Kits) activity reduced VEGF-C mRNA expression and secretion in cholangiocarcinoma cells and decreased their migration but not proliferation
Our data suggest that Hodgkin and Reed-Sternberg cells produce VEGFC, and VEGFC expression could be a novel prognostic factor in cHL (show CHRDL1 ELISA Kits).
high podoplanin (show PDPN ELISA Kits) expression is associated with aggressive tumor behavior, poor prognosis and metastatic regulation through interaction with VEGF-C.
Data indicate that microRNA miR (show MLXIP ELISA Kits)-101 negatively regulates vascular endothelial growth factor C (VEGF-C) protein expression post-transcriptionally.
The authors show that VEGF-C is necessary for perinatal lymphangiogenesis, but required for adult lymphatic vessel maintenance only in the intestine.
MT1-MMP (show MMP14 ELISA Kits) directly cleaves LYVE-1 (show LYVE1 ELISA Kits) on lymphatic endothelial cells to inhibit LYVE-1 (show LYVE1 ELISA Kits)-mediated lymphangiogenic responses and restrains the production of VEGF-C.
HA increases lymphangiogenesis in renal fibrosis model and also stimulates vascular endothelial cell growth factor (show FGF1 ELISA Kits)-C production from macrophages through Toll-like receptor 4 (show TLR4 ELISA Kits)-dependent signal pathway
Results showed that the VEGF-C/VEGFR-3 (show FLT4 ELISA Kits) system underlies the protective effect of ischemic preconditioning against forebrain ischemia in the mouse hippocampus
Vascular endothelial growth factor C/VEGFR-3 (show FLT4 ELISA Kits) signaling modifies HS and CCL21 (show CCL21 ELISA Kits) gradients around lymphatics, regulating lymphocyte migration.
Coronary artery stem development first requires VEGF-C to stimulate vessel growth around the outflow tract.
Data show that the expression of lymphatic vessel endothelial hyaluronan receptor 1 (LYVE 1 (show LYVE1 ELISA Kits)) was similar with vascular endothelial growth factor C (VEGF-C), but its peak appeared 1-2 d later than that of VEGF-C.
reveal the evolutionary conservation of the lymphatic-like phenotype of the Schlemm's canal (SC), implicate VEGF-C and VEGFR-3 (show FLT4 ELISA Kits) as critical regulators of SC lymphangiogenesis
Sinus venosus-derived and endocardial-derived migratory routes unite to form the coronary vasculature, with the former requiring VEGFC - a tissue-specific mediator of blood endothelial development.
suggest that correction of defective lymphatic function with VEGF-C has potential as a therapeutic strategy for inflammatory bowel disease.
The protein encoded by this gene is a member of the platelet-derived growth factor/vascular endothelial growth factor (PDGF/VEGF) family, is active in angiogenesis and endothelial cell growth, and can also affect the permeability of blood vessels. This secreted protein undergoes a complex proteolytic maturation, generating multiple processed forms which bind and activate VEGFR-3 receptors. Only the fully processed form can bind and activate VEGFR-2 receptors. This protein is structurally and functionally similar to vascular endothelial growth factor D.
vascular endothelial growth factor C
, vascular endothelial growth factor c
, FLT4 ligand DHM
, vascular endothelial growth factor-related protein
, flt4 ligand
, vascular endothelial growth factor C isoform 129
, vascular endothelial growth factor C isoform 184