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Dog (Canine) Monoclonal TUBB5 Primary Antibody for FACS, ICC - ABIN4363535
Tan, Toops, Lakkaraju: Protective responses to sublytic complement in the retinal pigment epithelium. in Proceedings of the National Academy of Sciences of the United States of America 2016
Human Polyclonal TUBB5 Primary Antibody for WB - ABIN4891280
Chandra, Hiniker, Kuo, Nussbaum, Liddle: α-Synuclein in gut endocrine cells and its implications for Parkinson's disease. in JCI insight 2017
Human Polyclonal TUBB5 Primary Antibody for ICC, IF - ABIN442178
Xu, Janocha, Leahy, Klatte, Dudzinski, Mavrakis, Comhair, Lauer, Cotton, Erzurum: A novel method for pulmonary research: assessment of bioenergetic function at the air-liquid interface. in Redox biology 2014
TUBB3 (show TUBB3 Antibodies) and TUBB4 (show TUBB3 Antibodies) are necessary for the transport and proper localization of N-cadherin (show CDH2 Antibodies) within the plasma membrane.
The data of this studies suggest that mutations in TUBB4A exceedingly rarely contribute to the etiology of isolated dystonia.
The different clinical phenotypes associated with TUBB4A reflect the selective and specific cellular effects of the causative mutations. Cellular specificity of disease pathogenesis is relevant to developing targeted treatments for this disabling condition.
Together, DYT4-associated TUBB4A mutants themselves form aberrant tubulin (show TUBB Antibodies) networks and inhibit neuronal process growth, possibly explaining progress through the pathological states at cellular levels.
Genetic screening targeted at currently known disease-causing mutations in TOR1A (show TOR1A Antibodies), THAP1 (show THAP1 Antibodies), and TUBB4 (show TUBB3 Antibodies) appears to have low diagnostic yield in sporadic spasmodic dysphonia. In our cohort, only 2 patients tested positive for novel/rare variants in THAP1 (show THAP1 Antibodies).
TUBB4A-mutated patients manifest a comparable clinical and neuroimaging picture but they can differ from each other in terms of rate of disease progression
our data indicate that TUBB4A coding mutations do not play a critical role in the broad population of isolated dystonia patients
a paclitaxel-resistant beta-tubulin (show TUBB Antibodies) isotype, betaIVa-tubulin (show TUBB Antibodies), was the most up-regulated gene compared with other beta-tubulin (show TUBB Antibodies) isotypes in H460 floating cells, concomitant with elevated ERK (show EPHB2 Antibodies) activation
The study adds complicated hereditary spastic paraplegia to the clinical spectrum of TUBB4A-associated neurological disorders.
Data suggested H-ABC (show ABCB6 Antibodies) and DYT4 belong to a continuous phenotypic spectrum associated with TUBB4A mutations.
The axial elastic modulus of tubulin (show TUBB Antibodies) diminishes as the length of the monomer increases.
Differentiation-mediated activation of JunD results in enhanced TUBB4a expression in P19 embryonal carcinoma cells.
beta 5-tubulin\; polymerizes to form microtubules\; member of a family of structural proteins
class IVa beta-tubulin
, tubulin 5 beta
, tubulin beta-4 chain
, tubulin beta-4A chain
, tubulin, beta 4 class IVa
, tubulin, beta, 5
, tubulin beta-3 chain
, tubulin, beta, 3
, tubulin, beta, 4
, tubulin beta-2B chain
, tubulin beta-5 chain
, tubulin, beta 2A
, tubulin, beta 2B class IIb
, tubulin, beta polypeptide
, beta 5-tubulin
, c(beta)7 tubulin
, tubulin beta 4'
, tubulin beta-7 chain
, tubulin, beta 4
, beta-tubulin class-III
, beta-Tubulin at 56D
, tubulin, beta 2C
, beta-4 tubulin
, LOW QUALITY PROTEIN: tubulin beta-4A chain
, Tubulin beta-4 chain
, tubulin beta 4A class IVa S homeolog
, tubulin beta5
, tubulin, beta 4A class IVa