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NAD-ME Protein (Myc-DYKDDDDK Tag)

NAD-ME Origin: Human Host: HEK-293 Cells Recombinant > 80 % as determined by SDS-PAGE and Coomassie blue staining AbP, STD
Catalog No. ABIN2726776
  • Target See all NAD-ME Proteins
    NAD-ME (NAD Dependent Malate Dehydrogenase (NAD-ME))
    Protein Type
    Recombinant
    Origin
    • 3
    • 1
    Human
    Source
    • 2
    • 2
    HEK-293 Cells
    Purification tag / Conjugate
    This NAD-ME protein is labelled with Myc-DYKDDDDK Tag.
    Application
    Antibody Production (AbP), Standard (STD)
    Characteristics
    • Recombinant human NAD-ME protein expressed in HEK293 cells.
    • Produced with end-sequenced ORF clone
    Purity
    > 80 % as determined by SDS-PAGE and Coomassie blue staining
    Top Product
    Discover our top product NAD-ME Protein
  • Application Notes
    Recombinant human proteins can be used for:
    Native antigens for optimized antibody production
    Positive controls in ELISA and other antibody assays
    Comment

    The tag is located at the C-terminal.

    Restrictions
    For Research Use only
  • Concentration
    50 μg/mL
    Buffer
    25 mM Tris.HCl, pH 7.3, 100 mM glycine, 10 % glycerol.
    Storage
    -80 °C
    Storage Comment
    Store at -80°C. Thaw on ice, aliquot to individual single-use tubes, and then re-freeze immediately. Only 2-3 freeze thaw cycles are recommended.
  • Target
    NAD-ME (NAD Dependent Malate Dehydrogenase (NAD-ME))
    Alternative Name
    Nad-Me (NAD-ME Products)
    Synonyms
    ODS1 Protein, AW120568 Protein, D030040L20Rik Protein, NAD-ME Protein, zgc:100941 Protein, malic enzyme 2 Protein, malic enzyme 2, NAD(+)-dependent, mitochondrial Protein, malic enzyme 2, NAD(+)-dependent, mitochondrial S homeolog Protein, ME2 Protein, Me2 Protein, me2.S Protein, me2 Protein
    Background
    This gene encodes a mitochondrial NAD-dependent malic enzyme, a homotetrameric protein, that catalyzes the oxidative decarboxylation of malate to pyruvate. It had previously been weakly linked to a syndrome known as Friedreich ataxia that has since been shown to be the result of mutation in a completely different gene. Certain single-nucleotide polymorphism haplotypes of this gene have been shown to increase the risk for idiopathic generalized epilepsy. Alternatively spliced transcript variants encoding different isoforms found for this gene.
    Molecular Weight
    63.4 kDa
    NCBI Accession
    NP_002387
    Pathways
    Production of Molecular Mediator of Immune Response
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