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anti-Human ZBTB16 Antibodies:
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Human Monoclonal ZBTB16 Primary Antibody for FACS - ABIN4895342
Gérart, Sibéril, Martin, Lenoir, Aguilar, Picard, Lantz, Fischer, Latour: Human iNKT and MAIT cells exhibit a PLZF-dependent proapoptotic propensity that is counterbalanced by XIAP. in Blood 2013
Show all 5 Pubmed References
Human Monoclonal ZBTB16 Primary Antibody for ChIP, ICC - ABIN2668927
Koken, Reid, Quignon, Chelbi-Alix, Davies, Kabarowski, Zhu, Dong, Chen, Chen, Tan, Licht, Waxman, de Thé, Zelent: Leukemia-associated retinoic acid receptor alpha fusion partners, PML and PLZF, heterodimerize and colocalize to nuclear bodies. in Proceedings of the National Academy of Sciences of the United States of America 1997
Show all 3 Pubmed References
Human Monoclonal ZBTB16 Primary Antibody for ICC, ELISA - ABIN969465
Felicetti, Errico, Bottero, Segnalini, Stoppacciaro, Biffoni, Felli, Mattia, Petrini, Colombo, Peschle, Carè: The promyelocytic leukemia zinc finger-microRNA-221/-222 pathway controls melanoma progression through multiple oncogenic mechanisms. in Cancer research 2008
Cow (Bovine) Polyclonal ZBTB16 Primary Antibody for WB - ABIN2780737
Deng, Zhang, Kang, Liu, Zhao, Gao, Li, Pan, Tang, Wang, Niu, Yang, Zeng: An unusual haplotype structure on human chromosome 8p23 derived from the inversion polymorphism. in Human mutation 2008
Show all 2 Pubmed References
Human Monoclonal ZBTB16 Primary Antibody for CyTOF, FACS - ABIN4899031
Hsieh, Botta, Gao, Li, Van Allen, Treacy, Cai, He, Sweeney, Brown, Balk, Nelson, Garraway, Kantoff: PLZF, a tumor suppressor genetically lost in metastatic castration-resistant prostate cancer, is a mediator of resistance to androgen deprivation therapy. in Cancer research 2015
Human Monoclonal ZBTB16 Primary Antibody for ELISA, PLA - ABIN2773728
Narimatu, Nagata, Sugizaki, Hamasaki: [A new method for immunological detection of fecal blood, using colloidal gold labeled with anti-human hemoglobin monoclonal antibody]. in Rinsho byori. The Japanese journal of clinical pathology 1992
Human Polyclonal ZBTB16 Primary Antibody for ICC, IF - ABIN4346320
Wasim, Carlet, Mansha, Greil, Ploner, Trockenbacher, Rainer, Kofler: PLZF/ZBTB16, a glucocorticoid response gene in acute lymphoblastic leukemia, interferes with glucocorticoid-induced apoptosis. in The Journal of steroid biochemistry and molecular biology 2010
ZBTB16 is a sensitive and specific marker for yolk sac (show ADCY10 Antibodies) tumour (YST) and is diagnostically superior to AFP (show AFP Antibodies) and glypican-3 (show GPC3 Antibodies) in metastatic and extragonadal settings.
Selected gene variants of the transcription factor ZBTB16 influence obesity-related parameters and serum lipid levels.
ZBTB16 is a highly sensitive and specific marker for yolk sac (show ADCY10 Antibodies) tumors.
Low ZBTB16 expression is associated with tongue squamous cell carcinoma.
High PLZF expression is associated with acute myeloid leukemia (show BCL11A Antibodies).
Emerging evidence shows that PLZF regulates the balance of self-renewal and differentiation in stem cells.
integration of PLZF ChIP-Seq and RNA Pol II (show 0 Antibodies) RNA-Seq datasets revealed that the early growth response 1 (EGR1 (show EGR1 Antibodies)) transcription factor is a PLZF target for which its level of expression must be reduced to enable progesterone dependent hESC decidualization
NLRP7 (show NLRP7 Antibodies) is involved in Hydatidiform Molar Pregnancy (HYDM1) and interacts with the transcriptional repressor ZBTB16
These results support a prognostic value for loss of cytoplasmic PLZF expression in the stratification of non-small cell lung cancer and a possible role of cytoplasmic shift and down-regulation of PLZF in the pathogenesis of NSCLC.
down-regulation of PLZF is an important molecular process for prostate tumor progression
Enhanced ZBTB16 promoted both white and brown-like adipogenesis of bovine intramuscular preadipocytes. Increased expressions of adipogenic transcription factors such as PPARgamma (show PPARG Antibodies), C/EBPalpha (show CEBPA Antibodies), FABP4 (show FABP4 Antibodies), and ADIPOQ participated in the pro-adipogenic effects of ZBTB16.
Zbtb16-encoded transcription factor PLZF directs the differentiation of multiple innate and innate-like cell lineages. Specific Runx1 (show RUNX1 Antibodies)-bound enhancer elements critically modulate lineage-dependent expressions of PLZF.
these findings demonstrate that PLZF controls ROS (show ROS1 Antibodies) levels, which in turn governs the inflammatory function of NKT (show CTSL1 Antibodies) cells
these data reveal the full profile of PLZF and SALL4 (show SALL4 Antibodies) regulatory targets in undifferentiated spermatogonia.
Several layers of regulation of the innate-like NKT (show CTSL1 Antibodies) effector program: First, PLZF bound and regulated genes encoding cytokine receptors as well as homing and adhesion receptors; second, PLZF bound and activated T-helper-specific transcription factor genes that in turn control T-helper-specific programs; finally, PLZF bound and suppressed the transcription of Bach2 (show BACH2 Antibodies), a potent general repressor of effector differentiation
Together, these results suggest a novel mechanism regulating endothelial PATZ1 (show ZNF278 Antibodies) expression based on the down-regulation of miR (show MLXIP Antibodies)-24 expression caused by hyperglycemia. Interfering with PATZ1 (show ZNF278 Antibodies) expression via miRNAs or miRNA mimics could potentially represent a new way to target endothelial PATZ1 (show ZNF278 Antibodies)-dependent signaling of vascular dysfunction in diabetes.
Results clearly indicate that PLZF promotes cell cycle exit and cell differentiation into hypertrophic chondrocytes.
plzf has a role in regulating HSC (show FUT1 Antibodies) function that is linked to cell cycle regulation
PLZF expression is highly specific to innate T cells and cannot be induced in conventional T cells via TCR-mediated activation or inflammatory challenge
PLZF expression at the innate lymphoid cell precursor stage has a long-range effect on the functional properties of mature ILC2s and highlights the importance of these cells for innate allergic responses in otherwise immunocompetent mice.
Data suggest that age-related increase in Plzf expression represents a novel molecular signature of spermatogonia aging by functionally arresting their differentiation.
This gene is a member of the Krueppel C2H2-type zinc-finger protein family and encodes a zinc finger transcription factor that contains nine Kruppel-type zinc finger domains at the carboxyl terminus. This protein is located in the nucleus, is involved in cell cycle progression, and interacts with a histone deacetylase. Specific instances of aberrant gene rearrangement at this locus have been associated with acute promyelocytic leukemia (APL). Alternate transcriptional splice variants have been characterized.
zinc finger and BTB domain-containing protein 16
, zinc finger protein 145 (Kruppel-like, expressed in promyelocytic leukemia)
, zinc finger protein PLZF
, promyelocytic leukemia zinc finger protein
, Green's luxoid
, zinc finger protein 145
, Polydactyly-luxate syndrome
, promyelocytic leukemia zinc finger
, zinc finger and BTB domain containing 16 protein
, promyelocytic leukemia zinc finger protein-A
, zinc finger and BTB domain containing 16
, zinc finger and BTB domain-containing protein 16-A
, zinc finger protein PLZF-A