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FOXN3 is a member of the forkhead/winged helix transcription factor family. Additionally we are shipping FOXN3 Proteins (6) and many more products for this protein.
Showing 10 out of 36 products:
Dog (Canine) Polyclonal FOXN3 Primary Antibody for WB - ABIN2777625
Yu, Zhang, Zhou, Wu, Qu, Wei, Xing, Dong, Zhai, Wan, Ouyang, Li, Zhang, Zhou, Zhang, Wu, He: Gene expression profiling in human fetal liver and identification of tissue- and developmental-stage-specific genes through compiled expression profiles and efficient cloning of full-length cDNAs. in Genome research 2001
Show all 2 Pubmed References
FOXN3 bind to beta-catenin and inhibited beta-catenin/TCF signaling by blocking the interaction between beta-catenin and TCF4. Loss of FOXN3 in colon cancer activates beta-catenin/TCF signaling and promotes the growth and migration of cancer cells.
FOXN3 functions as a tumor suppressor in hepatocellular carcinoma by downregulating the expression of E2F5.
the FOXN3-NEAT1-SIN3A complex promotes epithelial-to-mesenchymal transition and invasion of breast cancer cells in vitro as well as dissemination and metastasis of breast cancer in vivo
the rs8004664 risk allele drives excessive expression of FOXN3 during fasting and that FOXN3 regulates fasting blood glucose.
Foxn3 is a direct transcriptional suppressor of N-cadherin in colorectal metastasis.
MEN1 patients with MEN1 mutations leading to CHES1-loss of interaction have a higher risk of malignant pancreatic neuroendocrine tumors with an aggressive course of disease and disease-related death.
CHES1 decreases protein synthesis and cell proliferation in tumor cell lines but not in normal fibroblasts.
MicroRNA-574-5p has a critical role in TLR9 signaling enhanced tumor progression via down-regulating checkpoint suppressor 1
the Foxn3 mutation leads to partial embryonic and postnatal lethality, growth retardation, eye formation defects, dental anomalies and craniofacial defects.
Data suggest that CHES1 recruits Ski-interacting protein (SKIP) to repress genes important for tumorigenesis and the response to cancer treatments.
Meninis involved in the activation of S-phase arrest in multple endocrein neoplasia by interacting with CHES1.
Two novel homozygously deleted genes in hepatocellular carcinomas are caspase 3 and CHES1.
FOXN3 might contribute to the observed phenotype.
Four genes previously not examined in that respect in laryngeal carcinoma, occurred to be good markers of the neoplasm. They are: metal-proteinase ADAM12, cyclin-dependent kinase 2-CDK2, kinesin 14-KIF14, suppressor 1 of checkpoint-CHES1.
MIRN574 microRNA, mouse/*METAB
ARHGEF10 and FOXN3 are genes potentially involved in hair development and growth
FoxN3 regulates craniofacial and eye development by recruiting histone deacetylase complexes .
This gene is a member of the forkhead/winged helix transcription factor family. Checkpoints are eukaryotic DNA damage-inducible cell cycle arrests at G1 and G2. Checkpoint suppressor 1 suppresses multiple yeast checkpoint mutations including mec1, rad9, rad53 and dun1 by activating a MEC1-independent checkpoint pathway. Alternative splicing is observed at the locus, resulting in distinct isoforms.
checkpoint suppressor 1
, forkhead box protein N3
, forkhead box N3
, forkhead box protein FoxN3b
, checkpoint suppressor 1-like
, forkhead box protein N3-like
, forkhead box protein FoxN3a