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Mouse (Murine) TLR4 ELISA Kit for Sandwich ELISA - ABIN424269
Campo, Avenoso, DAscola, Prestipino, Scuruchi, Nastasi, Calatroni, Campo: Inhibition of hyaluronan synthesis reduced inflammatory response in mouse synovial fibroblasts subjected to collagen-induced arthritis. in Archives of biochemistry and biophysics 2012
Show all 13 references for ABIN424269
Human TLR4 ELISA Kit for Sandwich ELISA - ABIN414556
Kacerovsky, Andrys, Hornychova, Pliskova, Lancz, Musilova, Drahosova, Bolehovska, Tambor, Jacobsson: Amniotic fluid soluble Toll-like receptor 4 in pregnancies complicated by preterm prelabor rupture of the membranes. in The journal of maternal-fetal & neonatal medicine 2012
Show all 11 references for ABIN414556
Rat (Rattus) TLR4 ELISA Kit for Sandwich ELISA - ABIN416209
Kusano, Etoh, Inomata, Shiraishi, Kitano: CO(2) pneumoperitoneum increases secretory IgA levels in the gut compared with laparotomy in an experimental animal model. in Surgical endoscopy 2014
This study evaluated the relationship between 11 SNPs of TLR2 (show TLR2 ELISA Kits) and TLR4 and clinical parameters and genetic risk factors for psoriasis vulgaris in a cohort from southern China.
Our observations suggest that exposure of macrophages to Gram-negative bacteria influence the establishment and maintenance of HIV persistence in macrophages through a TLR4-dependent mechanism.
Patients with CD14 (show NDUFA2 ELISA Kits) C (-159) T gene polymorphism, a co-receptor of TLR4, have an increased risk of NAFLD (show TSC2 ELISA Kits) development.
Higher expression of TLR4 and KLF7 (show KLF7 ELISA Kits) may play a vital role in the process of inflammation induced by obesity in visceral adipose tissue
higher TLR4 levels in the early stage of aneurysmal subarachnoid hemorrhage is also associated with the neurological function outcome
TLR4 may play an important role in atherosclerosis and could be a potential therapeutic target for treatment of coronary artery disease.
the increased expression of TLR4 in the responsiveness of LPS (show IRF6 ELISA Kits)-treated THP1 (show GLI2 ELISA Kits) cells occurred in response to the upregulation of their respective receptors, as well as a tight binding of NF-kappaB (show NFKB1 ELISA Kits) in the TLR4 gene promoter.
analysis of TLR2 (show TLR2 ELISA Kits), TLR4 and TLR9 (show TLR9 ELISA Kits) genes and their significance as biological markers in patients with B-cell chronic lymphocytic leukemia
We found lower expression levels of TLR1 (show TLR1 ELISA Kits), TLR3 (show TLR3 ELISA Kits), TLR4, TLR7 (show TLR7 ELISA Kits) and TLR9 (show TLR9 ELISA Kits) in PBMCs from patients with ALL compared with those from control patients. We also observed that the PBMCs from patients with Pre-B and B ALL had lower TLR4 expression than controls
TLR-stimulated pancreatic cancer cells were specifically investigated for activated signaling pathways of VEGF (show VEGFA ELISA Kits)/PDGF (show PDGFA ELISA Kits) and anti-apoptotic Bcl-xL (show BCL2L1 ELISA Kits) expression as well as tumor cell growth.
this study shows that TLR4-dependent, M1 macrophage trafficking/polarization into the CNS as a key mechanistic link between acute traumatic brain injury and long-term, adaptive immune responses
We conclude that upon ligand binding, TLR4 activates PI3K/Akt (show AKT1 ELISA Kits) signaling to induce F-spondin (show SPON1 ELISA Kits) expression, subsequently control CREB (show CREB1 ELISA Kits)-mediated IL-6 (show IL6 ELISA Kits) production to promote VSMC migration. These findings provide vital insights into the essential role of F-spondin (show SPON1 ELISA Kits) in VSMC function and will be valuable for developing new therapeutic strategies against atherosclerosis.
inhibition of mu-opioid receptor expression blocks morphine and DAMGO increases in the translocation of NF-kB p65 protein in microglia.a low dose of morphine, exerting its effects via the mu-opioid receptor, increases the DNA-binding activity of NF-kB via PKCepsilon, while a high dose of morphine triggers a nonopiate receptor response mediated by TLR4 and, interestingly, PKC signalling
Chotosan can ameliorate Abeta1-2-induced memory dysfunction via inhibiting neuroinflammation and apoptosis at least partially mediated by TLR-4/NF-kappaB (show NFKB1 ELISA Kits) signaling pathway.
Toll-Like Receptor 4/MyD88 (show MYD88 ELISA Kits)-Mediated Signaling of Hepcidin (show HAMP ELISA Kits) Expression Causing Brain Iron Accumulation
The TLR4-HMGB1 signaling pathway affects the process of autoimmune myositis inflammation by regulating the expression of MHC-I and other pro-inflammatory cytokines
Orally administered poncirin is metabolized to ponciretin by gut (show GUSB ELISA Kits) microbiota and poncirin and ponciretin attenuates colitis by suppressing NF-kappaB (show NFKB1 ELISA Kits) activation through the inhibition of LPS binding on macrophages and correcting Th17/Treg cell imbalance.
this study shows that Tlr4 can regulate inflammation through regulation of generation of reactive oxygen species in in Aspergillus fumigatus keratitis
These findings suggest that extracellular Prx1 (show PRDX1 ELISA Kits)-mediated TLR4/NF-kappaB (show NFKB1 ELISA Kits) pathway activation probably contributes to neuroinflammatory injury after ICH (show ACE ELISA Kits), and thus blocking Prx1 (show PRDX1 ELISA Kits)-TLR4 signaling might provide a novel anti-neuroinflammatory strategy with extended therapeutic window for hemorrhagic stroke
Results showed that ulinastatin (show AMBP ELISA Kits) protected the brain against ischemic injury which may be due to the alleviation of inflammation reaction in early stage through downregulating TLR4 and NF-kappaB (show NFKB1 ELISA Kits) expression
Transcription levels of TLR2 (show TLR2 ELISA Kits), TLR4, and CD14 (show CD14 ELISA Kits) in Holstein cows with retained placenta significantly decreased between the first and the seventh day postpartum.
Bovine viral diarrhea virus type 2 infection modulates TLR4 responsiveness in differentiated myeloid cells.
TLR2 (show TLR2 ELISA Kits) and TLR4 mediate innate response against Cryptosporidium parvum in bovine intestinal epithelial cells.
TLR4 polymorphisms are associated with susceptibility to Mycobacterium avium ssp. paratuberculosis infection in Holsteins
positive correlation between lower neutrophil apoptosis and higher expression of TLR2 (show TLR2 ELISA Kits) and TLR4 with the formation of NETs and change in surface architecture.
Studied SNPs in the bovine toll-like receptor 4 (TLR4) and monocyte chemo attractant protein-1(CCL2 (show CCL2 ELISA Kits)) genes.
Studied bovine TLR4 gene in mastitis resistance by association as well as expression profiling analysis in crossbred cattle.
Findings indicate that intervertebral disc (IVD (show IVD ELISA Kits)) cells constitutively express TLR4.
Data suggest that granulosa cells from dominant follicles express functional TLR2 (show TLR2 ELISA Kits) and TLR4; granulosa cells appear to participate in innate immunity by responding to bacterial lipopolysaccharides/lipopeptides via TLR2 (show TLR2 ELISA Kits) and TLR4 signaling pathways.
The expressions of host TLR2 (show TLR2 ELISA Kits) and 4 genes were significantly higher in acidosis-resistant steers compared to those in acidosis-susceptible steers.
The expression of TLR4 protein and mRNA, the level of activated NF-kappaB (show NFKB1 ELISA Kits) (p65 (show SYT1 ELISA Kits)) were respectively detected.
Lipopolysaccharide upregulates the expression of rabbit TLR2 (show TLR2 ELISA Kits) and 4 in the uterine body and horn, and the expression of TLR4 in the ovary.
Polydatin might have a protective effect on lung ischemia/reperfusion injury by down-regulating TLR4 and NF-kappaB (show NFKB1 ELISA Kits) expression, then inhibiting the release of mediators of inflammation as ICAM-1 (show ICAM1 ELISA Kits).
TLR4 expression is upregulated in the brain after experimental subarachnoid haemorrhage
The elevated expression of TLR4 was detected after SAH (show ACSM3 ELISA Kits) and peaked on day 3 and 5. TLR4 is increasingly expressed in a parallel time course to the development of cerebral vasospasm in a rabbit experimental model of SAH (show ACSM3 ELISA Kits).
These results further confirm the involvement of the TLR4 signaling pathway in resistance to E. coli F18 (show MAMLD1 ELISA Kits) in Meishan weaned piglets.
Data suggest expression of TLR4 and NFKB (nuclear factor kappa B) are regulated by dietary factors affecting innate immunity; here, Lactobacillus acidophilus in feed down-regulates expression of TLR4 and NFKB in mononuclear cells after LPS (show IRF6 ELISA Kits) challenge.
At 30 days after autotransplantation of a pig kidney, mRNA expression increases for TLR4.
Data suggest TLR2 (show TLR2 ELISA Kits), TLR4, and calcium signaling in enterocytes play principal roles in mucosal immunity against enterotoxigenic Escherichia coli; probiotic Lactobacillus delbrueckii and its extracellular polysaccharides appear to stimulate TLR2 (show TLR2 ELISA Kits)/TLR4.
TLR2 (show TLR2 ELISA Kits) is required for the suppression of TLR4 signaling activation.
The current study screened for single nucleotide polymorphisms (SNPs) in the TLR4 gene and tested their association with Salmonella fecal shedding.
The role of TLR2 (show TLR2 ELISA Kits), TLR4 and RP105 (show CD180 ELISA Kits)/MD1 (show LY86 ELISA Kits) in the immunoregulatory effect of acidic exopolysaccharides from Lactobacillus plantarum N14 (show CLPTM1 ELISA Kits), is reported.
Data suggest expression of TLR4 in liver can be regulated by dietary factors; here, supplementation with aspartate down-regulates expression of TLR4 in liver in a model of liver disease.
Fish Oil attenuates the activation of the HPA (show HPSE ELISA Kits) axis induced by LPS (show IRF6 ELISA Kits) challenge. So it may be associated with decreasing the production of brain or peripheral proinflammatory cytokines through inhibition of TLR4 and NOD signaling pathways in weaned pigs.
Single nucleotide polymorphisms in TLR4 is associated with immune response to gram-negative bacterial infections.
The research findings suggest that Th17 cells are involved in active equine inflammatory bowel disease, and that TLR4 expression was increased in affected horses.
A low steady expression of TLR4, MD-2 (show LY96 ELISA Kits) and CD14 (show CD14 ELISA Kits) mRNA was demonstrated for the intestinal samples with no variation between the intestinal segments analysed.
In the present study, the authors show that TLR4 expression is significantly decreased following the exogenous expression of BPV-1 E2 and E7 in primary equine fibroblasts.
evidence that pulmonary intravascular macrophages are equipped with TLR4 to handle and rapidly respond to circulating endotoxins
TLR4/MD-2 (show LY96 ELISA Kits) complex is responsible for recognition of Rhodococcus spheroides lipopolysaccharide as an agonist in equine cells.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This receptor has been implicated in signal transduction events induced by lipopolysaccharide (LPS) found in most gram-negative bacteria. Mutations in this gene have been associated with differences in LPS responsiveness. Multiple transcript variants encoding different isoforms have been found for this gene.
, homolog of Drosophila toll
, lipopolysaccharide response
, Toll-like receptor4 protein
, Toll-like receptor 4-like protein