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KCNJ12 encodes an inwardly rectifying K+ channel which may be blocked by divalent cations. Additionally we are shipping Potassium Inwardly-Rectifying Channel, Subfamily J, Member 12 Antibodies (86) and many more products for this protein.
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This report is the first to describe the KCNJ12 gene as a cause of familial dilated cardiomyopathy in patients
Nav1.5 (show SCN5A Proteins) N-terminal domain binding to alpha1-syntrophin (show SNTA1 Proteins) increases membrane density of human Kir2.1 (show KCNJ2 Proteins), Kir2.2 and Nav1.5 (show SCN5A Proteins) channels
The augmentation of Ca(2 (show CA2 Proteins)+) influx and cytokine release suggests a physiological role for Kir2.2 in TLR4 (show TLR4 Proteins)-stimulated monocytes.
Unconventional role of the inwardly rectifying potassium channel (show KCNAB2 Proteins) Kir2.2 as a constitutive activator of RelA (show NFkBP65 Proteins) in cancer.
Kir2.1 (show KCNJ2 Proteins) and Kir2.2 have two distinct lipid requirements for activity.
Kir2.2 knockdown induces senescence of cancer cells by a mechanism involving reactive oxygen species accumulation.
Kir2.1 (show KCNJ2 Proteins) and Kir2.2 subunits exert neither a dominant nor an anomalous effect on any of the properties of heteromeric channels.
KIr2.1 (show KCNJ2 Proteins) and Kir2.2 are directly activated by membrane phosphatidylinositol 4,5-bisphosphate.
Molecular cloning of functional KCNJ12 with an arginine residue at position 285.
transcripts for Kir2.2 potassium channels are identified in proliferative smooth muscle cells
Kir2.2 in the brainstem plays a transient role in hypercapnic ventilatory response , possibly through central ventilatory chemosensitivity, during postnatal development.
Computer simulations grounded in biophysical measurements of prelimbic and infralimbic cortex slices suggest a dynamic interaction among Kir2, Kleak, and HCN channel currents in shaping membrane potential and temporal integration of synaptic potentials.
This gene encodes an inwardly rectifying K+ channel which may be blocked by divalent cations. This protein is thought to be one of multiple inwardly rectifying channels which contribute to the cardiac inward rectifier current (IK1). The gene is located within the Smith-Magenis syndrome region on chromosome 17.
ATP-sensitive inward rectifier potassium channel 12
, inward rectifier K(+) channel Kir2.2
, potassium channel, inwardly rectifying subfamily J member 12
, potassium inwardly-rectifying channel, subfamily J, member 12
, inwardly-rectifying potassium channel Kir2.2
, inward rectifier K(+) channel Kir2.2v
, inward rectifier K(+) channel Kir2.6
, potassium inwardly-rectifying channel, subfamily J, inhibitor 1
, atrium potassium channel IRK
, potassium inwardly-rectifying channel J12