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Mouse (Murine) TLR4 ELISA Kit for Sandwich ELISA - ABIN424269
Campo, Avenoso, DAscola, Prestipino, Scuruchi, Nastasi, Calatroni, Campo: Inhibition of hyaluronan synthesis reduced inflammatory response in mouse synovial fibroblasts subjected to collagen-induced arthritis. in Archives of biochemistry and biophysics 2012
Show all 13 references for ABIN424269
Human TLR4 ELISA Kit for Sandwich ELISA - ABIN414556
Kacerovsky, Andrys, Hornychova, Pliskova, Lancz, Musilova, Drahosova, Bolehovska, Tambor, Jacobsson: Amniotic fluid soluble Toll-like receptor 4 in pregnancies complicated by preterm prelabor rupture of the membranes. in The journal of maternal-fetal & neonatal medicine 2012
Show all 11 references for ABIN414556
Rat (Rattus) TLR4 ELISA Kit for Sandwich ELISA - ABIN416209
Kusano, Etoh, Inomata, Shiraishi, Kitano: CO(2) pneumoperitoneum increases secretory IgA levels in the gut compared with laparotomy in an experimental animal model. in Surgical endoscopy 2014
Co-silencing of the TLR-2 (show TLR2 ELISA Kits) and TLR-4 genes inhibited NF- kappaB (show NFKB1 ELISA Kits) expression and TNF-a (show TNF ELISA Kits)/IL-8 (show IL8 ELISA Kits) secretion, while increasing the survival rate of RGCs. Therefore, a high-glucose environment can potentiate the expression of TLR-2 (show TLR2 ELISA Kits) and TLR-4 in RGCs, activate the downstream signaling pathway
Detecting expression changes in TLR4 and MCP2 (show CCL8 ELISA Kits) in the peripheral blood is a feasible method for predicting the occurrence of abortion in women of child-bearing age
The TLR-4 gene abnormalities may trigger inflammation around calcified neurocysticercosis leading to an increase in perilesional edema and provocation of seizures.
Quercetin supplementation enhanced the inhibition of nuclear translocation of NF-kappaB (show NFKB1 ELISA Kits) and the release of cytokines. TLR4 inhibition study confirmed the downstream signaling mechanism mediated by NF-kappaB (show NFKB1 ELISA Kits) which is involved in the oxLDL-induced inflammatory response
Analysis of TLR4 and HBD3 (show DEFB103A ELISA Kits) expression in epithelial cells demonstrates the key role of the innate immune factors in the pathogenesis of inflammatory and destructive periodontal lesions.
this paper shows that HMGB1 (show HMGB1 ELISA Kits)-TLR4 signaling plays an important role in the pathogenesis of upper airway inflammation
this study shows an increased expression of TLR4 in pemphigus and bullous pemphigoid (show DST ELISA Kits) lesions
higher expression of CD14 (show NDUFA2 ELISA Kits)/TLR2 (show TLR2 ELISA Kits) was revealed in patients (90,07%) compared to controls (85,48%) as well as CD14 (show NDUFA2 ELISA Kits)/TLR4 in patients (90,53%) compared to controls (87,25%) (statistically significant difference, p < 0,05). The results of this study could provide new understanding of UTIs' pathogenesis in children.
Methicillin-resistant Staphylococcus aureus infection can enhance non small cell lung cancer cell metastasis by up-regulating TLR4/MyD88 (show MYD88 ELISA Kits) signaling.
the novel role of PFKFB3 (show PFKFB3 ELISA Kits) in induction of paclitaxel resistance by raising lactate production and activating TLR4 signaling, was characterized.
LPS was shown to inhibit osteoblast differentiation by suppressing the expression of ALP, OCN, and Runx2 (show RUNX2 ELISA Kits) in a TLR-4-dependent manner.
In summary, our study illustrates the potential signal transduction process of PTEN while stimulated by LPS: by increasing the association of TLR4, PTEN recruits to its phosphoinositide substrate PI(3,4,5)P3 located on the cell membrane and exerts its dephosphorylated function and subsequently depresses the activity of downstream molecule Akt (show AKT1 ELISA Kits) and results in activation of NF-kappaB (show NFKB1 ELISA Kits), followed by the secretion of inflamma...
Results showed the participation of neuroimmune system activation and the TLR4 signaling response since deficient mice in TLR4 (TLR4-KO) are protected against molecular and behavioral alterations of ethanol in the adolescent brain.
suggest that spinal TLR4 signaling is important for spinal astrocyte activation and astrogliosis that may underlie alloknesis and chronic itch
Stress-induces a broad inflammatory response in mouse hippocampus that involves TLR4, GSK3, and downstream inflammatory signaling, and these stress responses contribute to susceptibility to depression-like behavior in mice.
beta-elemene anti-inflammatory effect is due to the down-regulation of TLR4/MyD88 (show MYD88 ELISA Kits) signaling pathway
Distinct innate immune cell populations are associated with the differences observed in the Tlr4-mutant model.
The data ogf (show PENK ELISA Kits) this study suggested that TLR4 is crucial for positive motivational behavior under approach-avoidance conflict.
WISP1 (show WISP1 ELISA Kits) might contribute to hepatic ischemia reperfusion injury in mice and possibly depends on TLR4/TRIF (show RNF138 ELISA Kits) signaling.
Loss of Toll-like Receptor 4 on Macrophage Promotes the Development of Steatohepatitis-related Hepatocellular Carcinoma.
Transcription levels of TLR2 (show TLR2 ELISA Kits), TLR4, and CD14 (show CD14 ELISA Kits) in Holstein cows with retained placenta significantly decreased between the first and the seventh day postpartum.
Bovine viral diarrhea virus type 2 infection modulates TLR4 responsiveness in differentiated myeloid cells.
TLR2 (show TLR2 ELISA Kits) and TLR4 mediate innate response against Cryptosporidium parvum in bovine intestinal epithelial cells.
TLR4 polymorphisms are associated with susceptibility to Mycobacterium avium ssp. paratuberculosis infection in Holsteins
positive correlation between lower neutrophil apoptosis and higher expression of TLR2 (show TLR2 ELISA Kits) and TLR4 with the formation of NETs and change in surface architecture.
Studied SNPs in the bovine toll-like receptor 4 (TLR4) and monocyte chemo attractant protein-1(CCL2 (show CCL2 ELISA Kits)) genes.
Studied bovine TLR4 gene in mastitis resistance by association as well as expression profiling analysis in crossbred cattle.
Findings indicate that intervertebral disc (IVD (show IVD ELISA Kits)) cells constitutively express TLR4.
Data suggest that granulosa cells from dominant follicles express functional TLR2 (show TLR2 ELISA Kits) and TLR4; granulosa cells appear to participate in innate immunity by responding to bacterial lipopolysaccharides/lipopeptides via TLR2 (show TLR2 ELISA Kits) and TLR4 signaling pathways.
The expressions of host TLR2 (show TLR2 ELISA Kits) and 4 genes were significantly higher in acidosis-resistant steers compared to those in acidosis-susceptible steers.
The expression of TLR4 protein and mRNA, the level of activated NF-kappaB (show NFKB1 ELISA Kits) (p65 (show SYT1 ELISA Kits)) were respectively detected.
Lipopolysaccharide upregulates the expression of rabbit TLR2 (show TLR2 ELISA Kits) and 4 in the uterine body and horn, and the expression of TLR4 in the ovary.
Polydatin might have a protective effect on lung ischemia/reperfusion injury by down-regulating TLR4 and NF-kappaB (show NFKB1 ELISA Kits) expression, then inhibiting the release of mediators of inflammation as ICAM-1 (show ICAM1 ELISA Kits).
TLR4 expression is upregulated in the brain after experimental subarachnoid haemorrhage
The elevated expression of TLR4 was detected after SAH (show ACSM3 ELISA Kits) and peaked on day 3 and 5. TLR4 is increasingly expressed in a parallel time course to the development of cerebral vasospasm in a rabbit experimental model of SAH (show ACSM3 ELISA Kits).
These results further confirm the involvement of the TLR4 signaling pathway in resistance to E. coli F18 (show MAMLD1 ELISA Kits) in Meishan weaned piglets.
Data suggest expression of TLR4 and NFKB (nuclear factor kappa B) are regulated by dietary factors affecting innate immunity; here, Lactobacillus acidophilus in feed down-regulates expression of TLR4 and NFKB in mononuclear cells after LPS (show IRF6 ELISA Kits) challenge.
At 30 days after autotransplantation of a pig kidney, mRNA expression increases for TLR4.
Data suggest TLR2 (show TLR2 ELISA Kits), TLR4, and calcium signaling in enterocytes play principal roles in mucosal immunity against enterotoxigenic Escherichia coli; probiotic Lactobacillus delbrueckii and its extracellular polysaccharides appear to stimulate TLR2 (show TLR2 ELISA Kits)/TLR4.
TLR2 (show TLR2 ELISA Kits) is required for the suppression of TLR4 signaling activation.
The current study screened for single nucleotide polymorphisms (SNPs) in the TLR4 gene and tested their association with Salmonella fecal shedding.
The role of TLR2 (show TLR2 ELISA Kits), TLR4 and RP105 (show CD180 ELISA Kits)/MD1 (show LY86 ELISA Kits) in the immunoregulatory effect of acidic exopolysaccharides from Lactobacillus plantarum N14 (show CLPTM1 ELISA Kits), is reported.
Data suggest expression of TLR4 in liver can be regulated by dietary factors; here, supplementation with aspartate down-regulates expression of TLR4 in liver in a model of liver disease.
Fish Oil attenuates the activation of the HPA (show HPSE ELISA Kits) axis induced by LPS (show IRF6 ELISA Kits) challenge. So it may be associated with decreasing the production of brain or peripheral proinflammatory cytokines through inhibition of TLR4 and NOD signaling pathways in weaned pigs.
Single nucleotide polymorphisms in TLR4 is associated with immune response to gram-negative bacterial infections.
The research findings suggest that Th17 cells are involved in active equine inflammatory bowel disease, and that TLR4 expression was increased in affected horses.
A low steady expression of TLR4, MD-2 (show LY96 ELISA Kits) and CD14 (show CD14 ELISA Kits) mRNA was demonstrated for the intestinal samples with no variation between the intestinal segments analysed.
In the present study, the authors show that TLR4 expression is significantly decreased following the exogenous expression of BPV-1 E2 and E7 in primary equine fibroblasts.
evidence that pulmonary intravascular macrophages are equipped with TLR4 to handle and rapidly respond to circulating endotoxins
TLR4/MD-2 (show LY96 ELISA Kits) complex is responsible for recognition of Rhodococcus spheroides lipopolysaccharide as an agonist in equine cells.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This receptor has been implicated in signal transduction events induced by lipopolysaccharide (LPS) found in most gram-negative bacteria. Mutations in this gene have been associated with differences in LPS responsiveness. Multiple transcript variants encoding different isoforms have been found for this gene.
, homolog of Drosophila toll
, lipopolysaccharide response
, Toll-like receptor4 protein
, Toll-like receptor 4-like protein