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High HRAS (show HRAS ELISA Kits) expression is associated with hepatocarcinogenesis.
p21-associated inhibition of early-stage malignant progression and the intense expression in papilloma outgrowths, identifies a novel, significant antagonism between p21 and ras(Ha)/ROCK2 (show ROCK2 ELISA Kits)/NF-kappaB (show NFKB1 ELISA Kits) signalling in skin carcinogenesis.these data show that ROCK2 (show ROCK2 ELISA Kits) activation induces malignancy in ras(Ha)-initiated/promoted papillomas in the context of p53 (show TP53 ELISA Kits) loss and novel NF-kappaB (show NFKB1 ELISA Kits) expression
this study shows that retinoic acid stabilizes HRas (show HRAS ELISA Kits) protein during neurogenesis.
we provide genetic evidence that the wild-type H-Ras and K-Ras proteins are bioequivalent in spite of their different structural and biological properties
loss of one allele of Hras (show HRAS ELISA Kits) increased the sensitivity of mice to this carcinogen, and this effect was further exacerbated by the loss of the second Hras (show HRAS ELISA Kits) allele. However, loss of one or both alleles of Nras (show NRAS ELISA Kits) failed to alter tumor burden, either in the absence or presence of Hras (show HRAS ELISA Kits), after exposure to urethane.
H-ras (show HRAS ELISA Kits) isoform mediates protection against pressure overload-induced cardiac dysfunction in part through activation of AKT (show AKT1 ELISA Kits)/PI3K signaling pathway.
The long intergenic non-coding RNA CCR492 functions as a let-7 competitive endogenous RNA to de-repress c-Myc (show MYC ELISA Kits) expression and to promote cell transformation assisted by the constitutively active H-Ras (show HRAS ELISA Kits).
these contrasting signatures precisely match those proposed to confer bias toward Hras (show HRAS ELISA Kits)(CAA61CTA) versus Braf (show BRAF ELISA Kits)(GTG636GAG) mutations in the original tumor sets. Our findings highlight a novel mechanism whereby exposure history acts through strand-biased mutagenesis to specify activation of preferred oncogenes
The abnormal expression of epidermal cytokeratins suggests that Ha-Ras and Bcl-2 (show BCL2 ELISA Kits) suppress the terminal differentiation and sustain the stem cell-like features in epidermal keratinocytes
Ras (G12V)-induced cyclin D1 (show CCND1 ELISA Kits) protein synthesis was markedly suppressed by the knockdown of IL-33 (show IL33 ELISA Kits).
Suggest that loss of RASSF7 expression results in apoptosis in nucleus pulposus cells in human intervertebral disc degeneration.
truncated RASSF7 could act as an oncogene (show RAB1A ELISA Kits) in a small subset of tumours where it is mutated in this way.
The RASSF gene family members RASSF5 (show RASSF5 ELISA Kits), RASSF6 (show RASSF6 ELISA Kits) and RASSF7 show frequent DNA methylation (show HELLS ELISA Kits) in neuroblastoma (show ARHGEF16 ELISA Kits).
RASSF7 acts in concert with N-Ras (show NRAS ELISA Kits) to constitute a stress-sensitive temporary mechanism of apoptotic regulation.
RASSF7 regulates the microtubule cytoskeleton and is required for spindle formation, Aurora B (show AURKB ELISA Kits) activation and chromosomal congression during mitosis.
"rare" alleles of HRas1 minisatellite are associated with increased risk of papillary thyroid cancer formation in children and adolescents after Chernobyl accident
Ras proteins bind GDP/GTP and possess intrinsic GTPase activity.
, neuroblastoma ras oncogene
, transforming protein N-Ras
, v-Ha-ras Harvey rat sarcoma viral oncogene homolog
, GTPase HRas
, transforming protein p21
, v-Ha-ras Harvey rat sarcoma viral oncogene-like protein
, H-ras 1 protein
, c-Ha-ras p21 protein
, c-Ha-ras transgene
, transforming protein P21
, HRAS1-related cluster protein 1
, HRAS1-related cluster-1
, Ras association (RalGDS/AF-6) domain family 7
, ras association domain-containing protein 7