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Human Vimentin ELISA Kit for Sandwich ELISA - ABIN414529
Stratmann, Engelbrecht, Espelage, Klusmeier, Tiemann, Gawlowski, Mattern, Eisenacher, Meyer, Rabbani, Thornalley, Tschoepe, Poschmann, Stühler: Glyoxalase 1-knockdown in human aortic endothelial cells - effect on the proteome and endothelial function estimates. in Scientific reports 2016
Show all 2 Pubmed References
Human Vimentin ELISA Kit for Sandwich ELISA - ABIN1379905
Leader, Patel, Collins, Henry: Anti-alpha 1-antichymotrypsin staining of 194 sarcomas, 38 carcinomas, and 17 malignant melanomas. Its lack of specificity as a tumour marker. in The American journal of surgical pathology 1987
Human Vimentin ELISA Kit for Sandwich ELISA - ABIN2685880
Dinets, Pernemalm, Kjellin, Sviatoha, Sofiadis, Juhlin, Zedenius, Larsson, Lehtiö, Höög: Differential protein expression profiles of cyst fluid from papillary thyroid carcinoma and benign thyroid lesions. in PLoS ONE 2015
Rat (Rattus) Vimentin ELISA Kit for Sandwich ELISA - ABIN578166
Teshigawara, Kuboyama, Shigyo, Nagata, Sugimoto, Matsuya, Tohda: A novel compound, denosomin, ameliorates spinal cord injury via axonal growth associated with astrocyte-secreted vimentin. in British journal of pharmacology 2013
TRIM56 (show TRIM56 ELISA Kits) is the ubiquitin ligase that is degrading vimentin in ovarian cancer cells, regulation cell migration and neoplasm invasiveness.
TIS21 (show BTG2 ELISA Kits) attenuated Doxorubicin-induced cancer cell senescence by inhibiting linear actin nucleation via Nox4 (show NOX4 ELISA Kits)-ROS (show ROS1 ELISA Kits)-ABI2-DRF (show MPO ELISA Kits) signal cascade
this study indicates that OPN (show SPP1 ELISA Kits) can induce epithelial-mesenchymal transition of hepatocellular carcinoma cells through increasing vimentin stability
The findings support a mechanism in which miR (show MLXIP ELISA Kits)-375 suppresses RUNX1 (show RUNX1 ELISA Kits) levels, resulting in reduced vimentin and L-plastin (show LCP1 ELISA Kits) expression. Knockdown of RUNX1 (show RUNX1 ELISA Kits), L-plastin (show LCP1 ELISA Kits), and vimentin resulted in significant reductions in cell invasion in vitro, indicating the functional significance of miR (show MLXIP ELISA Kits)-375 regulation of specific proteins involved in head and neck squamous cell carcinoma (HNSCC) invasion.
Vimentin expression was an adverse prognostic factor for DSS (show NR0B1 ELISA Kits) in TSCC patients, even after the adjustment for cell differentiation, pathological stage, and expression levels of Snail (show SNAI1 ELISA Kits), Twist, E-cadherin (show CDH1 ELISA Kits), and N-cadherin (show CDH2 ELISA Kits). Snail (show SNAI1 ELISA Kits), E-cadherin (show CDH1 ELISA Kits), N-cadherin (show CDH2 ELISA Kits), and Vimentin were associated with tumorigenesis and pathological outcomes
circulating anti-vimentin IgG autoantibody levels are much greater in idiopathic pulmonary fibrosis subjects than in normal controls
RhoA (show RHOA ELISA Kits)/ROCK and Raf-1 (show RAF1 ELISA Kits)/CK2 (show CSNK2A1 ELISA Kits) pathway are responsible for TNF-alpha (show TNF ELISA Kits)-mediated endothelial cytotoxicity via regulation of the vimentin cytoskeleton.
It was concluded that islet cell expression of vimentin indicates a degree of plasticity and dedifferentiation with potential loss of cellular identity in diabetes.
findings have identified a role for members of these signaling pathways in the regulation of EGF (show EGF ELISA Kits)-induced vimentin expression in the MDA-MB-468 breast cancer cell line
vimentin regulates the differentiation switch via modulation of K5/K14 (show KRT14 ELISA Kits) expression. Moreover, because there was a significant correlation between high vimentin-K14 (show KRT14 ELISA Kits) expression and recurrence/poor survival in oral cancer patients, vimentin-K14 (show KRT14 ELISA Kits) together may prove to be the novel markers for the prognostication of human oral cancer.
Protein phosphatase 1 (show PPP1CB ELISA Kits) is a key protein serine/threonine phosphatase that controls vimentin Ser (show SIGLEC1 ELISA Kits)-56 dephosphorylation in smooth muscle.
These findings identify vimentin as a positive regulator of stemness in the developing mouse mammary gland and in breast cancer cells.
This study is the first to show that vimentin has an important role in tumor metastasis in vivo in the setting of pre-diabetes and endogenous hyperinsulinemia.
These findings identify two specific sites on vimentin that are phosphorylated by Cadmium.
both arthritis-susceptible and -resistant mice can generate cellular and humoral immunity to Vim.
vimentin knockout neurons were insensitive to the axonotrophic effects of Clostridium botulinum C3 exoenzyme
These findings suggest that Plk1 (show PLK1 ELISA Kits) regulates smooth muscle contraction by modulating vimentin phosphorylation at Ser (show SIGLEC1 ELISA Kits)-56.
findings thus show that the inability to produce GFAP (show GFAP ELISA Kits) and Vim affects normal retinal physiology and that the effect of IF deficiency on retinal cell survival differs, depending on the underlying pathologic condition
these findings identify a hereto-unappreciated role for serine-38 phosphorylated vimentin as an important determinant of myofibroblast sensitivity to Withaferin A.
Vimentin expression increased after traumatic brain injury and was positively correlated with edema and neurological impairments.
Knockdown of filamin A (show FLNA ELISA Kits) or vimentin in normal cells profoundly suppresses apical extrusion of the neighbouring transformed cells.
Immunocytochemistry for Vimentin detection in nuclei of IVF (show SCN5A ELISA Kits) and NT bovine embryos
These results indicate that the inner mass differentiates dynamically in blastocysts, as reflected by the expression of vimentin; higher vimentin expression may reflect the higher developmental competence of embryos.
an intact vimentin intermediate filament network contributes to the maintenance of the chondrocyte phenotype
Vimentin- like transcript was expressed in both chordocytes and chordoblasts, whereas the elastin (show ELN ELISA Kits)- like transcript was uniquely expressed in the chordoblasts lining the notochordal sheath.
This study evaluated the expression pattern of vimentin in testes of mature Arabian stallions and correlated its distribution with grade of seminiferous tubule impairment as indicated by a Johnsen score.
As the inner cell mass forms the epiblast, SSEA1 (show FUT4 ELISA Kits) is lost and VIMENTIN is lost and re-expressed.
These observations indicate that vimentin serves as a putative receptor for Japanese encephalitis virus in porcine kidney cells.
Interaction of nucleocapsid protein of transmissible gastroenteritis virus with host vimentin is required for virus infection.
protein(s) that associated with RPTPbeta (show PTPRZ1 ELISA Kits) in response to IGF-I (show IGF1 ELISA Kits) and IGFBP-2 (show IGFBP2 ELISA Kits) in vascular smooth muscle cells
This study demonstrates that maternal VIM, as a genomic protector, is crucial for nuclear reprogramming in porcine cloned embryos.
Vim expression in corneal epithelium is found in a cell population composed of highly motile cells.
This gene encodes a member of the intermediate filament family. Intermediate filamentents, along with microtubules and actin microfilaments, make up the cytoskeleton. The protein encoded by this gene is responsible for maintaining cell shape, integrity of the cytoplasm, and stabilizing cytoskeletal interactions. It is also involved in the immune response, and controls the transport of low-density lipoprotein (LDL)-derived cholesterol from a lysosome to the site of esterification. It functions as an organizer of a number of critical proteins involved in attachment, migration, and cell signaling. Mutations in this gene causes a dominant, pulverulent cataract.
, vimentin 1
, class-III intermediate microfilament