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FAT4 antibody

FAT4 Reactivity: Human WB, ICC Host: Rabbit Polyclonal unconjugated
Catalog No. ABIN1112888
  • Target See all FAT4 Antibodies
    FAT4 (FAT Tumor Suppressor Homolog 4 (FAT4))
    Reactivity
    • 17
    • 15
    • 15
    Human
    Host
    • 29
    • 3
    Rabbit
    Clonality
    • 29
    • 3
    Polyclonal
    Conjugate
    • 8
    • 3
    • 2
    • 2
    • 2
    • 2
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    This FAT4 antibody is un-conjugated
    Application
    • 20
    • 13
    • 13
    • 8
    • 3
    • 3
    • 2
    • 1
    • 1
    • 1
    Western Blotting (WB), Immunocytochemistry (ICC)
    Purification
    affinity purified
    Isotype
    IgG
    Top Product
    Discover our top product FAT4 Primary Antibody
  • Application Notes
    WB (1:500-1000), ICC (1:50-100). Other applications have not been tested. The optimal dilutions should be determined by end user.
    Restrictions
    For Research Use only
  • Format
    Liquid
    Buffer
    TBS (pH7.4), 0.5% BSA, 40% Glycerol and 0.05% Sodium Azide.
    Preservative
    Sodium azide, Thimerosal (Merthiolate)
    Storage
    4 °C/-20 °C
    Storage Comment
    Store at 4 °C after thawing (1 week). Aliquot and store at -20 °C for long term (at least one year). Avoid repeated freeze and thaw cycles.
    Expiry Date
    12 months
  • Target
    FAT4 (FAT Tumor Suppressor Homolog 4 (FAT4))
    Alternative Name
    FAT tumor suppressor homolog 4 / Protocadherin Fat 4 (FAT4 Products)
    Synonyms
    CDHF14 antibody, CDHR11 antibody, FAT-J antibody, FATJ antibody, NBLA00548 antibody, 6030410K14Rik antibody, 9430004M15 antibody, FAT atypical cadherin 4 antibody, FAT4 antibody, Fat4 antibody
    Background
    FAT tumor suppressor homolog 4 (FAT4), also known as cadherin family member 14 (CDHF14), is a 4,924-amino acid protein with 34 extracellular cadherin repeats, It is expressed broadly in fetal brain, infant brain, brain tumor, and colorectal cancer. Saburi et al. (2008) demonstrated that FAT4 regulates vertebrate PCP and that loss of PCP signaling may underlie some cystic diseases in humans.
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