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Mouse (Murine) TLR4 ELISA Kit for Sandwich ELISA - ABIN424269
Campo, Avenoso, DAscola, Prestipino, Scuruchi, Nastasi, Calatroni, Campo: Inhibition of hyaluronan synthesis reduced inflammatory response in mouse synovial fibroblasts subjected to collagen-induced arthritis. in Archives of biochemistry and biophysics 2012
Show all 12 references for ABIN424269
Human TLR4 ELISA Kit for Sandwich ELISA - ABIN414556
Kacerovsky, Andrys, Hornychova, Pliskova, Lancz, Musilova, Drahosova, Bolehovska, Tambor, Jacobsson: Amniotic fluid soluble Toll-like receptor 4 in pregnancies complicated by preterm prelabor rupture of the membranes. in The journal of maternal-fetal & neonatal medicine 2012
Show all 9 references for ABIN414556
Rat (Rattus) TLR4 ELISA Kit for Sandwich ELISA - ABIN416209
Kusano, Etoh, Inomata, Shiraishi, Kitano: CO(2) pneumoperitoneum increases secretory IgA levels in the gut compared with laparotomy in an experimental animal model. in Surgical endoscopy 2014
Our data suggested that MIF (show AMH ELISA Kits) affected the intrinsic properties of tumors and the host immune response in tumor microenvironment by regulating the TLR4/HMGB1 (show HMGB1 ELISA Kits) axis, leading to metastasis of breast cancer
an association between TLR4 rs10759931 polymorphism (OR = 0.086, CI: 0.04-0.18, P = <0.00001)and colon cancer; the rs2770150 polymorphism is associated with colon cancer in women aged over 50 years.
expression and activity of TLR2 (show TLR2 ELISA Kits) and 4 in peripheral blood monocytes as well as B cells in in type-2 diabetic subjects; surface expression of TLR2 (show TLR2 ELISA Kits) and 4 was significantly reduced in B cells of the newly diagnosed type-2 diabetic subjects and was associated with decreased cellular activation and cytokine secretion
PAUF mediated dendritic cell activation and immune stimulation are dependent on TLR4
TLR4 signaling initiates microglial IL-6 (show IL6 ELISA Kits) secretion through glioma stem cells, not bulk glioma cells.
Heat shock protein 60 stimulates the migration of vascular smooth muscle cells via Toll-like receptor 4 and ERK MAPK (show MAPK1 ELISA Kits) activation
TAK (show CDK9 ELISA Kits)-242 also significantly suppressed the expression of TLR4, MyD88 (show MYD88 ELISA Kits) and the activation of NF-kappaB (show NFKB1 ELISA Kits) in lungs, suggesting that TAK (show CDK9 ELISA Kits)-242-mediated inhibition occurred largely through the TLR4/NF-kappaB (show NFKB1 ELISA Kits) signal pathway.
PRG4 (show PRG4 ELISA Kits) binds to TLR2 (show TLR2 ELISA Kits) and TLR4 and this binding mediates a novel anti-inflammatory role for PRG4 (show PRG4 ELISA Kits)
Suggest an essential role of TLR4-Src (show SRC ELISA Kits) signaling in macrophages in the pathogenesis of atherosclerosis.
This study describes a novel posttranslational regulation of the HOIP (show RNF31 ELISA Kits)-containing linear-ubiquitin-chain assembly complex (LUBAC)-mediated linear ubiquitination that is critical for specifically directing TLR4-mediated NF-kappaB (show NFKB1 ELISA Kits) activation.
HMGB1 (show HMGB1 ELISA Kits)-TLR2 (show TLR2 ELISA Kits)/TLR4 signaling cascade.
ProTalpha preconditioning-induced prevention of retinal ischemic damage is mediated by selective activation of the TIR-domain-containing adapter-inducing interferon-beta (show IFNB1 ELISA Kits) interferon regulatory factor 3 (show IRF3 ELISA Kits) pathway downstream of toll-like receptor 4 in microglia
in microglia, activation of toll-like receptor 4 (TLR4) by lipopolysaccharide (LPS) down-regulates miR (show MLXIP ELISA Kits)-26a
Ubiquitin-specific Protease 20 Regulates the Reciprocal Functions of beta-Arrestin2 in Toll-like Receptor 4-promoted Nuclear Factor kappaB (NFkappaB) Activation.
Heat shock protein 60 (show HSPD1 ELISA Kits) stimulates the migration of vascular smooth muscle cells via Toll-like receptor 4 and ERK MAPK (show MAPK1 ELISA Kits) activation
Results demonstrate a selective regulation of TLR4 signaling by EGFR (show EGFR ELISA Kits) and highlight the potential use of EGFR (show EGFR ELISA Kits) inhibitors to treat septic shock syndrome.
THe results of this study suggested that enhanced glial TLR4 signalling during disease progression contributes to end-stage ALS pathology in hSOD1 (show SOD1 ELISA Kits)(G93A) mice.
These results evidence the versatility of neuroimaging techniques to monitor the role of toll-like receptor 4 after cerebral ischemia
These results expand on our knowledge of the roles for TLR2 (show TLR2 ELISA Kits) and TLR4 in C. burnetii infection and suggest various roles for these receptors that are dictated by the site of infection.
Bovine viral diarrhea virus type 2 infection modulates TLR4 responsiveness in differentiated myeloid cells.
TLR2 (show TLR2 ELISA Kits) and TLR4 mediate innate response against Cryptosporidium parvum in bovine intestinal epithelial cells.
TLR4 polymorphisms are associated with susceptibility to Mycobacterium avium ssp. paratuberculosis infection in Holsteins
positive correlation between lower neutrophil apoptosis and higher expression of TLR2 (show TLR2 ELISA Kits) and TLR4 with the formation of NETs and change in surface architecture.
Studied SNPs in the bovine toll-like receptor 4 (TLR4) and monocyte chemo attractant protein-1(CCL2 (show CCL2 ELISA Kits)) genes.
Studied bovine TLR4 gene in mastitis resistance by association as well as expression profiling analysis in crossbred cattle.
Findings indicate that intervertebral disc (IVD (show IVD ELISA Kits)) cells constitutively express TLR4.
Data suggest that granulosa cells from dominant follicles express functional TLR2 (show TLR2 ELISA Kits) and TLR4; granulosa cells appear to participate in innate immunity by responding to bacterial lipopolysaccharides/lipopeptides via TLR2 (show TLR2 ELISA Kits) and TLR4 signaling pathways.
The expressions of host TLR2 (show TLR2 ELISA Kits) and 4 genes were significantly higher in acidosis-resistant steers compared to those in acidosis-susceptible steers.
TLR4 and downstream adaptor molecules, transcription factors and cytokines were up-regulated when endometrial epithelial cells were stimulated with LPS (show IRF6 ELISA Kits)
The expression of TLR4 protein and mRNA, the level of activated NF-kappaB (show NFKB1 ELISA Kits) (p65 (show SYT1 ELISA Kits)) were respectively detected.
Lipopolysaccharide upregulates the expression of rabbit TLR2 (show TLR2 ELISA Kits) and 4 in the uterine body and horn, and the expression of TLR4 in the ovary.
Polydatin might have a protective effect on lung ischemia/reperfusion injury by down-regulating TLR4 and NF-kappaB (show NFKB1 ELISA Kits) expression, then inhibiting the release of mediators of inflammation as ICAM-1 (show ICAM1 ELISA Kits).
TLR4 expression is upregulated in the brain after experimental subarachnoid haemorrhage
The elevated expression of TLR4 was detected after SAH (show ACSM3 ELISA Kits) and peaked on day 3 and 5. TLR4 is increasingly expressed in a parallel time course to the development of cerebral vasospasm in a rabbit experimental model of SAH (show ACSM3 ELISA Kits).
Data suggest expression of TLR4 and NFKB (nuclear factor kappa B) are regulated by dietary factors affecting innate immunity; here, Lactobacillus acidophilus in feed down-regulates expression of TLR4 and NFKB in mononuclear cells after LPS (show IRF6 ELISA Kits) challenge.
At 30 days after autotransplantation of a pig kidney, mRNA expression increases for TLR4.
Data suggest TLR2 (show TLR2 ELISA Kits), TLR4, and calcium signaling in enterocytes play principal roles in mucosal immunity against enterotoxigenic Escherichia coli; probiotic Lactobacillus delbrueckii and its extracellular polysaccharides appear to stimulate TLR2 (show TLR2 ELISA Kits)/TLR4.
TLR2 (show TLR2 ELISA Kits) is required for the suppression of TLR4 signaling activation.
The current study screened for single nucleotide polymorphisms (SNPs) in the TLR4 gene and tested their association with Salmonella fecal shedding.
The role of TLR2 (show TLR2 ELISA Kits), TLR4 and RP105 (show CD180 ELISA Kits)/MD1 (show LY86 ELISA Kits) in the immunoregulatory effect of acidic exopolysaccharides from Lactobacillus plantarum N14 (show CLPTM1 ELISA Kits), is reported.
Data suggest expression of TLR4 in liver can be regulated by dietary factors; here, supplementation with aspartate down-regulates expression of TLR4 in liver in a model of liver disease.
Fish Oil attenuates the activation of the HPA (show HPSE ELISA Kits) axis induced by LPS (show IRF6 ELISA Kits) challenge. So it may be associated with decreasing the production of brain or peripheral proinflammatory cytokines through inhibition of TLR4 and NOD signaling pathways in weaned pigs.
Single nucleotide polymorphisms in TLR4 is associated with immune response to gram-negative bacterial infections.
The complete coding sequence of TLR4 gene in Min pig and 3 variants with single point mutations were obtained.
A low steady expression of TLR4, MD-2 (show LY96 ELISA Kits) and CD14 (show CD14 ELISA Kits) mRNA was demonstrated for the intestinal samples with no variation between the intestinal segments analysed.
In the present study, the authors show that TLR4 expression is significantly decreased following the exogenous expression of BPV-1 E2 and E7 in primary equine fibroblasts.
evidence that pulmonary intravascular macrophages are equipped with TLR4 to handle and rapidly respond to circulating endotoxins
TLR4/MD-2 (show LY96 ELISA Kits) complex is responsible for recognition of Rhodococcus spheroides lipopolysaccharide as an agonist in equine cells.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This receptor has been implicated in signal transduction events induced by lipopolysaccharide (LPS) found in most gram-negative bacteria. Mutations in this gene have been associated with differences in LPS responsiveness. Multiple transcript variants encoding different isoforms have been found for this gene.
, homolog of Drosophila toll
, lipopolysaccharide response
, Toll-like receptor4 protein
, Toll-like receptor 4-like protein