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Rabbit CD4 ELISA Kit for Sandwich ELISA - ABIN367419
Li, Feng, Huang, Zhu, Xu, Sui, Xu, Han, Qin: Hematologic and immunological characteristics of Henoch-Schönlein purpura in rat and rabbit models induced with ovalbumin based on type III hypersensitivity. in Scientific reports 2015
Chicken CD4 ELISA Kit for Competition ELISA - ABIN456834
Zhang, Huang, Li, Sui, Wang, Liu, Xu, Yan, Song, Li: Identification and Molecular Characterization of Microneme 5 of Eimeria acervulina. in PLoS ONE 2014
TCF-1-deficient CD4+ CD8+ double positive thymocytes fail to undergo TCR alpha Valpha14-Jalpha18 rearrangement and produce significantly fewer Natural killer T cells.
High-fat diet - induced type 2 diabetes decreases the number of ileum IL17 (show IL17A ELISA Kits)/RORgammaT CD4 T cells.
Results indicate that hypomethylation of Cd4 antigen correlates with stable CD4 expression.
CD4 is expressed in distinct nanoclusters and does not colocalize with T-cell receptor and active protein tyrosine kinase (show YES1 ELISA Kits) p56lck (show LCK ELISA Kits)
This study establishes an important role of IgE in abdominal aortic aneurysms pathogenesis by activating CD4+ T cells, mast cells, and macrophages.
5-kb cis (show CISH ELISA Kits)-element is required in postselection thymocytes for helper lineage commitment, presumably mediating the maintenance of CD4 expression, and suggest that inactivation of the cis (show CISH ELISA Kits)-element by DNA methylation (show HELLS ELISA Kits)
Both type I CD8+ cytotoxic (Tc1) cells and interleukin (IL)-17-producing CD8+ (Tc17) cells mediate effective antitumor immunity through distinct effector mechanisms, but Tc1 cells are superior to Tc17 cells in mediating tumor regression.
Accumulation of CD4 positive effector T cells is a critical step in the progression from mild glomerulonephritis to severe crescentic glomerulonephritis accompanied by tubulointerstitial inflammation and loss of kidney function.
Thymic selection does not appear to play an important role in CD4+CD8+ T cell receptor (TCR)beta repertoire overlap between individuals.
CD4-positive cell deficiency impairs IFN-gamma (show IFNG ELISA Kits) production by CD8 (show CD8A ELISA Kits)-positive effector cells at the site of Mycobacterium tuberculosis infection in mice.
Mouse leukemia cell lines that could express hCD4 and CCR5 (show CCR5 ELISA Kits) were thus established to facilitate normal entry of HIV-1 so that a human CD4/CCR5 (show CCR5 ELISA Kits) transgenic mice cell model can be used to investigate the transmission and pathogenesis of HIV/AIDS and potential antiviral drugs against this disease.
The percentage of lamina propia CD4+LAP+ cells is increased in active ulcerative colitis, showing reduced suppressor activity due to their increased proportion of intracellular IL-17 (show IL17A ELISA Kits) expression.
The study gives insights into the role of CD4 on cell membrane mechanical characteristics.
A decrease of CD4(+) CD25 (show IL2RA ELISA Kits)(+) CD127 (show IL7R ELISA Kits)(low) FoxP3 (show FOXP3 ELISA Kits)(+) regulatory T cells with impaired suppressive function had been found in untreated ulcerative colitis patients.
Redox shuffling of the allosteric disulfide results in previously undescribed conformational changes in CD4 that are likely important for its interaction with its protein partners.
Increased levels of activated and highly susceptible HIV-1 target cells, reduced CD4 and enhanced CXCR4 (show CXCR4 ELISA Kits) cell surface expression, together with the high susceptibility to FAS (show FAS ELISA Kits)-induced programmed cell death may contribute to the rapid CD4+ T cell depletion.
HRB (show HRB ELISA Kits) knock-down affected CD4 downregulation induced by Nef but not by HIV-1 Vpu.
Increased CD4, IL-17 (show IL17A ELISA Kits), and COX-2 (show COX2 ELISA Kits) expression are associated with subclinical inflammation in malar melasma.
Sustained expression of CD83 (show CD83 ELISA Kits) was observed when CD4+ T cells were induced by transforming growth factor-beta to differentiate into CD4+CD25 (show IL2RA ELISA Kits)+ forkhead box P3 (show FOXP3 ELISA Kits)+ regulatory T (iTreg) cells.
CHD in Chinese population is strongly associated with HLA-DRB1*01 and DRB1*04 haplotypes, and formation of CD4(+)CD28(null) T cells was related to HLA-DRB1*01, DRB1*04, and DRB1*15 alleles.
The frequency and expression of a CD4 variant in microminipigs are described.
These data help clarify the regulatory mechanism of DNA methylation (show HELLS ELISA Kits) of the CD4 gene in non-immune cell response to virus replication.
These results suggest that CD4 plays a role in production traits as well as in immune function. The identified SNPs could be used as genetic markers for selection of dairy cows with improved fat percentage. We propose further studies of these SNPs in a larger population as well as further investigations of the function of this gene.
These results suggested that the SNPs in CD4 and STAT5b (show STAT5B ELISA Kits) may be potential genetic markers for SCS (show TWIST1 ELISA Kits) and milk/protein (show CSN2 ELISA Kits) yields selecting and warrant further functional research.
The DNA methylation (show HELLS ELISA Kits) level of the CD4 gene was strongly influenced by mastitis in Chinese Holstein cattle.
These findings revealed that despite the existence of a distinct bovine CD4(+)CD25 (show IL2RA ELISA Kits)(high) T cell population, which showed Foxp3 (show FOXP3 ELISA Kits) transcription/expression, natural regulatory activity did not reside in this cell population
The absence of CD4 T cells results in failure to produce antibodies that neutralize CD4-independent SIV Envs and CD4-pretriggered control SIV Envs.
study found noticeable variation in the first variable region V1 of CD4 and in intron six among the subspecies of chimpanzees.
This gene encodes a membrane glycoprotein of T lymphocytes that interacts with major histocompatibility complex class II antigenes and is also a receptor for the human immunodeficiency virus. This gene is expressed not only in T lymphocytes, but also in B cells, macrophages, and granulocytes. It is also expressed in specific regions of the brain. The protein functions to initiate or augment the early phase of T-cell activation, and may function as an important mediator of indirect neuronal damage in infectious and immune-mediated diseases of the central nervous system. Multiple alternatively spliced transcript variants encoding different isoforms have been identified in this gene.
, T-cell surface glycoprotein CD4
, T-cell differentiation antigen L3T4
, T-cell surface antigen T4/Leu-3
, T-cell surface glycoprotein CD4 precursor (T-cell surface antigen T4/Leu-3) (T-cell differentiation antigen L3T4)
, CD4 antigen (p55)
, CD4 receptor
, W3/25 antigen
, lymphocyte antigen CD4
, cell surface glycoprotein CD4