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GcgR (show GCGR Proteins) knockout (Gcgr (show GCGR Proteins)(-/-)) mice displayed lower blood glucose levels accompanied by elevated plasma ghrelin (show GHRL Proteins) levels. Hyperglycemia was averted in streptozocin treated Gcgr (show GCGR Proteins)(-/-) mice and the plasma ghrelin (show GHRL Proteins) level was further increased.
Findings show that the loss of embryonic GlyRalpha2 ultimately impairs the formation of cortical circuits in the mature brain; results demonstrate a physiological role for alpha2 GlyRs (show GARS Proteins) during cortical network formation and homeostasis
glucagon receptor (show GCGR Proteins) antagonist improves glycemia in diet-induced obese angptl4 (show ANGPTL4 Proteins) knockout mice without increasing glucagon (show GCG Proteins) levels or alpha-cell proliferation, underscoring the importance of this protein.
genetic inactivation of Glra2, the gene coding the alpha2 subunit of GlyRs (show GARS Proteins), disrupts dorsal cortical progenitor homeostasis with an impaired capability of apical progenitors to generate basal progenitors.
Data indicate that the exocrine pancreas in the glucagon receptor Gcgr (show GCGR Proteins)-/- mice exhibited larger nuclear size than in WT or heterozygous controls, most obviously at old ages.
Simultaneous and sufficient activation of GLP1R (show GLP1R Proteins) is required to reduce GCCR (show NR3C1 Proteins) mediated blood glucose elevation following administration of a GLP1R (show GLP1R Proteins)/GCGR (show GCGR Proteins) co-agonist.
Knockdown of liver glucagon receptor (show GCGR Proteins) in mice reduces blood glucose and increases blood LDL levels.
Endogenous activation of Glra2 promotes neuronal migration by regulating nucleokinesis.
Gcgr (show GCGR Proteins)(-/-) mice became lethargic (show CACNB4 Proteins) & cachexic & died early. Autopsy revealed numerous PNETs up to 15 mm in diameter in most well-preserved Gcgr (show GCGR Proteins)(-/-) pancreata.
Data suggest that GcgR (show GCGR Proteins) activation raises hepatic expression of fibroblast growth factor 21 (FGF21 (show FGF21 Proteins)) and increases circulating levels of FGF21 (show FGF21 Proteins); GcgR (show GCGR Proteins) activation induces body weight loss and stimulates lipid metabolism.
investigated neural progenitor cells in respect to their glycine receptor (show GLRB Proteins) function and subunit expression using electrophysiology, calcium imaging, immunocytochemistry, and quantitative real-time PCR
Enhancement of azurophil granule-phagosome fusion via glycine receptor alpha 2 subunit (GlyRalpha2)/transient receptor potential melastatin (show TRPM1 Proteins) (TRPM)2 (show CLU Proteins)/p38 MAP kinase (show MAPK14 Proteins) signaling is a novel target for enhancement of neutrophil bactericidal activity.
Effects of 12 times normal atmospheric pressure of helium-oxygen gas (pressure) on ethanol-induced potentiation of GlyR function in Xenopus oocytes expressing human alpha1, alpha2 or the mutant alpha1(A52S) GlyRs were measured by voltage clamp technics
The molecular basis for the differential sensitivity of GlyR alpha(1) and GlyR alpha(2) to Zn(2+) potentiation is reported.
The glycine receptor consists of two subunits, alpha and beta, and acts as a pentamer. The protein encoded by this gene is an alpha subunit and can bind strychnine. Several transcript variants encoding different isoforms have been found for this gene.
glycine receptor, alpha 2
, glycin receptor,alpha 2
, glycine receptor subunit alpha-2
, glycine receptor subunit alpha-2-like
, glucagon receptor perhaps same as Niddm3
, glycine receptor alpha 2 subunit
, glycine receptor, alpha-2 polypeptide
, Glycine receptor alpha 2 subunit (glycine receptor, neonatal)
, glycine receptor strychnine-binding subunit
, glycine receptor, alpha 2 subunit