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miR (show MLXIP Proteins)-595 acts as a phenotypic regulator of methotrexate sensitivity in CEM/C1 cells by targeting SLC19A1.
RFC, IL15 and VDR germline variants are associated with minimal residual disease in pediatric B-cell precursor ALL
We identified eight novel variants in SLC19A1 and twelve novel variants in FOLR1 (show FOLR1 Proteins), FOLR2 (show FOLR2 Proteins), and FOLR3 (show FOLR3 Proteins). Pathogenic variants include c.1265delG in SLC19A1 resulting in an early stop codon, four large insertion deletion variants in FOLR3 (show FOLR3 Proteins), and a stop_gain variant in FOLR3 (show FOLR3 Proteins)
Reduced SLC19A1 expression in human adipocytes induces DNA hypermethylation, resulting in increased expression of specific proinflammatory genes, including CCL2 (show CCL2 Proteins). This constitutes an epigenetic mechanism that might link dysfunctional adipocytes to white adipose tissue inflammation and Insulin (show INS Proteins) resistance.
data suggested that the RFC1 (show RFC1 Proteins) A80G G allele was an APOE (show APOE Proteins) 4-independent risk factor for late-onset Alzheimer's disease (AD
MTHFR (show MTHFR Proteins) 677C/T and RFC1 (show RFC1 Proteins) 80G/A polymorphisms may serve as predictors of toxicity during maintenance chemotherapy in childhood acute lymphoblastic leukemia or lymphoma.
Neonatal RFC1 (show RFC1 Proteins) polymorphism influenced total homocysteinein neonates
There was an association between RFC1 (show RFC1 Proteins) A80G variant and the risk of nonsymdromic cleft lip with or without palate.
the G80A mutation in the RFC1 (show RFC1 Proteins) gene is associated with a greater risk for sporadic breast cancer and oxidative DNA damage
We demonstrated no difference in tHcy, folates, vitamin B12 (show NDUFB3 Proteins) levels and allelic frequencies of C677T and G80A polymorphisms in MTHFR (show MTHFR Proteins) and RFC (show RFC1 Proteins) genes between obese and no obese Tunisian children.
The mRNAs encoding Folr2 and the intestinal folate transporter Slc46a1 were not detected in 2-cel embryos and blastocysts.
Interleukin-6 (show IL6 Proteins) regulated the efficacy of methotrexate by decreasing the expression of SLC19A1.
First evidence of RFC expression in rapidly dividing cells of developing neural tube, craniofacial region, limb buds and heart duggests that these regions may be particularly susceptible to folate deficiency.
Inactivation of RFC1 (show RFC1 Proteins) impacts the expression of several ligands and interacting proteins in the cubilin (show CUBN Proteins)-amnionless (show AMN Proteins)-megalin (show LRP2 Proteins) complex that are involved in the maternal-fetal transport of folate and other nutrients.
Intestinal folate uptake process undergoes differentiation-dependent regulation and that this regulation is mediated via changes in the level of expression of both the RFC and PCFT (show SLC46A1 Proteins).
The membrane protein encoded by this gene is a transporter of folate and is involved in the regulation of intracellular concentrations of folate. Three transcript variants encoding different isoforms have been found for this gene.
solute carrier family 19 (folate transporter), member 1
, folate transporter 1
, intestinal folate carrier 1
, placental folate transporter
, reduced folate carrier protein
, solute carrier family 19 member 1
, intestinal folate carrier protein
, reduced folate carrier 1
, solute carrier family 19 (sodium/hydrogen exchanger), member 1
, methotrexate carrier 1
, methotrexate carrier 2
, methotrexate carrier 5
, methotrexate carrier 6
, solute carrier family 19, member 1
, 58 kDa hydrophobic protein
, Folate carrier protein
, Methotrexate uptake protein
, Solute carrier family 19 member 1