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MOK belongs to the MAP kinase superfamily. Additionally we are shipping MOK Antibodies (60) and many more products for this protein.
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our results suggest MOK promoter hypomethylation is a common event and contributes to MOK overexpression in acute myeloid leukemia (show BCL11A Proteins)
The findings indicate a statistically significant association of p.Gly82Ser polymorphism in RAGE (show AGER Proteins) with DR in T2DM patients.
the expressions of ICK/MAK (show MAK Proteins)/MOK proteins in the intestinal tract can be differentially and dynamically regulated, implicating a significant functional diversity within this group of protein kinases.
Our results suggest that RAGE (show AGER Proteins) may be important in tumor invasion and could be a potential predictor for the prognosis of hepatocellular carcinoma patients.
PBMNC from type 2 diabetics were more sensitive to innate immune stimulation with LPS (show IRF6 Proteins) and monoclonal agonist anti-TLR4 (show TLR4 Proteins) than were cells from ND. The actions of LPS (show IRF6 Proteins), anti-TLR4 (show TLR4 Proteins) and anti-RAGE (show AGER Proteins) potentiated the production of IL-6 (show IL6 Proteins) and TNF-alpha (show TNF Proteins) in both groups.
The RAGE (show AGER Proteins) pathway may play an important role in STAT3 (show STAT3 Proteins) induction in glioma-associated macrophages and microglia, a process that may be mediated through S100B (show S100B Proteins).
RAGE (show AGER Proteins) was demonstrated in all 8 yolk sac (show ADCY10 Proteins) tumors and 21 of 26 embryonal carcinomas. In yolk sac (show ADCY10 Proteins) tumors, RAGE (show AGER Proteins) reactivity was diffusely present throughout the tumors. In embryonal carcinomas, RAGE (show AGER Proteins) was identified only in yolk sac (show ADCY10 Proteins) components
identification of MOK, a member of the mitogen-activated protein kinase (show MAPK1 Proteins) superfamily, as one of the genes induced by a caudal-related homeobox (show Lbx1 Proteins) transcription factor, Cdx2 (show CDX2 Proteins)
May provide suitable targets for immunotherapy of renal cell carcinoma.
Compared RAGE (show AGER Proteins) and PAX-2 (show PAX2 Proteins) staining in metastatic clear renal cell carcinoma.
identification of MOK, a member of the mitogen-activated protein kinase (show MAPK1 Proteins) superfamily, as one of the genes induced by a caudal-related homeobox (show PRRX1 Proteins) transcription factor, Cdx2 (show CDX2 Proteins)
RAGE (show AGER Proteins) inactivation inhibits the atherosclerosis through reducing oxLDL-induced pro-inflammatory responses and oxidative stress in hyperlipidaemia.
Suppression of Egr-1 (show EGR1 Proteins) may contribute to the protective mechanisms underlying the beneficial impact of RAGE (show AGER Proteins) blockade or deletion in hepatic ischemia/reperfusion injury.
This gene belongs to the MAP kinase superfamily. The gene was found to be regulated by caudal type transcription factor 2 (Cdx2) protein. The encoded protein, which is localized to epithelial cells in the intestinal crypt, may play a role in growth arrest and differentiation of cells of upper crypt and lower villus regions. Multiple alternatively spliced transcript variants encoding different isoforms have been observed for this gene.
MAPK/MAK/MRK overlapping kinase
, renal cell carcinoma antigen
, renal tumor antigen 1
, renal tumor antigen
, MAPK/MAK/MRK/ overlapping kinase
, MOK protein kinase
, T/STK 30