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CHN1 and TNFAIP3 are candidate biomarkers for esophageal squamous cell carcinomas
These results identify the ubiquitin-editing enzyme A20 as a novel endogenous mechanism for negative regulation of fibrotic response intensity
Our data propose an additional novel mechanism to explain the known NF-kappaB inhibitory effects of A20: by affecting p105 ubiquitination and subsequently its degradation and limited processing.
A20 promotes metastasis of aggressive basal-like breast cancers through multi-monoubiquitylation of Snail1 (show SNAI1 ELISA Kits).
Low TNFAIP3 expression is associated with colitis-associated colorectal cancer.
The miR-17-92 is a critical contributor to CML (show BCR ELISA Kits) leukemogenesis via targeting A20 and activation of NF-kappaB (show NFKB1 ELISA Kits) signaling.
IM could upregulate A20 protein to inhibit the activation of NF-kappaB (show NFKB1 ELISA Kits) pathway in Jurkat T cells, which was independent of the ABIN1 (show TNIP1 ELISA Kits) protein.
this meta-analysis confirms that TNFAIP3 gene polymorphisms may play important roles in the pathogenesis of Rheumatoid arthritis
Report a significant association between the biased usage of IGHV4-34 (binds to the carbohydrate I/i antigens) and inactivating mutation of TNFAIP3 [encoding a global negative regulator of the canonical nuclear factor-kappaB (NF-kappaB (show NFKB1 ELISA Kits)) pathway] in ocular adnexal MALT lymphomas.
TNFAIP3 rs10499194 T allele and CT genotype were associated with an increased risk for AIH-1 (show AMELX ELISA Kits)
These results suggest that Leishmania exploits A20 and UCP2 (show UCP2 ELISA Kits) to impair inflammasome activation for disease propagation
holding thymic production of natural Treg cells in check, A20 integrates Treg cell activity and increased effector T cell survival into an efficient CD4 (show CD4 ELISA Kits)(+) T cell response
these studies establish A20 as a crucial hepatoprotective factor.
These results suggested that A20 is involved in regulating intracerebral hemorrhage-induced inflammatory injury in both the central and peripheral system
the ubiquitin-modifying enzyme TNFAIP3/A20, an upstream regulator of T cell receptor (TCR) signaling in T cells, is an essential cell-intrinsic regulator of Natural killer T differentiation.
these data reveal a crucial role of A20 in regulating the immunomodulatory activities of mesenchymal stem cells.
Therefore, A20 acts as a novel endogenous regulator of STAT1 (show STAT1 ELISA Kits) that prevents onset of enthesitis.
This study demonstrated that miR (show MLXIP ELISA Kits)-136-5p affected the expression of A20 in IL-17 (show IL17A ELISA Kits)-stimulated astrocytes.
Our data suggest that NAOs have a beneficial preventive effect in septic shock correlated with the enhancement of IL-10 (show IL10 ELISA Kits) via the induction of A20 and COX-2 (show COX2 ELISA Kits).
Data show the characterization and deduced amino acid sequence analysis for A20 gene which expression increases in MDBK cells infected by BVDV-1. Also, regulatory regions for NF-kappaB (show NFKB1 ELISA Kits) and Sp-1 (show SP1 ELISA Kits) additionally to an ARE element for mRNA stability were detected
This gene was identified as a gene whose expression is rapidly induced by the tumor necrosis factor (TNF). The protein encoded by this gene is a zinc finger protein and ubiqitin-editing enzyme, and has been shown to inhibit NF-kappa B activation as well as TNF-mediated apoptosis. The encoded protein, which has both ubiquitin ligase and deubiquitinase activities, is involved in the cytokine-mediated immune and inflammatory responses. Several transcript variants encoding the same protein have been found for this gene.
tumor necrosis factor, alpha-induced protein 3
, OTU domain-containing protein 7C
, TNF alpha-induced protein 3
, putative DNA-binding protein A20
, tumor necrosis factor alpha-induced protein 3
, tumor necrosis factor inducible protein A20
, zinc finger protein A20
, tumor necrosis factor induced protein 3