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NOS1AP encodes a cytosolic protein that binds to the signaling molecule, neuronal nitric oxide synthase (nNOS). Additionally we are shipping NOS1AP Antibodies (77) and NOS1AP Proteins (8) and many more products for this protein.
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Findings showed that NOS1AP (rs348624, rs12742393 and rs1415263), DISC1 (rs821633 and rs1000731), DAOA (rs2391191) and GSK3B (rs6438552) SNPs had no association with development of early-onset schizophrenia; however, our finding suggested statistically significant role of the interaction of NOS1AP, DISC1, DAOA and GSK3B polymorphisms in schizophrenia susceptibility.
Novel Hispanic/Latino-specific Single Nucleotide Polymorphism at NOS1AP implicate gene regulatory dysfunction in QT prolongation.
If the expression of Capon is decreased, myeloma cells are adhered to fibronectin (show FN1 ELISA Kits) or bone marrow stromal cells (bone marrow mesenchymal stem cells). In addition, the sensitivity of the cell line to chemotherapeutic agents was reduced after silencing Capon in the myeloma cell line which was adhered to bone marrow mesenchymal stem cells.
sex was identified as a moderator of the association between NOS1AP sequence variants and QTc prolongation in two long QT syndrome founder populations
Association between NOS1AP and PTSD severity, depression, anxiety, and stress was found. NOS1AP was associated with resilience.
gender modulated the interaction between NOS1AP promoter DNA methylation in intracranial aneurysm and brain arteriovenous malformation BAVM patients
results hint towards an involvement of NOS-I (show NOS1 ELISA Kits)/NOS1AP interaction in the regulation of dendritic spine plasticity
Results from genome-wide DNA methylation, functional network analysis and pyrosequencing, show selective CpG sites (NOS1AP, BID, and GABRB1) differentially methylated in smokers and chronic obstructive pulmonary disease patients compared to nonsmokers.
Data indicate that overexpression of nitric oxide synthase 1 adaptor protein short form (CAPON-S) led to the inactivation of the proto-oncogene (show RAB1A ELISA Kits) protein Akt (Akt (show AKT1 ELISA Kits)) signaling pathway.
SNPs in the NOS1AP gene influence QTc interval duration but we have not demonstrated a direct association with the risk of sudden cardiac death
Genetic variations at Nos1ap were associated with Oxidative Stress Induced (show SQSTM1 ELISA Kits) Ventricular Arrhythmia and Heart Failure, which may cause Sudden Cardiac Death.
Capon expression is increased in skeletal muscle by position, repair, NOS (show NOS ELISA Kits) activity, and dystrophy.
long-term expression of cocaine-induced behavioral sensitization in females (adolescent and adult) is nNOS (show NOS1 ELISA Kits)-independent, unlike previous finding in adult males.
This gene encodes a cytosolic protein that binds to the signaling molecule, neuronal nitric oxide synthase (nNOS). This protein has a C-terminal PDZ-binding domain that mediates interactions with nNOS and an N-terminal phosphotyrosine binding (PTB) domain that binds to the small monomeric G protein, Dexras1. Studies of the related mouse and rat proteins have shown that this protein functions as an adapter protein linking nNOS to specific targets, such as Dexras1 and the synapsins. Alternative splicing results in multiple transcript variants encoding different isoforms.
nitric oxide synthase 1 (neuronal) adaptor protein
, carboxyl-terminal PDZ ligand of neuronal nitric oxide synthase protein
, carboxyl-terminal PDZ ligand of neuronal nitric oxide synthase protein-like
, C-terminal PDZ domain ligand of neuronal nitric oxide synthase (CAPON)
, C-terminal PDZ ligand of neuronal nitric oxide synthase protein
, ligand of neuronal nitric oxide synthase with carboxyl-terminal PDZ domain
, nitric oxide synthase 1 adaptor protein
, C-terminal PDZ domain ligand of neuronal nitric oxide synthase