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TNFRSF6B belongs to the tumor necrosis factor receptor superfamily. Additionally we are shipping TNFRSF6B Antibodies (81) and TNFRSF6B Proteins (10) and many more products for this protein.
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Human TNFRSF6B ELISA Kit for Sandwich ELISA - ABIN1979508
Lancrajan, Schneider-Stock, Naschberger, Schellerer, Stürzl, Enz: Absolute quantification of DcR3 and GDF15 from human serum by LC-ESI MS. in Journal of cellular and molecular medicine 2015
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DcR3 treatment caused HepG2 cell cytoskeleton remodeling, inhibited E-cadherin (show CDH1 ELISA Kits) expression, and promoted cell migration.
DcR3 gene polymorphisms are associated with sporadic breast infiltrating ductal carcinoma risk in Northeast Chinese females
Study found that both transcriptional expression and post-transcriptional DcR3 expression DcR3 expression were significantly up-regulated in colorectal cancer (CRC (show CALR ELISA Kits)) tissues. DcR3 was found to be playing a critical role in CRC (show CALR ELISA Kits) proliferation and migration in vitro and in tumorigenesis and metastasis in vivo.
Higher plasma DcR3 level was related to better coronary collateral circulation (CCC) formation and displayed potent predictive power for CCC status in patients with coronary artery disease.
High serum DcR3 levels are associated with disease severity in nonatopic asthma patients.
Results show that DcR3 engages TNF (show TNF ELISA Kits) ligands: FasL (show FASL ELISA Kits), LIGHT, and TL1A (show TNFSF15 ELISA Kits) in a structurally similar fashion and its promiscuity is due to recognition of backbone atoms and invariant side chains.
These results raise the possibility for involvement of TL1A (show TNFSF15 ELISA Kits)/DR3/DR3 (show TNFRSF25 ELISA Kits)-mediated mechanisms in epithelial-mesenchymal interactions and the development of inflammation-induced intestinal fibrosis in Crohn's disease.
when idiopathic pulmonary fibrosis cells interact with collagen matrix, aberrantly activated Akt (show AKT1 ELISA Kits) increases DcR3 expression via GSK-3beta-NFATc1 (show NFATC1 ELISA Kits) and protects IPF cells from the FasL (show FASL ELISA Kits)-dependent apoptotic pathway.
Coexpression of TNFRSF6B and PCDHGA3 was observed immunohistochemically in FL18 cells, suggesting potential cooperation in tumor cell maintenance.
Loss of DcR3 impaired the growth and invasive property of HCC cell line of HepG2.
This gene belongs to the tumor necrosis factor receptor superfamily. The encoded protein is postulated to play a regulatory role in suppressing FasL- and LIGHT-mediated cell death. It acts as a decoy receptor that competes with death receptors for ligand binding. Over-expression of this gene has been noted in gastrointestinal tract tumors. Read-through transcription into this gene from the neighboring upstream gene, which encodes regulator of telomere elongation helicase 1 (RTEL1), generates a non-coding transcript.
decoy receptor 3
, decoy receptor for Fas ligand
, tumor necrosis factor receptor superfamily member 6B
, tumor necrosis factor receptor superfamily, member 6b
, tumor necrosis factor receptor superfamily, member 6b, decoy