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Rat (Rattus) Vimentin ELISA Kit for Sandwich ELISA - ABIN578166
Teshigawara, Kuboyama, Shigyo, Nagata, Sugimoto, Matsuya, Tohda: A novel compound, denosomin, ameliorates spinal cord injury via axonal growth associated with astrocyte-secreted vimentin. in British journal of pharmacology 2013
This study evaluated the expression pattern of vimentin in testes of mature Arabian stallions and correlated its distribution with grade of seminiferous tubule impairment as indicated by a Johnsen score.
Vimentin- like transcript was expressed in both chordocytes and chordoblasts, whereas the elastin (show ELN ELISA Kits)- like transcript was uniquely expressed in the chordoblasts lining the notochordal sheath.
These results indicate that liprin-alpha1 (show PPFIA1 ELISA Kits) localizes to different adhesion and cytoskeletal structures to regulate vimentin intermediate filament network, thereby altering the invasion and growth properties of the cancer cells.
High vimentin expression is associated with invasion and metastasis of hypoxic non-small cell lung cancer.
High expression of vimentin is associated with glioma.
Our studies first validated that CPEB4 interacts with Vimentin and indicated that high CPEB4 expression in astrocytic tumors correlates closely with a clinically aggressive future, and that CPEB4 might represent a valuable prognostic marker for patients with astrocytic tumors.
Analysis of vimentin and Ki67 (show MKI67 ELISA Kits) expression can straightforwardly be assessed in CTCs from patients with mCRPC. Poorer survival outcomes were observed in vimentin- and Ki67 (show MKI67 ELISA Kits)-positive CTC patients.
Vimentin, Nestin (show NES ELISA Kits) and WT1 (show WT1 ELISA Kits). Sox2 (show SOX2 ELISA Kits) was expressed by the stem/progenitor cells of the ventricular zone, whereas the postmitotic neurons of the cortical plate were immunostained by PAX2 (show PAX2 ELISA Kits) and NSE (show ENO2 ELISA Kits).
immunohistochemical pattern helps to differentiate cystic renal oncocytoma from tubulocystic renal cell carcinoma
The present study therefore indicated that IL-6 (show IL6 ELISA Kits) promoted the process of EMT (show ITK ELISA Kits) in HIBECs as characterized by increased migration and invasion of HIBECs and the typical changes in mRNA and protein expression of the EMT (show ITK ELISA Kits) markers E-cadherin (show CDH1 ELISA Kits) and vimentin.
Data show that over-expression of vimentin ameliorated cytotoxicity of gambogic acid (GA) in HeLa cells.
High vimentin and low beta4 integrin protein levels are associated with poor survival in oral squamous carcinoma patients.
Immunocytochemistry for Vimentin detection in nuclei of IVF (show SCN5A ELISA Kits) and NT bovine embryos
These results indicate that the inner mass differentiates dynamically in blastocysts, as reflected by the expression of vimentin; higher vimentin expression may reflect the higher developmental competence of embryos.
an intact vimentin intermediate filament network contributes to the maintenance of the chondrocyte phenotype
Knockdown of filamin A (show FLNA ELISA Kits) or vimentin in normal cells profoundly suppresses apical extrusion of the neighbouring transformed cells.
findings thus show that the inability to produce GFAP (show GFAP ELISA Kits) and Vim affects normal retinal physiology and that the effect of IF deficiency on retinal cell survival differs, depending on the underlying pathologic condition
these findings identify a hereto-unappreciated role for serine-38 phosphorylated vimentin as an important determinant of myofibroblast sensitivity to Withaferin A.
Vimentin expression increased after traumatic brain injury and was positively correlated with edema and neurological impairments.
Annexin, lamin (show LMNA ELISA Kits), and vimentin were identified as universal dystrophic markers
Astrocytes deficient of GFAP (show GFAP ELISA Kits) and vimentin showed decreased Notch (show NOTCH1 ELISA Kits) signal sending competence and altered expression of Notch (show NOTCH1 ELISA Kits) signaling pathway-related genes
Absence of GFAP (show GFAP ELISA Kits), or both GFAP (show GFAP ELISA Kits) and vimentin, alters Alzheimer's disease-induced changes in gene expression profile of astrocytes, showing a compensation of the decrease of neuronal support genes and a trend for a higher inflammatory expression profile
Vimentin is not only a major organizing element of the intermediate filament network but is also involved in both binding and uptake of C3 exoenzyme.
Vimentin has a role in regulating activation of the NLRP3 (show NLRP3 ELISA Kits) inflammasome
protein(s) that associated with RPTPbeta (show PTPRZ1 ELISA Kits) in response to IGF-I (show IGF1 ELISA Kits) and IGFBP-2 (show IGFBP2 ELISA Kits) in vascular smooth muscle cells
Data suggest MAP kinase (show MAPK1 ELISA Kits)-interacting kinases (Mnk1 (show MKNK1 ELISA Kits), Mnk2 (show MKNK2 ELISA Kits)) regulate cell migration/wound healing, expression of Vim, stability of Vim, and binding of eIF4E (eukaryotic translation initiation factor 4E (show EIF4E ELISA Kits))/Cyfip1 (cytoplasmic FMR1 interacting protein 1 (show CYFIP1 ELISA Kits)).
As the inner cell mass forms the epiblast, SSEA1 (show FUT4 ELISA Kits) is lost and VIMENTIN is lost and re-expressed.
These observations indicate that vimentin serves as a putative receptor for Japanese encephalitis virus in porcine kidney cells.
Interaction of nucleocapsid protein of transmissible gastroenteritis virus with host vimentin is required for virus infection.
This study demonstrates that maternal VIM, as a genomic protector, is crucial for nuclear reprogramming in porcine cloned embryos.
This gene encodes a member of the intermediate filament family. Intermediate filamentents, along with microtubules and actin microfilaments, make up the cytoskeleton. The protein encoded by this gene is responsible for maintaining cell shape, integrity of the cytoplasm, and stabilizing cytoskeletal interactions. It is also involved in the immune response, and controls the transport of low-density lipoprotein (LDL)-derived cholesterol from a lysosome to the site of esterification. It functions as an organizer of a number of critical proteins involved in attachment, migration, and cell signaling. Mutations in this gene causes a dominant, pulverulent cataract.
, vimentin 1
, class-III intermediate microfilament