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The lysosphingolipid sphingosine 1-phosphate (S1P) regulates cell proliferation, apoptosis, motility, and neurite retraction.
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Bat Polyclonal S1PR5 Primary Antibody for IHC (p) - ABIN4351860
van Doorn, Lopes Pinheiro, Kooij, Lakeman, van het Hof, van der Pol, Geerts, van Horssen, van der Valk, van der Kam, Ronken, Reijerkerk, de Vries: Sphingosine 1-phosphate receptor 5 mediates the immune quiescence of the human brain endothelial barrier. in Journal of neuroinflammation 2012
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Cow (Bovine) Polyclonal S1PR5 Primary Antibody for IHC, WB - ABIN2775524
Guinn, Bland, Lodi, Liggins, Tobal, Petters, Wells, Banham, Mufti: Humoral detection of leukaemia-associated antigens in presentation acute myeloid leukaemia. in Biochemical and biophysical research communications 2005
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Human Polyclonal S1PR5 Primary Antibody for IHC, IHC (p) - ABIN4351866
Wünsche, Koch, Goldschmeding, Schwalm, Meyer Zu Heringdorf, Huwiler, Pfeilschifter: Transforming growth factor β2 (TGF-β2)-induced connective tissue growth factor (CTGF) expression requires sphingosine 1-phosphate receptor 5 (S1P5) in human mesangial cells. in Biochimica et biophysica acta 2015
Human Polyclonal S1PR5 Primary Antibody for IHC (p) - ABIN4351861
Strochlic, Dwivedy, van Horck, Falk, Holt: A role for S1P signalling in axon guidance in the Xenopus visual system. in Development (Cambridge, England) 2007
Dog (Canine) Polyclonal S1PR5 Primary Antibody for IHC (p), WB - ABIN4351875
Mannioui, Vauzanges, Fini, Henriet, Sekizar, Azoyan, Thomas, Pasquier, Giovannangeli, Demeneix, Lubetzki, Zalc: The Xenopus tadpole: An in vivo model to screen drugs favoring remyelination. in Multiple sclerosis (Houndmills, Basingstoke, England) 1970
Reduced DNA methylation (show HELLS Antibodies) may underlie the increased expression of the S1PR5 gene in alveolar macrophages and associated defective efferocytosis in COPD (show ARCN1 Antibodies).
Data indicate that the mitotic kinase Polo-like kinase 1 (PLK1 (show PLK1 Antibodies)) was an important effector of S1P (show MBTPS1 Antibodies)-S1P5 signaling, and a new function of the SphK1 (show SPHK1 Antibodies)-S1P (show MBTPS1 Antibodies) pathway specifically in the control of mitosis in HeLa cells.
TGF-beta2 (show TGFB2 Antibodies) dependent upregulation of S1P5 is required for the induction of pro-fibrotic CTGF (show CTGF Antibodies) by TGF-beta (show TGFB1 Antibodies) in mesangial cells.
This report is the first to demonstrate a reduction in S1P5 in multiple sclerosis lesions, which parallels that of myelin loss.
Data show that sphingosine kinase SphK1 (show SPHK1 Antibodies) and sphingosine-1-phosphate (S1P (show MBTPS1 Antibodies)) receptors S1P1 (show S1PR1 Antibodies), S1P2 (show S1PR2 Antibodies), S1P3 (show S1PR3 Antibodies), and S1P5 were expressed from primary, up to recurrent and secondary glioblastomas, with sphingosine kinase SphK2 (show SPHK2 Antibodies) levels were highest in primary tumors.
Edg-5 (show S1PR2 Antibodies) receptor in brain endothelial cells contributes to optimal barrier formation and maintenance of immune quiescence of the barrier endothelium.
The decreased expression level of S1PR5 on NK cells is associated with graft versus host disease occurrence after allogeneic hematopoietic stem cell transplantation.
The molecular identity, functional properties, and expression profile of the S1P5 (Edg-8) receptor have been characterized.
FTY720 induces time-dependent modulation of S1P (show MBTPS1 Antibodies) receptors on human OPCs with consequent functional responses that are directly relevant for the remyelination process.
The centrosomal sphingosine-1-phosphate receptor 5 might function as an intracellular target of sphingosine-1-phosphate linked to regulation of mitosis.
Coordinated changes in CXCR4 (show CXCR4 Antibodies) and S1P5 responsiveness govern NK-cell trafficking.
Fndings suggest that S1P (show S1PR1 Antibodies)(5) may mediate the effects of S1P (show S1PR1 Antibodies) in terms of regulating ERK-1 (show MAPK3 Antibodies)/2 signaling in ES cells.
Edg8/S1P5 activation on oligodendroglial cells modulates two distinct functional pathways mediating either process retraction or cell survival and that these effects depend on the developmental stage of the cell.
Sphingosine 1-phosphate receptors regulate chemokine (show CCL1 Antibodies)-driven transendothelial migration of lymph node but not splenic T cells.
KLF2 (show KLF2 Antibodies) regulates T cell homeostasis at least partly by controlling CD62L (show SELL Antibodies) and S1P1 (show S1PR1 Antibodies) expression, and therefore T cell egress from the thymus and circulation in the periphery.
These findings identify S1P5 as a T-bet-induced gene that is required for natural killer cell egress from lymph nodes and bone marrow.
The lysosphingolipid sphingosine 1-phosphate (S1P) regulates cell proliferation, apoptosis, motility, and neurite retraction. Its actions may be both intracellular as a second messenger and extracellular as a receptor ligand. S1P and the structurally related lysolipid mediator lysophosphatidic acid (LPA) signal cells through a set of G protein-coupled receptors known as EDG receptors. Some EDG receptors (e.g., EDG1\; MIM 601974) are S1P receptors\; others (e.g., EDG2\; MIM 602282) are LPA receptors.
S1P receptor 5
, S1P receptor Edg-8
, endothelial differentiation G-protein-coupled receptor 8
, endothelial differentiation, sphingolipid G-protein-coupled receptor, 8
, sphingosine 1-phosphate receptor 5
, sphingosine 1-phosphate receptor EDG8
, sphingosine 1-phosphate receptor Edg-8
, Sphingosine 1-phosphate receptor Edg-8
, endothelial differentiation, sphingolipid G-protein-coupled receptor 8
, lysophospholipid receptor B4
, sphingosine-1-phosphate receptor LPB4
, nerve growth factor-regulated G-protein-coupled receptor 1