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anti-Mouse (Murine) WNT3A Antibodies:
anti-Human WNT3A Antibodies:
anti-Rat (Rattus) WNT3A Antibodies:
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Human Polyclonal WNT3A Primary Antibody for IF (p), IHC (p) - ABIN733178
Gao, Yang, Gao, Xu, Niu, Li, Wen: Salvianolic acid B improves bone marrow-derived mesenchymal stem cell differentiation into alveolar epithelial cells type I via Wnt signaling. in Molecular medicine reports 2015
Show all 4 Pubmed References
Human Polyclonal WNT3A Primary Antibody for ELISA, WB - ABIN2477145
Skalská: [Use of linear logistic regression in classification of observations]. in Sborník v?deckých prací Léka?ské fakulty Karlovy univerzity v Hradci Králové. Supplementum 1991
Show all 2 Pubmed References
Human Polyclonal WNT3A Primary Antibody for IHC (p), ELISA - ABIN2477144
Lai, Cheng, Cheng, Feasel, Beste, Peng, Nusrat, Moreno: SOX4 interacts with plakoglobin in a Wnt3a-dependent manner in prostate cancer cells. in BMC cell biology 2011
Show all 2 Pubmed References
Human Monoclonal WNT3A Primary Antibody for ELISA, ICC - ABIN449669
Denysenko, Annovazzi, Cassoni, Melcarne, Mellai, Schiffer: WNT/β-catenin Signaling Pathway and Downstream Modulators in Low- and High-grade Glioma. in Cancer genomics & proteomics 2015
Cow (Bovine) Polyclonal WNT3A Primary Antibody for IHC, WB - ABIN2787841
Chiquet, Blanton, Burt, Ma, Stal, Mulliken, Hecht: Variation in WNT genes is associated with non-syndromic cleft lip with or without cleft palate. in Human molecular genetics 2008
These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (show CTNNB1 Antibodies) and link eNOS (show NOS3 Antibodies)-derived NO to the modulation by VEGF (show VEGFA Antibodies) of Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies)-induced endothelial cell proliferation.
These results indicate that PEDF (show SERPINF1 Antibodies) counters Wnt (show WNT2 Antibodies) signaling to allow for osteoblast differentiation and provides a mechanistic insight into how the PEDF (show SERPINF1 Antibodies) null state results in OI type VI.
These results suggest novel mechanisms for Wnt3a-induced osteoblast proliferation and cell survival via Npnt (show NPNT Antibodies) gene expression
this study shows that the Wnt3a expression levels in dendritic cells influences the generation of memory T cells after 5 days in co-culture with naive T cells through activation of the Wnt (show WNT2 Antibodies) canonical pathway
Wnt3a induces Osx (show SP7 Antibodies) expression via p38 MAPK (show MAPK14 Antibodies) signaling in dental follicle cells. Wnt3a-induced Osx (show SP7 Antibodies) expression was inhibited in the presence of p38 mitogen-activated protein kinase (show MAPK14 Antibodies) (MAPK (show MAPK1 Antibodies)) inhibitors (SB203580 and SB202190) at gene and protein levels, as assessed by real-time PCR and immunocytohistochemistry, respectively.
Furthermore, pigment epithelium-derived factor (PEDF (show SERPINF1 Antibodies)), a secreted glycoprotein known for its anti-tumor properties, blocked Wnt3a-directed induction of autophagy proteins. Autophagy inhibition was complemented by reciprocal regulation of the oxidative stress enzymes, superoxide dismutase 2 (SOD2 (show SOD2 Antibodies)) and catalase (show CAT Antibodies).
Ginkgo biloba exocarp extract inhibits tumor angiogenesis, which may be closely relevant to its effect in blockage of Wnt /beta-catenin-VEGF signaling pathway in Lewis lung carcinoma.
Western blot analysis demonstrated that following BMP2 (show BMP2 Antibodies) and BMP7 (show BMP7 Antibodies) cotransfection of MC3T3E1 cells, the protein expression levels of BMP2 (show BMP2 Antibodies), BMP4 (show BMP4 Antibodies), BMP6 (show BMP6 Antibodies), BMP7 (show BMP7 Antibodies), BMP9 (show GDF2 Antibodies) and Wnt3a were increased compared with control cells
Wnt3a promotes macrophage-mediated bacterial killing by elevating CRAMP and BD1 (show DEFB1 Antibodies) levels.
Wnt3a acutely reduces nuclear acetyl-CoA (show LPCAT1 Antibodies), the necessary substrate for histone acetyltransferases, resulting in a global decrease in histone acetylation.
PEGylated Wnt3A liposomes associated with skeletal stem cell populations in human bone marrow and promoted osteogenesis.
The AChE plays a role in osteoblastic differentiation and is regulated by both Wnt3a and Runx2 (show RUNX2 Antibodies).
In promoting the self-renewal symmetric division of hTERT(high) prostate cancer cells, WNT3a dramatically decreased the ratio of hTERT(high) prostate cancer cells undergoing asymmetric division. Increased WNT (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) signal activation was also detected in hTERT(high) prostate cancer cells. hTERT-mediated CSC properties were at least partially dependent on beta-catenin (show CTNNB1 Antibodies).
the results indicate neighboring structural elements within full-length Wnt3a affect saposin-like subdomain (SLD) conformational stability. Moreover, SLD function(s) in Wnt (show WNT2 Antibodies) proteins appear to have evolved away from those commonly attributed to SAPLIP family members.
Data suggest that PORCN (show PORCN Antibodies) exhibits substrate specificity that includes a Wnt3a peptide fragment (residues 199-219, with disulfide bonds); recombinant PORCN (show PORCN Antibodies) containing a point mutation (R228C) associated with focal dermal hypoplasia exhibits impaired acylation activity toward Wnt3a peptide fragment. (PORCN (show PORCN Antibodies) = porcupine (show PORCN Antibodies) O-acyltransferase; Wnt3a = Wnt (show WNT2 Antibodies) family member 3A)
CPE (show CPE Antibodies) through its N'-terminal sequence, forms aggregates with Wnt3a and possible endoplasmic reticulum (ER) stress leading to its loss of function.
Our findings suggest that Pyk2 (show PTK2B Antibodies) plays an important role in the coordination of stabilization of beta-catenin (show CTNNB1 Antibodies) in the crosstalk between Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) and Wnt (show WNT2 Antibodies)/Ca(2 (show CA2 Antibodies)+) signaling pathways upon Wnt3a stimulation in differentiating hNPCs.
Macrophage polarization seems to have a key role in the progression of pediatric non-alcoholic fatty liver disease; the modulation of macrophage polarization could drive hepatic progenitor cell response by Wnt3a production and beta-catenin (show CTNNB1 Antibodies) phosphorylation.
this study shows that Wnt3a promotes differentiation of dendritic cells, but inhibits their maturation
Taken together, our results suggest that canonical Wnt signaling and its antagonist, sFRP1, regulate proliferation of human CSCs. Furthermore, excess sFRP1 in elderly patients causes CSC aging.
Data suggest that Wnt3, Wnt3a, and Wnt8a (show WNT8A Antibodies) bind to their respective receptors (Fz8 (show FZD2 Antibodies), Lrp6 (show LRP5 Antibodies), and Lypd6 (show LYPD6 Antibodies)) in ordered plasma membrane environments; ordered plasma membrane environments appear to be essential for binding of Wnt (show WNT2 Antibodies) proteins to their receptor complexes and stimulation of downstream signaling activity.
It was shown that Wnt3a-Wnt8a (show WNT8A Antibodies)/beta-catenin (show CTNNB1 Antibodies) signaling directly regulates ciliogenic transcription factor foxj1a expression and ciliogenesis in zebrafish Kupffer's vesicle.
In zebrafish embryos lacking Wnt3a, Wnt1 (show WNT1 Antibodies) and Wnt10b (show WNT10B Antibodies), the expression of engrailed orthologs, pax2a and fgf8 (show FGF8 Antibodies) is not maintained after mid-somitogenesis
data suggest a specific role for Wnt3a in the development of cardiac NCCs; propose that this function of wnt3a in r6 is partially mediated by crip2 (show CRIP2 Antibodies) expression in the premigratory cardiac NCCs, which subsequently affects cardiac function and PA patterning
Sulf1 (show SULF1 Antibodies) does not affect Wnt3a-mediated activation of canonical Wnt (show WNT2 Antibodies) signaling.
Wnt3a protein alone is sufficient to rescue the severe loss of inner ear structures resulting from dorsal but not ventral half ablations.
hindbrain-repressive Wnt3a/Meis3/Tsh1 circuit promotes neuronal differentiation and coordinates tissue maturation
Wnt3a thus acts downstream of FAK (show PTK2 Antibodies) to balance anterior-posterior cell fate specification in the developing neural plate
Data suggest a new model for neural anteroposterior patterning, in which Wnt3a from the paraxial mesoderm induces posterior cell fates via direct activation of a crucial transcription factor in the overlying neural plate.
Wnt3A secretion from tectal cells along with ephrin-B1 (show EFNB1 Antibodies) signaling are specifically responsible for enhanced neural responses in the developing optic tectum.
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene is a member of the WNT gene family. It encodes a protein which shows 96% amino acid identity to mouse Wnt3A protein, and 84% to human WNT3 protein, another WNT gene product. This gene is clustered with WNT14 gene, another family member, in chromosome 1q42 region.
wingless-type MMTV integration site family, member 3A
, protein Wnt-3a-like
, protein Wnt-3a
, vestigial tail
, wingless-type MMTV integration site family, member 3 like
, wnt3 like
, wingless-type MMTV integration site family, member 3
, Wnt-3a homolog
, Wnt3a variant 3
, wingless-type MMTV integration site family member 3a
, wingless-related MMTV integration site 3A