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KCNK9 encodes a member of the superfamily of potassium channel proteins containing two pore-forming P domains, and is highly expressed in the cerebellum. Additionally we are shipping KCNK9 Antibodies (29) and many more products for this protein.
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In conclusion, we have identified the first selective activator of the two-pore domain potassium channel TASK3.
KCNK9 imprinting syndrome is a rare cause of intellectual disability, congenital hypotonia, palatal abnormalities, and occasional seizures.
Our results provide evidence for new loci influencing abdominal visceral (BBS9 (show BBS9 Proteins), ADCY8 (show ADCY8 Proteins), KCNK9) and subcutaneous (MLLT10 (show MLLT10 Proteins)/DNAJC1 (show DNAJC1 Proteins)/EBLN1 (show EBLN2 Proteins)) fat, and confirmed a locus (THNSL2 (show THNSL2 Proteins)) previously reported to be associated with abdominal fat in women.
TASK-1 (show KCNK3 Proteins) and TASK-3 may form heterodimers in human atrial cardiomyocytes.
During conductance simulation experiments, both TASK-3 and TREK-1 (show KCNK2 Proteins) channels were able to repolarise the membrane once AP threshold was reached
The findings of this study suggest that variations in KCNK9 genes are associated with both mild and severe persistent breast pain after breast cancer surgery.
Diacylglycerol mediates regulation of TASK1 and TASK3 potassium channels by GNAQ.
Interference with TASK-3 channel expression, therefore, induces caspase (show CASP3 Proteins)-dependent and -independent apoptosis of melanoma cells, most likely via causing mitochondrial depolarization.
K2P3.1 (show KCNK3 Proteins) and K2P9.1 undergo rapid dynamin (show DNM1 Proteins)-dependent endocytosis
Mutations of KCNK9 or epigenetic disturbances within the PEG13 imprinted cluster do not significantly contribute to the cause of the developmental disabilities tested in this study.
results demonstrate that neurotensin (NT (show NTS Proteins)) transiently increased the neuronal excitability of dentate gyrus granule cells (GCs (show UGCG Proteins)) by inhibiting TASK-3 channels; NT-mediated increases in neuronal excitability facilitate long-term potentiation at the perforant path-GC synapses
KCNK9 channels are not involved in hearing.
THe results of this study suggested that dysfunction of KCNK9 causes a migration defect in the cortex via an activity-dependent mechanism.
Oxygen and mitochondrial inhibitors modulate both monomeric and heteromeric TASK-1 (show KCNK3 Proteins) and TASK-3 channels in mouse carotid body type-1 cells.
Data indicate that in 1-day-old Task3-/- mice, renal renin (show REN Proteins) concentration was lower than in wild-type mice.
DPP6 (show DPP6 Proteins) co-expression with TASK-3 results in the formation of a protein complex that enhances resting membrane potassium conductance.
An extracellular ion pathway plays a central role in the cooperative gating of a K(2P) K+ channel (show KCNC4 Proteins) by extracellular pH
Task3 plays an important role in the adaptation of aldosterone secretion to dietary salt intake.
This study demonistrated that kcnk9 gene expression in mouse dorsal raphe nucleus
TASK3 channel genes may contribute to the development of low renin (show REN Proteins) essential hypertension and idiopathic hyperaldosteronism in mice.
This gene encodes a member of the superfamily of potassium channel proteins containing two pore-forming P domains, and is highly expressed in the cerebellum. Amplification and overexpression of this gene has been observed in several types of human carcinomas, notably in breast cancer. This gene is imprinted in fetal brain, with preferential expression from the maternal allele.
potassium channel, subfamily K, member 9
, TWIK-related acid-sensitive K(+) channel 3
, TWIK-related acid-sensitive K+ channel 3
, acid-sensitive potassium channel protein TASK-3
, potassium channel TASK3
, potassium channel subfamily K member 9
, two pore K(+) channel KT3.2
, two pore potassium channel KT3.2
, potassium channel subfamily K member 9 (Task-3)
, potassium channel, subfamily K, member 9 (Task-3)
, TASK-1 potassium channel