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The protein encoded by FUT6 is a Golgi stack membrane protein that is involved in the creation of sialyl-Lewis X, an E-selectin ligand. Additionally we are shipping Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Kits (10) and Fucosyltransferase 6 (Alpha (1,3) Fucosyltransferase) Proteins (4) and many more products for this protein.
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data suggest that both FUT5 and FUT6 can promote the development of colorectal cancer (CRC) via the PI3K/Akt signalling pathway, which is regulated by miR-125a-3p. miR-125a-3p may serve as a predictive biomarker and a potential therapeutic target in CRC treatment.
meta-analysis identified new variants, rs3760775 (P = 1.2 x 10-23) and rs78060698 (P = 8.3 x 10-17) in FUT6 to be associated with circulating B12 concentrations.
ur results strongly showed that the low expression of FUT6 regulated by miR-106b contributed to cell migration, invasion, and proliferation in human breast cancer.
There were significant differences between the type 2 diabetes mellitus patients and controls in the risk allele distributions of rs3792267 (CAPN10) (P = 0.002), rs1501299 (APM1) (P = 0.017), and rs3760776 (FUT6) (P = 0.031).
The T allele of rs3760776 might repress the transcription of the FUT6 gene.
In normal colon samples a significant relationship between sLe(x) expression and the ratio between FUT6/B4GALNT2 activities exists, demonstrating for the first time a role for B4GALNT2 in sLe(x) inhibition in vivo
Results suggest that FUT4-, FUT6- or FUT8-mediated MDR in human HCC is associated with the activation of the PI3K/Akt pathway and the expression of MRP1.
these results suggest that FUT6 plays an important role in hepatocellular carcinoma growth by regulating the PI3K/Akt signaling pathway.
The biosynthesis of the selectin-ligand sialyl Lewis x in colorectal cancer tissues is regulated by fucosyltransferase VI and can be inhibited by an RNA interference-based approach.
GALNT14 and FUT3/6 H-scores were significantly higher in non-small cell lung cancer cell lines sensitive to dulanermin and drozitumab versus resistant cell lines
endo-beta-galactosidase-sensitive and mAb FH6-reactive carbohydrate chains are generated under the control of expression levels of FUT6 and involved in the adhesion of colon carcinoma cells to liver sections.
5'UT-exons A or C have higher expression of FUT8 transcripts and higher alpha6-fucosyltransferase activity
IL-1 beta-induced sLeX expression on HuH-7 cells was suppressed by transfection of gene-specific small interference RNAs against FUT VI and ST3Gal IV but not against FUT IV and ST3Gal III.
two defined regions in the 5'-flanking region of the FUT VI transcription start site are critical for FUT VI transcription in HepG2 cells
results suggested that PKR is the primary target for HSV-1 early RNA during induction of FUT3, FUT5, and FUT6
characterisation of FUT4 and FUT6 alpha-(1 --> 2)-fucosyltransferases
FUT6 contributed to fucosylation of root arabinogalactan-proteins.
AtFUT4 and AtFUT6 are the first enzymes to be characterized for AGP glycosylation
The protein encoded by this gene is a Golgi stack membrane protein that is involved in the creation of sialyl-Lewis X, an E-selectin ligand. Mutations in this gene are a cause of fucosyltransferase-6 deficiency. Two transcript variants encoding the same protein have been found for this gene.
, fucosyltransferase VI
, galactoside 3-L-fucosyltransferase
, fucosyltransferase 3 (galactoside 3(4)-L-fucosyltransferase, Lewis blood group)
, alpha (1,3) fucosyltransferase
, fucosyltransferase 3 (galactoside 3(4)-L-fucosyltransferase, Lewis blood group included)
, alpha 1,3 fucosyltransferase
, blood group Lewis alpha-4-fucosyltransferase
, fucosyltransferase III
, galactoside 3(4)-L-fucosyltransferase
, lewis FT
, alpha 1,3/4 fucosyltransferase Lewis 1
, fucosyltransferase 6 (alpha (1,3) fucosyltransferase)
, fucosyltransferase 6 S homeolog