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Quantitative real-time PCR confirmed that the histone H3 (show HIST3H3 Proteins) and H4 tails were important to preserve stringent transcriptional repression of Snf1-dependent genes when glucose was present.
Findings suggest that protective effects of endotoxin exposure on asthma may vary depending upon the presence or absence of a polymorphism in ACAA1.
Molecular cloning, gene structure and tissue distribution of 3-ketoacyl-CoA thiolase A.
This gene encodes an enzyme operative in the beta-oxidation system of the peroxisomes. Deficiency of this enzyme leads to pseudo-Zellweger syndrome. Alternative splicing results in multiple transcript variants.
3-ketoacyl-CoA thiolase, peroxisomal
, acetyl-Coenzyme A acyltransferase 1
, peroxisomal 3-oxoacyl-CoA thiolase
, peroxisomal 3-oxoacyl-Coenzyme A thiolase
, 3-ketoacyl-CoA thiolase A, peroxisomal
, acetyl-CoA acyltransferase A
, beta-ketothiolase A
, peroxisomal 3-ketoacyl-CoA thiolase
, peroxisomal 3-oxoacyl-CoA thiolase A
, thiolase A
, 3-ketoacyl-CoA thiolase B, peroxisomal
, acetyl-Coenzyme A acyltransferase 1 (peroxisomal 3-oxoacyl-Coenzyme A thiolase)
, mitochondrial acetyl-Coenzyme A acyltransferase 1
, acyl carrier protein synthase (acaA-1)
, acetyl-CoA acyltransferase 1
, 3-ketoacyl-CoA thiolase, peroxisomal-like
, Acetyl-CoA acyltransferase 3-oxo acyl-CoA thiolase A peroxisomal
, acetyl-CoA acyltransferase 1A
, acetyl-CoA acyltransferase 3-oxo acyl-CoA thiolase A 1 peroxisomal
, acetyl-CoA acyltransferase, 3-oxo acyl-CoA thiolase A
, acetyl-Coenzyme A acyltransferase 1A